Central presynaptic terminals are enriched in ATP but the majority lack mitochondria
about
Structural and Genetic Studies Demonstrate Neurologic Dysfunction in Triosephosphate Isomerase Deficiency Is Associated with Impaired Synaptic Vesicle DynamicsX-linked intellectual disability gene CASK regulates postnatal brain growth in a non-cell autonomous manner.Glycolytic Enzymes Localize to Synapses under Energy Stress to Support Synaptic FunctionBioenergetic status modulates motor neuron vulnerability and pathogenesis in a zebrafish model of spinal muscular atrophy.A validated active contour method driven by parabolic arc model for detection and segmentation of mitochondria.Neuronal acid-induced [Zn²⁺]i elevations calibrated using the low-affinity ratiometric probe FuraZin-1.Analysis of Brain Mitochondria Using Serial Block-Face Scanning Electron Microscopy.Integration between Glycolysis and Glutamate-Glutamine Cycle Flux May Explain Preferential Glycolytic Increase during Brain Activation, Requiring Glutamate.Mitochondrial support of persistent presynaptic vesicle mobilization with age-dependent synaptic growth after LTP.CASK stabilizes neurexin and links it to liprin-α in a neuronal activity-dependent manner.Differential Presynaptic ATP Supply for Basal and High-Demand Transmission.Correction: Central Presynaptic Terminals Are Enriched in ATP but the Majority Lack Mitochondria.Sleep Loss Promotes Astrocytic Phagocytosis and Microglial Activation in Mouse Cerebral Cortex.Brain energetics during the sleep-wake cycle.Additional mechanisms for brain activation failure due to reduced glucose metabolism-a commentary on Zilberter and Zilberter: The vicious circle of hypometabolism in neurodegenerative diseases.Unraveling Synaptic GCaMP Signals: Differential Excitability and Clearance Mechanisms Underlying Distinct Ca2+ Dynamics in Tonic and Phasic Excitatory, and Aminergic Modulatory Motor Terminals in Drosophila.Neurotransmitter Diversity In Pre-Synaptic Terminals Located in the Parvicellular Neuroendocrine Paraventricular Nucleus of the Rat and Mouse Hypothalamus.
P2860
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P2860
Central presynaptic terminals are enriched in ATP but the majority lack mitochondria
description
2015 nî lūn-bûn
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2015 թուականին հրատարակուած գիտական յօդուած
@hyw
2015 թվականին հրատարակված գիտական հոդված
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年论文
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name
Central presynaptic terminals are enriched in ATP but the majority lack mitochondria
@ast
Central presynaptic terminals are enriched in ATP but the majority lack mitochondria
@en
Central presynaptic terminals are enriched in ATP but the majority lack mitochondria
@nl
type
label
Central presynaptic terminals are enriched in ATP but the majority lack mitochondria
@ast
Central presynaptic terminals are enriched in ATP but the majority lack mitochondria
@en
Central presynaptic terminals are enriched in ATP but the majority lack mitochondria
@nl
prefLabel
Central presynaptic terminals are enriched in ATP but the majority lack mitochondria
@ast
Central presynaptic terminals are enriched in ATP but the majority lack mitochondria
@en
Central presynaptic terminals are enriched in ATP but the majority lack mitochondria
@nl
P2093
P2860
P3181
P1433
P1476
Central presynaptic terminals are enriched in ATP but the majority lack mitochondria
@en
P2093
Helen R Clark
Jeffery Willis
Konark Mukherjee
Sarika Srivastava
Sidney K Walker
Vrushali Chavan
Xinran Liu
P2860
P304
P3181
P356
10.1371/JOURNAL.PONE.0125185
P407
P577
2015-01-01T00:00:00Z