Mycobacterium tuberculosis uses host triacylglycerol to accumulate lipid droplets and acquires a dormancy-like phenotype in lipid-loaded macrophages - Wikidata - awgldk - TriplyDB

Mycobacterium tuberculosis uses host triacylglycerol to accumulate lipid droplets and acquires a dormancy-like phenotype in lipid-loaded macrophages

Mycobacterium tuberculosis uses host triacylglycerol to accumulate lipid droplets and acquires a dormancy-like phenotype in lipid-loaded macrophages

http://www.wikidata.org/entity/Q28478716

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Nocardia brasiliensis induces formation of foamy macrophages and dendritic cells in vitro and in vivo Lipid body-phagosome interaction in macrophages during infectious diseases: host defense or pathogen survival strategy?Mycobacterium tuberculosis: Manipulator of Protective Immunity The lipid droplet-a well-connected organelle Heterogeneity in tuberculosis pathology, microenvironments and therapeutic responses Translating basic science insight into public health action for multidrug- and extensively drug-resistant tuberculosis Mycobacterium tuberculosis: success through dormancy Feeding uninvited guests: mTOR and AMPK set the table for intracellular pathogens Lipids at the interface of virus-host interactions Hypoxia-mediated regulation of macrophage functions in pathophysiology Lipid Droplet Formation, Their Localization and Dynamics during Leishmania major Macrophage Infection Lipid Body Organelles within the Parasite Trypanosoma cruzi: A Role for Intracellular Arachidonic Acid Metabolism Experimental Models of Foamy Macrophages and Approaches for Dissecting the Mechanisms of Lipid Accumulation and Consumption during Dormancy and Reactivation of Tuberculosis Reprogramming neutral lipid metabolism in mouse dendritic leucocytes hosting live Leishmania amazonensis amastigotes Assessment of treatment response in tuberculosis MmPPOX inhibits Mycobacterium tuberculosis lipolytic enzymes belonging to the hormone-sensitive lipase family and alters mycobacterial growth An acyl-CoA synthetase in Mycobacterium tuberculosis involved in triacylglycerol accumulation during dormancy Novel inhibitors of cholesterol degradation in Mycobacterium tuberculosis reveal how the bacterium's metabolism is constrained by the intracellular environment Replication rates of Mycobacterium tuberculosis in human macrophages do not correlate with mycobacterial antibiotic susceptibility Mycobacterial Metabolic Syndrome: LprG and Rv1410 Regulate Triacylglyceride Levels, Growth Rate and Virulence in Mycobacterium tuberculosis The Mycobacterium tuberculosis regulatory network and hypoxia Three-dimensional in vitro models of granuloma to study bacteria-host interactions, drug-susceptibility, and resuscitation of dormant mycobacteria Metabolic Perspectives on Persistence Comparative analyses of transport proteins encoded within the genomes of Mycobacterium tuberculosis and Mycobacterium leprae Delayed bactericidal response of Mycobacterium tuberculosis to bedaquiline involves remodelling of bacterial metabolism.Lipid accumulation during the establishment of kleptoplasty in Elysia chlorotica.Bovis Bacillus Calmette-Guerin (BCG) infection induces exosomal miRNA release by human macrophages Rv3723/LucA coordinates fatty acid and cholesterol uptake in Mycobacterium tuberculosis.Pathogenicity of Mycobacterium tuberculosis is expressed by regulating metabolic thresholds of the host macrophage.Oleoyl coenzyme A regulates interaction of transcriptional regulator RaaS (Rv1219c) with DNA in mycobacteria.Simvastatin increases the in vivo activity of the first-line tuberculosis regimen.Manipulation of the mononuclear phagocyte system by Mycobacterium tuberculosis.Wax ester synthesis is required for Mycobacterium tuberculosis to enter in vitro dormancy.Human granuloma in vitro model, for TB dormancy and resuscitation.Characterizing virulence-specific perturbations in the mitochondrial function of macrophages infected with Mycobacterium tuberculosis.Identification of residues involved in substrate specificity and cytotoxicity of two closely related cutinases from Mycobacterium tuberculosis The Sculpting of the Mycobacterium tuberculosis Genome by Host Cell-Derived Pressures Macrophage immunoregulatory pathways in tuberculosis.Cyclic AMP-dependent resuscitation of dormant Mycobacteria by exogenous free fatty acids.Non-replicating Mycobacterium tuberculosis elicits a reduced infectivity profile with corresponding modifications to the cell wall and extracellular matrix.

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Mycobacterium tuberculosis uses host triacylglycerol to accumulate lipid droplets and acquires a dormancy-like phenotype in lipid-loaded macrophages

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2011 nî lūn-bûn

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2011 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2011 թվականի հունիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Mycobacterium tuberculosis use ...... pe in lipid-loaded macrophages

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Mycobacterium tuberculosis use ...... pe in lipid-loaded macrophages

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Mycobacterium tuberculosis use ...... pe in lipid-loaded macrophages

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Mycobacterium tuberculosis use ...... pe in lipid-loaded macrophages

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Mycobacterium tuberculosis use ...... pe in lipid-loaded macrophages

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Mycobacterium tuberculosis use ...... pe in lipid-loaded macrophages

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Mycobacterium tuberculosis use ...... pe in lipid-loaded macrophages

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JOURNAL.PPAT.1002093

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Mycobacterium tuberculosis use ...... pe in lipid-loaded macrophages

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Chirajyoti Deb

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Hédia Maamar

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Jaiyanth Daniel

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Pappachan E Kolattukudy

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Tatiana D Sirakova

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P2860

Mycobacterium tuberculosis isocitrate lyases 1 and 2 are jointly required for in vivo growth and virulence

Biochemical differentiation of Mycobacterium tuberculosis grown in vivo and in vitro

The receptor-mediated uptake, survival, replication, and drug sensitivity of Mycobacterium tuberculosis within the macrophage-like cell line THP-1: a comparison with human monocyte-derived macrophages

Comparison of activities of rifapentine and rifampin against Mycobacterium tuberculosis residing in human macrophages

An in vitro model for sequential study of shiftdown of Mycobacterium tuberculosis through two stages of nonreplicating persistence

Is adipose tissue a place for Mycobacterium tuberculosis persistence?

Cytological and transcript analyses reveal fat and lazy persister-like bacilli in tuberculous sputum

A novel in vitro multiple-stress dormancy model for Mycobacterium tuberculosis generates a lipid-loaded, drug-tolerant, dormant pathogen

Induction of a novel class of diacylglycerol acyltransferases and triacylglycerol accumulation in Mycobacterium tuberculosis as it goes into a dormancy-like state in culture

A novel lipase belonging to the hormone-sensitive lipase family induced under starvation to utilize stored triacylglycerol in Mycobacterium tuberculosis

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10.1371/JOURNAL.PPAT.1002093

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Mycobacterium tuberculosis

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3121879

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