Inactivation of the glial fibrillary acidic protein gene, but not that of vimentin, improves neuronal survival and neurite growth by modifying adhesion molecule expression
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Vimentin is a novel AKT1 target mediating motility and invasionProliferation, neurogenesis and regeneration in the non-mammalian vertebrate brainNestin expression in the cell lines derived from glioblastoma multiforme.Axonal plasticity and functional recovery after spinal cord injury in mice deficient in both glial fibrillary acidic protein and vimentin genes.Development of NMDAR antagonists with reduced neurotoxic side effects: a study on GK11Characterization of adult rat astrocyte culturesPhotomechanical wave-driven delivery of siRNAs targeting intermediate filament proteins promotes functional recovery after spinal cord injury in ratsThe effect of glial fibrillary acidic protein expression on neurite outgrowth from retinal explants in a permissive environment.A novel and efficient gene transfer strategy reduces glial reactivity and improves neuronal survival and axonal growth in vitro.Identification of barriers to retinal engraftment of transplanted stem cells.Proliferative reactive gliosis is compatible with glial metabolic support and neuronal function.Finding degrees of separation: experimental approaches for astroglial and oligodendroglial cell isolation and genetic targetingCNF1 improves astrocytic ability to support neuronal growth and differentiation in vitro.The 14-3-3 protein epsilon isoform expressed in reactive astrocytes in demyelinating lesions of multiple sclerosis binds to vimentin and glial fibrillary acidic protein in cultured human astrocytesGlial and perivascular structures in the subfornical organ: distinguishing the shell and core.The methyl binding domain 3/nucleosome remodelling and deacetylase complex regulates neural cell fate determination and terminal differentiation in the cerebral cortex.Extracellular vimentin interacts with insulin-like growth factor 1 receptor to promote axonal growth.eIF5A1/RhoGDIα pathway: a novel therapeutic target for treatment of spinal cord injury identified by a proteomics approach.Marrow stromal cell transplantation in stroke and traumatic brain injuryA Promising Tool in Retina Regeneration: Current Perspectives and Challenges When Using Mesenchymal Progenitor Stem Cells in Veterinary and Human Ophthalmological Applications.Treadmill exercise induces hippocampal astroglial alterations in ratsHydrocortisone stimulates neurite outgrowth from mouse retinal explants by modulating macroglial activityPharmacologic inhibition of reactive gliosis blocks TNF-α-mediated neuronal apoptosis.The role of astrocytes in mediating exogenous cell-based restorative therapy for strokeLLDT-67 attenuates MPTP-induced neurotoxicity in mice by up-regulating NGF expression.Astrocyte hepcidin is a key factor in LPS-induced neuronal apoptosis.The E. coli CNF1 as a pioneering therapy for the central nervous system diseases.Role of GFAP in CNS injuries.In Silico Prediction and Validation of Gfap as an miR-3099 Target in Mouse Brain.Catenin delta-1 (CTNND1) phosphorylation controls the mesenchymal to epithelial transition in astrocytic tumors.A novel compound, denosomin, ameliorates spinal cord injury via axonal growth associated with astrocyte-secreted vimentin.Molecular Mechanisms Mediating Retinal Reactive Gliosis Following Bone Marrow Mesenchymal Stem Cell Transplantation.Multifunctionalized electrospun silk fibers promote axon regeneration in central nervous system.A robust model for simultaneously inducing corneal neovascularization and retinal gliosis in the mouse eye.Bone marrow stromal cells reduce ischemia-induced astrocytic activation in vitro.Müller glia reactivity follows retinal injury despite the absence of the glial fibrillary acidic protein gene in Xenopus.RCCS bioreactor-based modelled microgravity induces significant changes on in vitro 3D neuroglial cell cultures.
P2860
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P2860
Inactivation of the glial fibrillary acidic protein gene, but not that of vimentin, improves neuronal survival and neurite growth by modifying adhesion molecule expression
description
2001 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2001
@ast
im August 2001 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2001/08/15)
@sk
vědecký článek publikovaný v roce 2001
@cs
wetenschappelijk artikel (gepubliceerd op 2001/08/15)
@nl
наукова стаття, опублікована в серпні 2001
@uk
مقالة علمية (نشرت في 15-8-2001)
@ar
name
Inactivation of the glial fibr ...... g adhesion molecule expression
@ast
Inactivation of the glial fibr ...... g adhesion molecule expression
@en
Inactivation of the glial fibr ...... g adhesion molecule expression
@nl
type
label
Inactivation of the glial fibr ...... g adhesion molecule expression
@ast
Inactivation of the glial fibr ...... g adhesion molecule expression
@en
Inactivation of the glial fibr ...... g adhesion molecule expression
@nl
prefLabel
Inactivation of the glial fibr ...... g adhesion molecule expression
@ast
Inactivation of the glial fibr ...... g adhesion molecule expression
@en
Inactivation of the glial fibr ...... g adhesion molecule expression
@nl
P2093
P921
P3181
P1476
Inactivation of the glial fibr ...... g adhesion molecule expression
@en
P2093
E Colucci-Guyon
M Giménez y Ribotta
P304
P3181
P356
10.1523/JNEUROSCI.21-16-06147.2001
P407
P577
2001-08-15T00:00:00Z