SALL4 as an Epithelial-Mesenchymal Transition and Drug Resistance Inducer through the Regulation of c-Myc in Endometrial Cancer
about
SALL4, the missing link between stem cells, development and cancerOncofetal gene SALL4 and prognosis in cancer: A systematic review with meta-analysis.Epithelial-to-Mesenchymal Transition in the Female Reproductive Tract: From Normal Functioning to Disease Pathology.Knockdown of SALL4 inhibits the proliferation and reverses the resistance of MCF-7/ADR cells to doxorubicin hydrochloride.SALL4 promotes gastric cancer progression through activating CD44 expressionJQ1 suppresses tumor growth via PTEN/PI3K/AKT pathway in endometrial cancer.High expression of SALL4 and fascin, and loss of E-cadherin expression in undifferentiated/dedifferentiated carcinomas of the endometrium: An immunohistochemical and clinicopathologic study.Aberrantly Expressed SALL4 Promotes Cell Proliferation via β-Catenin/c-Myc Pathway in Human Choriocarcinoma Cells.Functional and clinical significance of SALL4 in breast cancer.EMT reversal in human cancer cells after IR knockdown in hyperinsulinemic mice.Promoter-level transcriptome in primary lesions of endometrial cancer identified biomarkers associated with lymph node metastasis.Let-7d increases ovarian cancer cell sensitivity to a genistein analog by targeting c-Myc.SALL4 suppresses PTEN expression to promote glioma cell proliferation via PI3K/AKT signaling pathway.SALL4 promotes glycolysis and chromatin remodeling via modulating HP1α-Glut1 pathway.KPNA2 promotes migration and invasion in epithelial ovarian cancer cells by inducing epithelial-mesenchymal transition via Akt/GSK-3β/Snail activation.Higher expression of SALL4 predicts poor cancer prognosis: A meta-analysis.SALL4 oncogene is an immunogenic antigen presented in various HLA-DR contexts.Salidroside inhibits the proliferation and migration of gastric cancer cells via suppression of Src‑associated signaling pathway activation and heat shock protein 70 expression.SALL4 activates TGF-β/SMAD signaling pathway to induce EMT and promote gastric cancer metastasis
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P2860
SALL4 as an Epithelial-Mesenchymal Transition and Drug Resistance Inducer through the Regulation of c-Myc in Endometrial Cancer
description
2015 nî lūn-bûn
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2015 թուականին հրատարակուած գիտական յօդուած
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2015 թվականին հրատարակված գիտական հոդված
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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2015年论文
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name
SALL4 as an Epithelial-Mesench ...... of c-Myc in Endometrial Cancer
@ast
SALL4 as an Epithelial-Mesench ...... of c-Myc in Endometrial Cancer
@en
SALL4 as an Epithelial-Mesench ...... of c-Myc in Endometrial Cancer
@nl
type
label
SALL4 as an Epithelial-Mesench ...... of c-Myc in Endometrial Cancer
@ast
SALL4 as an Epithelial-Mesench ...... of c-Myc in Endometrial Cancer
@en
SALL4 as an Epithelial-Mesench ...... of c-Myc in Endometrial Cancer
@nl
prefLabel
SALL4 as an Epithelial-Mesench ...... of c-Myc in Endometrial Cancer
@ast
SALL4 as an Epithelial-Mesench ...... of c-Myc in Endometrial Cancer
@en
SALL4 as an Epithelial-Mesench ...... of c-Myc in Endometrial Cancer
@nl
P2093
P2860
P1433
P1476
SALL4 as an Epithelial-Mesench ...... of c-Myc in Endometrial Cancer
@en
P2093
P2860
P304
P356
10.1371/JOURNAL.PONE.0138515
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P577
2015-01-01T00:00:00Z