Antioxidant Treatment and Induction of Autophagy Cooperate to Reduce Desmin Aggregation in a Cellular Model of Desminopathy - Wikidata - awgldk - TriplyDB

Antioxidant Treatment and Induction of Autophagy Cooperate to Reduce Desmin Aggregation in a Cellular Model of Desminopathy

Antioxidant Treatment and Induction of Autophagy Cooperate to Reduce Desmin Aggregation in a Cellular Model of Desminopathy

http://www.wikidata.org/entity/Q28547713

about

Disrupted autophagy undermines skeletal muscle adaptation and integrity "Get the Balance Right": Pathological Significance of Autophagy Perturbation in Neuromuscular Disorders Myofibrillar Myopathies: New Perspectives from Animal Models to Potential Therapeutic Approaches.Redox Control of Skeletal Muscle Regeneration.Desmin and αB-crystallin interplay in the maintenance of mitochondrial homeostasis and cardiomyocyte survival.The role of αB-crystallin in skeletal and cardiac muscle tissues.Sevoflurane Ameliorates Myocardial Cell Injury by Inducing Autophagy via the Deacetylation of LC3 by SIRT1.Molecular insights into cardiomyopathies associated with desmin (DES) mutations

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Antioxidant Treatment and Induction of Autophagy Cooperate to Reduce Desmin Aggregation in a Cellular Model of Desminopathy

http://www.wikidata.org/entity/Q28547713

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2015 nî lūn-bûn

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2015 թուականին հրատարակուած գիտական յօդուած

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2015 թվականին հրատարակված գիտական հոդված

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2015年の論文

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年学术文章

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2015年學術文章

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Antioxidant Treatment and Indu ...... Cellular Model of Desminopathy

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Blandine Gausserès

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Eva Cabet

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Florence Delort

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Patrick Vicart

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Sabrina Batonnet-Pichon

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P2860

Guidelines for the use and interpretation of assays for monitoring autophagy in higher eukaryotes

Structural and functional roles of desmin in mouse skeletal muscle during passive deformation.

Immunological characterization of the subunit of the 100 A filaments from muscle cells

A brain serine/threonine protein kinase activated by Cdc42 and Rac1

Myofibrillar myopathies

N-acetyl-L-cysteine prevents stress-induced desmin aggregation in cellular models of desminopathy

Pak1 kinase homodimers are autoinhibited in trans and dissociated upon activation by Cdc42 and Rac1

Methods in mammalian autophagy research

Myofibrillar myopathies: new developments.

The ubiquitin-proteasome system in cardiac proteinopathy: a quality control perspective.

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Alain Lilienbaum

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