DMH1 increases glucose metabolism through activating Akt in L6 rat skeletal muscle cells

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DMH1 (4-[6-(4-isopropoxyphenyl)pyrazolo[1,5-a]pyrimidin-3-yl]quinoline) inhibits chemotherapeutic drug-induced autophagy.Effects of Acute Cold Stress on Liver -GlcNAcylation and Glycometabolism in Mice

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DMH1 increases glucose metabolism through activating Akt in L6 rat skeletal muscle cells

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2014 թուականին հրատարակուած գիտական յօդուած

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DMH1 increases glucose metabolism through activating Akt in L6 rat skeletal muscle cells

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DMH1 increases glucose metabolism through activating Akt in L6 rat skeletal muscle cells

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DMH1 increases glucose metabolism through activating Akt in L6 rat skeletal muscle cells

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DMH1 increases glucose metabolism through activating Akt in L6 rat skeletal muscle cells

@ast

DMH1 increases glucose metabolism through activating Akt in L6 rat skeletal muscle cells

@en

DMH1 increases glucose metabolism through activating Akt in L6 rat skeletal muscle cells

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DMH1 increases glucose metabolism through activating Akt in L6 rat skeletal muscle cells

@ast

DMH1 increases glucose metabolism through activating Akt in L6 rat skeletal muscle cells

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DMH1 increases glucose metabolism through activating Akt in L6 rat skeletal muscle cells

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DMH1 increases glucose metabolism through activating Akt in L6 rat skeletal muscle cells

@en

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Chun-Yan Ma

http://www.w3.org/2001/XMLSchema#string

De-Li Dong

http://www.w3.org/2001/XMLSchema#string

Na Li

http://www.w3.org/2001/XMLSchema#string

Xiao-Ming Xu

http://www.w3.org/2001/XMLSchema#string

Xin Xie

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Yong-Hui Zhang

http://www.w3.org/2001/XMLSchema#string

Yu Zhao

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Mutation of a nuclear succinate dehydrogenase gene results in mitochondrial respiratory chain deficiency

Protein phosphatase 2A: a highly regulated family of serine/threonine phosphatases implicated in cell growth and signalling

Free fatty acid-induced PP2A hyperactivity selectively impairs hepatic insulin action on glucose metabolism

AMPK inhibition blocks ROS-NFκB signaling and attenuates endotoxemia-induced liver injury

Bone morphogenetic protein-4 contributes to the down-regulation of Kv4.3 K+ channels in pathological cardiac hypertrophy

AMP-activated protein kinase: ancient energy gauge provides clues to modern understanding of metabolism

Regulation of protein kinase cascades by protein phosphatase 2A.

Management of cellular energy by the AMP-activated protein kinase system.

DMH1, a novel BMP small molecule inhibitor, increases cardiomyocyte progenitors and promotes cardiac differentiation in mouse embryonic stem cells.

(4-[6-(4-isopropoxyphenyl)pyrazolo [1,5-a]pyrimidin-3-yl] quinoline) is a novel inhibitor of autophagy

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e107776

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scholarly article

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10.1371/JOURNAL.PONE.0107776

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English

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2014-01-01T00:00:00Z

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266027063

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25247550

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DMH1

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4172596

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DMH1 increases glucose metabolism through activating Akt in L6 rat skeletal muscle cells

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DMH1 increases glucose metabolism through activating Akt in L6 rat skeletal muscle cells

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P1433

P1476

P2093

P2860

P304

P31

P356

P407

P433

P478

P577

P5875

P698

P921

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P407

P433

P478

P577

P5875

P698

P921

P932