Growth factor and Th2 cytokine signaling pathways converge at STAT6 to promote arginase expression in progressive experimental visceral leishmaniasis

Growth factor and Th2 cytokine signaling pathways converge at STAT6 to promote arginase expression in progressive experimental visceral leishmaniasis

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http://www.wikidata.org/entity/Q28540097
Characterization of microRNA expression profiles in Leishmania-infected human phagocytes.Cross talk between histone deacetylase 4 and STAT6 in the transcriptional regulation of arginase 1 during mouse dendritic cell differentiation.STAT6-Dependent Collagen Synthesis in Human Fibroblasts Is Induced by Bovine Milk.M2 Polarization of Monocytes-Macrophages Is a Hallmark of Indian Post Kala-Azar Dermal Leishmaniasis.Splenic CD4+ T Cells in Progressive Visceral Leishmaniasis Show a Mixed Effector-Regulatory Phenotype and Impair Macrophage Effector Function through Inhibitory Receptor Expression.Transcriptional Profiling in Experimental Visceral Leishmaniasis Reveals a Broad Splenic Inflammatory Environment that Conditions Macrophages toward a Disease-Promoting Phenotype.Mycobacterium leprae-induced Insulin-like Growth Factor I attenuates antimicrobial mechanisms, promoting bacterial survival in macrophages.Therapeutic effect of ursolic acid in experimental visceral leishmaniasisThe function of cancer-shed gangliosides in macrophage phenotype: involvement with angiogenesis.Metabolism via Arginase or Nitric Oxide Synthase: Two Competing Arginine Pathways in MacrophagesIn vivo characterization of two additional Leishmania donovani strains using the murine and hamster model.Leishmania major Infection-Induced VEGF-A/VEGFR-2 Signaling Promotes Lymphangiogenesis That Controls Disease.M1 polarization and the effect of PGE2 on TNF-α production by lymph node cells from dogs with visceral leishmaniasis.Leishmania proteophosphoglycans regurgitated from infected sand flies accelerate dermal wound repair and exacerbate leishmaniasis via insulin-like growth factor 1-dependent signalling.Insulin-Like Growth Factor-I as an Effector Element of the Cytokine IL-4 in the Development of a Infection
P2860

Growth factor and Th2 cytokine signaling pathways converge at STAT6 to promote arginase expression in progressive experimental visceral leishmaniasis

Item
http://www.wikidata.org/entity/Q28540097
description
2014 nî lūn-bûn
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2014 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Growth factor and Th2 cytokine ...... imental visceral leishmaniasis
@ast
Growth factor and Th2 cytokine ...... imental visceral leishmaniasis
@en
Growth factor and Th2 cytokine ...... imental visceral leishmaniasis
@nl
type
label
Growth factor and Th2 cytokine ...... imental visceral leishmaniasis
@ast
Growth factor and Th2 cytokine ...... imental visceral leishmaniasis
@en
Growth factor and Th2 cytokine ...... imental visceral leishmaniasis
@nl
prefLabel
Growth factor and Th2 cytokine ...... imental visceral leishmaniasis
@ast
Growth factor and Th2 cytokine ...... imental visceral leishmaniasis
@en
Growth factor and Th2 cytokine ...... imental visceral leishmaniasis
@nl
P1433
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Growth factor and Th2 cytokine ...... imental visceral leishmaniasis
@en
P2093
P2860
P304
P31
P3181
P356
10.1371/JOURNAL.PPAT.1004165
P407
P433
P478
P577
P5875
P698
P921
P932