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An oxidative stress mechanism of shikonin in human glioma cells

An oxidative stress mechanism of shikonin in human glioma cells

http://www.wikidata.org/entity/Q28537574

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The evidence for natural therapeutics as potential anti-scarring agents in burn-related scarring Shikonin Isolated from Lithospermum erythrorhizon Downregulates Proinflammatory Mediators in Lipopolysaccharide-Stimulated BV2 Microglial Cells by Suppressing Crosstalk between Reactive Oxygen Species and NF-κB.Synthesis and Biological Evaluation of Lipophilic 1,4-Naphthoquinone Derivatives against Human Cancer Cell Lines.Folate Deficiency Triggered Apoptosis of Synoviocytes: Role of Overproduction of Reactive Oxygen Species Generated via NADPH Oxidase/Mitochondrial Complex II and Calcium Perturbation.Shikonin Suppresses NLRP3 and AIM2 Inflammasomes by Direct Inhibition of Caspase-1.MDA5 complements TLR3 in suppression of neuroblastoma Synergistic antioxidant activity of resveratrol with genistein in high-glucose treated Madin-Darby canine kidney epithelial cells.Mitochondrial Dysfunction in Gliomas: Pharmacotherapeutic Potential of Natural Compounds.One-pot synthesis of 1,4-dihydroxy-2-((E)-1-hydroxy-4-phenylbut-3-enyl)anthracene-9,10-diones as novel shikonin analogs and evaluation of their antiproliferative activities.Expression of Tim4 in Glioma and its Regulatory Role in LN-18 Glioma Cells.Shikonin Inhibits the Migration and Invasion of Human Glioblastoma Cells by Targeting Phosphorylated β-Catenin and Phosphorylated PI3K/Akt: A Potential Mechanism for the Anti-Glioma Efficacy of a Traditional Chinese Herbal Medicine Shikonin selectively induces apoptosis in human prostate cancer cells through the endoplasmic reticulum stress and mitochondrial apoptotic pathway.Lycorine induces programmed necrosis in the multiple myeloma cell line ARH-77.Propyl Gallate Exerts an Antimigration Effect on Temozolomide-Treated Malignant Glioma Cells through Inhibition of ROS and the NF-κB Pathway.Shikonin induces glioma cell necroptosis in vitro by ROS overproduction and promoting RIP1/RIP3 necrosome formation.Pyruvate kinase isozyme M2 and glutaminase might be promising molecular targets for the treatment of gastric cancer.Reprogramming of leukemic cell metabolism through the naphthoquinonic compound Quambalarine B.Yellow and green pigments from Calophyllum inophyllum L. seed oil induce cell death in colon and lung cancer cells.

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An oxidative stress mechanism of shikonin in human glioma cells

http://www.wikidata.org/entity/Q28537574

description

2014 nî lūn-bûn

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2014 թուականին հրատարակուած գիտական յօդուած

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2014 թվականին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

An oxidative stress mechanism of shikonin in human glioma cells

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An oxidative stress mechanism of shikonin in human glioma cells

@en

An oxidative stress mechanism of shikonin in human glioma cells

@nl

type

label

An oxidative stress mechanism of shikonin in human glioma cells

@ast

An oxidative stress mechanism of shikonin in human glioma cells

@en

An oxidative stress mechanism of shikonin in human glioma cells

@nl

prefLabel

An oxidative stress mechanism of shikonin in human glioma cells

@ast

An oxidative stress mechanism of shikonin in human glioma cells

@en

An oxidative stress mechanism of shikonin in human glioma cells

@nl

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An oxidative stress mechanism of shikonin in human glioma cells

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Ching-Hsein Chen

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Fung-Jou Lu

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Jen-Tsung Yang

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Jin-Yi Wu

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Zih-Ling Li

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P2860

Reactive oxygen species: role in the development of cancer and various chronic conditions

Targeting NADPH oxidases in vascular pharmacology

Reactive oxygen species (ROS) homeostasis and redox regulation in cellular signaling

Bid-induced mitochondrial membrane permeabilization waves propagated by local reactive oxygen species (ROS) signaling.

Reactive oxygen species in cancer cells: live by the sword, die by the sword.

Interaction of the mitochondria-targeted antioxidant MitoQ with phospholipid bilayers and ubiquinone oxidoreductases.

Oxidative stress and apoptosis: impact on cancer therapy.

Reactive oxygen species-dependent activation of Bax and poly(ADP-ribose) polymerase-1 is required for mitochondrial cell death induced by triterpenoid pristimerin in human cervical cancer cells.

NADPH oxidase biology and the regulation of tyrosine kinase receptor signaling and cancer drug cytotoxicity

Fisetin inhibits growth, induces G₂ /M arrest and apoptosis of human epidermoid carcinoma A431 cells: role of mitochondrial membrane potential disruption and consequent caspases activation.

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