Early lead exposure increases the leakage of the blood-cerebrospinal fluid barrier, in vitro
about
Neurotoxic effects and biomarkers of lead exposure: a reviewTight junction proteins and oxidative stress in heavy metals-induced nephrotoxicityLead-induced accumulation of beta-amyloid in the choroid plexus: role of low density lipoprotein receptor protein-1 and protein kinase CLead exposure increases levels of β-amyloid in the brain and CSF and inhibits LRP1 expression in APP transgenic miceAn in vivo animal study assessing long-term changes in hypothalamic cytokines following perinatal exposure to a chemical mixture based on Arctic maternal body burdenInvolvement of insulin-degrading enzyme in the clearance of beta-amyloid at the blood-CSF barrier: Consequences of lead exposureIncreased beta-amyloid levels in the choroid plexus following lead exposure and the involvement of low-density lipoprotein receptor protein-1The interaction of carbon nanotubes with an in vitro blood-brain barrier model and mouse brain in vivoAcrylamide exposure impairs blood-cerebrospinal fluid barrier functionRegulation of brain copper homeostasis by the brain barrier systems: effects of Fe-overload and Fe-deficiency.Acute stress to excised vocal fold epithelium from reactive oxygen species.Translocation of LRP1 targeted carbon nanotubes of different diameters across the blood-brain barrier in vitro and in vivoUpregulation of zinc transporter 2 in the blood-CSF barrier following lead exposureCulture of choroid plexus epithelial cells and in vitro model of blood-CSF barrier.The distributional nexus of choroid plexus to cerebrospinal fluid, ependyma and brain: toxicologic/pathologic phenomena, periventricular destabilization, and lesion spread.Ototoxicity of Divalent Metals.Malathion/oxon and lead acetate increase gene expression and protein levels of transient receptor potential canonical channel subunits TRPC1 and TRPC4 in rat endothelial cells of the blood-brain barrier.Regional distribution and accumulation of lead in caprine brain tissues following a long-term oral dosing regimen.Neurotoxicity of Metal Mixtures.Gestational lead exposure induces developmental abnormalities and up-regulates apoptosis of fetal cerebellar cells in rats.Beneficial effects of garlic on learning and memory deficits and brain tissue damages induced by lead exposure during juvenile rat growth is comparable to the effect of ascorbic acid.
P2860
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P2860
Early lead exposure increases the leakage of the blood-cerebrospinal fluid barrier, in vitro
description
2007 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի մարտին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2007
@ast
im März 2007 veröffentlichter wissenschaftlicher Artikel
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scientific journal article
@en
vedecký článok (publikovaný 2007/03/01)
@sk
vědecký článek publikovaný v roce 2007
@cs
wetenschappelijk artikel (gepubliceerd op 2007/03/01)
@nl
наукова стаття, опублікована в березні 2007
@uk
مقالة علمية (نشرت في مارس 2007)
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name
Early lead exposure increases ...... spinal fluid barrier, in vitro
@ast
Early lead exposure increases ...... spinal fluid barrier, in vitro
@en
Early lead exposure increases ...... spinal fluid barrier, in vitro
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type
label
Early lead exposure increases ...... spinal fluid barrier, in vitro
@ast
Early lead exposure increases ...... spinal fluid barrier, in vitro
@en
Early lead exposure increases ...... spinal fluid barrier, in vitro
@nl
prefLabel
Early lead exposure increases ...... spinal fluid barrier, in vitro
@ast
Early lead exposure increases ...... spinal fluid barrier, in vitro
@en
Early lead exposure increases ...... spinal fluid barrier, in vitro
@nl
P2860
P356
P1476
Early lead exposure increases ...... spinal fluid barrier, in vitro
@en
P2093
Lewis Zhichang Shi
P2860
P304
P356
10.1177/0960327107070560
P577
2007-03-01T00:00:00Z