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Metabolic regulation of mycobacterial growth and antibiotic sensitivity

Metabolic regulation of mycobacterial growth and antibiotic sensitivity

http://www.wikidata.org/entity/Q28478251

about

Activity of 3-ketosteroid 9α-hydroxylase (KshAB) indicates cholesterol side chain and ring degradation occur simultaneously in Mycobacterium tuberculosis Next-generation antimicrobials: from chemical biology to first-in-class drugs Acylation of Biomolecules in Prokaryotes: a Widespread Strategy for the Control of Biological Function and Metabolic Stress Experimental Models of Foamy Macrophages and Approaches for Dissecting the Mechanisms of Lipid Accumulation and Consumption during Dormancy and Reactivation of Tuberculosis Mycobacterium tuberculosis Transcription Machinery: Ready To Respond to Host Attacks New insights into TB physiology suggest untapped therapeutic opportunities Equation-free analysis of two-component system signalling model reveals the emergence of co-existing phenotypes in the absence of multistationarity Lipidomic analysis links mycobactin synthase K to iron uptake and virulence in M. tuberculosis Succinate dehydrogenase is the regulator of respiration in Mycobacterium tuberculosis High Persister Mutants in Mycobacterium tuberculosis The Mycobacterium tuberculosis regulatory network and hypoxia Metabolic Perspectives on Persistence Rv3723/LucA coordinates fatty acid and cholesterol uptake in Mycobacterium tuberculosis.Oleoyl coenzyme A regulates interaction of transcriptional regulator RaaS (Rv1219c) with DNA in mycobacteria.Slow growth of Mycobacterium tuberculosis at acidic pH is regulated by phoPR and host-associated carbon sources.Energetics of Respiration and Oxidative Phosphorylation in Mycobacteria.Extensive in vivo resilience of persistent Salmonella Wax ester synthesis is required for Mycobacterium tuberculosis to enter in vitro dormancy.Outcome correlation of smear-positivity but culture-negativity during standard anti-tuberculosis treatment in Taiwan Mycobacterium tuberculosis metabolism Comparative Ser/Thr/Tyr phosphoproteomics between two mycobacterial species: the fast growing Mycobacterium smegmatis and the slow growing Mycobacterium bovis BCG.Deficiency of the novel exopolyphosphatase Rv1026/PPX2 leads to metabolic downshift and altered cell wall permeability in Mycobacterium tuberculosis Gut permeability, its interaction with gut microflora and effects on metabolic health are mediated by the lymphatics system, liver and bile acid.In-Vivo Gene Signatures of Mycobacterium tuberculosis in C3HeB/FeJ Mice.Clonal Diversification and Changes in Lipid Traits and Colony Morphology in Mycobacterium abscessus Clinical Isolates.Metabolic modeling predicts metabolite changes in Mycobacterium tuberculosis.Antibiotic efficacy is linked to bacterial cellular respiration.Interactions among filamentous fungi Aspergillus niger, Fusarium verticillioides and Clonostachys rosea: fungal biomass, diversity of secreted metabolites and fumonisin production.Targeting Mycobacterium tuberculosis Tumor Necrosis Factor Alpha-Downregulating Genes for the Development of Antituberculous Vaccines.Inhibitors of Mycobacterium tuberculosis DosRST signaling and persistence.Bactericidal Antibiotics Induce Toxic Metabolic Perturbations that Lead to Cellular Damage Trace incorporation of heavy water reveals slow and heterogeneous pathogen growth rates in cystic fibrosis sputum.Targeting drug tolerance in mycobacteria: a perspective from mycobacterial biofilms Host-directed therapy targeting the Mycobacterium tuberculosis granuloma: a review Immune activation of the host cell induces drug tolerance in Mycobacterium tuberculosis both in vitro and in vivo.Multiscale Model of Mycobacterium tuberculosis Infection Maps Metabolite and Gene Perturbations to Granuloma Sterilization Predictions Fresh approaches to anti-infective therapies.Iron and citrate export by a major facilitator superfamily pump regulates metabolism and stress resistance in Salmonella Typhimurium.Stringent Response Factors PPX1 and PPK2 Play an Important Role in Mycobacterium tuberculosis Metabolism, Biofilm Formation, and Sensitivity to Isoniazid In Vivo.The Minimal Unit of Infection: Mycobacterium tuberculosis in the Macrophage.

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Metabolic regulation of mycobacterial growth and antibiotic sensitivity

http://www.wikidata.org/entity/Q28478251

description

2011 nî lūn-bûn

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2011 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2011 թվականի մայիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Metabolic regulation of mycobacterial growth and antibiotic sensitivity

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Metabolic regulation of mycobacterial growth and antibiotic sensitivity

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Metabolic regulation of mycobacterial growth and antibiotic sensitivity

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Metabolic regulation of mycobacterial growth and antibiotic sensitivity

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Metabolic regulation of mycobacterial growth and antibiotic sensitivity

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Metabolic regulation of mycobacterial growth and antibiotic sensitivity

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Metabolic regulation of mycobacterial growth and antibiotic sensitivity

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JOURNAL.PBIO.1001065

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Metabolic regulation of mycobacterial growth and antibiotic sensitivity

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Alice H Li

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Christopher M Sassetti

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Seung-Hun Baek

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P2860

Comprehensive identification of conditionally essential genes in mycobacteria

PA-824 kills nonreplicating Mycobacterium tuberculosis by intracellular NO release

A common mechanism of cellular death induced by bactericidal antibiotics

The salicylate-derived mycobactin siderophores of Mycobacterium tuberculosis are essential for growth in macrophages

An in vitro model for sequential study of shiftdown of Mycobacterium tuberculosis through two stages of nonreplicating persistence

Prospects for advancing tuberculosis control efforts through novel therapies

Cytological and transcript analyses reveal fat and lazy persister-like bacilli in tuberculous sputum

A novel in vitro multiple-stress dormancy model for Mycobacterium tuberculosis generates a lipid-loaded, drug-tolerant, dormant pathogen

Induction of a novel class of diacylglycerol acyltransferases and triacylglycerol accumulation in Mycobacterium tuberculosis as it goes into a dormancy-like state in culture

DosS responds to a reduced electron transport system to induce the Mycobacterium tuberculosis DosR regulon

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