TGF-beta1 and integrin synergistically facilitate the differentiation of rat podocytes by increasing alpha-smooth muscle actin expression

TGF-beta1 and integrin synergistically facilitate the differentiation of rat podocytes by increasing alpha-smooth muscle actin expression

Item
http://www.wikidata.org/entity/Q28566545
Fate-determining mechanisms in epithelial-myofibroblast transition: major inhibitory role for Smad3The role of retinoic acid receptors in the signal pathway of all-trans retinoic acid-induced differentiation in adriamycin-induced podocyte injury.Transforming growth factor-beta stimulates intestinal epithelial focal adhesion kinase synthesis via Smad- and p38-dependent mechanisms.Expression and localization of Ski determine cell type-specific TGFbeta signaling effects on the cell cycleEarly protective role of MST1 knockdown in response to experimental diabetic nephropathyA Decrease in Glomerular Endothelial Cells and Endothelial-mesenchymal Transition during Glomerulosclerosis in the Tensin2-deficient Mice (ICGN strain).Lack of α8 integrin leads to morphological changes in renal mesangial cells, but not in vascular smooth muscle cells.Constitutional Nephrin Deficiency in Conditionally Immortalized Human Podocytes Induced Epithelial-Mesenchymal Transition, Supported by β-Catenin/NF-kappa B Activation: A Consequence of Cell Junction Impairment?C5b-9 glomerular deposition and tubular alpha3beta1-integrin expression are implicated in the development of chronic lesions and predict renal function outcome in immunoglobulin A nephropathy.Albumin overload down-regulates integrin-β1 through reactive oxygen species-endoplasmic reticulum stress pathway in podocytes.Negative regulation of inducible nitric-oxide synthase expression mediated through transforming growth factor-beta-dependent modulation of transcription factor TCF11.Acute kidney injury and progression of renal failure after fetal programming in the offspring of diabetic rats.Cyclin D1 expression in podocytes: regulated by mitogens in collaboration with integrin-extracellular matrix interaction through extracellular signal-regulated kinase.
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TGF-beta1 and integrin synergistically facilitate the differentiation of rat podocytes by increasing alpha-smooth muscle actin expression

Item
http://www.wikidata.org/entity/Q28566545
description
2006 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2006
@ast
scientific journal article
@en
vedecký článok (publikovaný 2006/09/01)
@sk
vědecký článek publikovaný v roce 2006
@cs
wetenschappelijk artikel (gepubliceerd op 2006/09/01)
@nl
наукова стаття, опублікована у вересні 2006
@uk
مقالة علمية (نشرت في سبتمبر 2006)
@ar
name
TGF-beta1 and integrin synergi ...... smooth muscle actin expression
@ast
TGF-beta1 and integrin synergi ...... smooth muscle actin expression
@en
TGF-beta1 and integrin synergi ...... smooth muscle actin expression
@nl
type
label
TGF-beta1 and integrin synergi ...... smooth muscle actin expression
@ast
TGF-beta1 and integrin synergi ...... smooth muscle actin expression
@en
TGF-beta1 and integrin synergi ...... smooth muscle actin expression
@nl
prefLabel
TGF-beta1 and integrin synergi ...... smooth muscle actin expression
@ast
TGF-beta1 and integrin synergi ...... smooth muscle actin expression
@en
TGF-beta1 and integrin synergi ...... smooth muscle actin expression
@nl
P1433
P1476
P2093
P304
P31
P356
P433
P478
P577
P698
P356
P1433
P1476
TGF-beta1 and integrin synergi ...... smooth muscle actin expression
@en
P2093
P304
P31
P356
10.1016/J.TRSL.2006.03.008
P433
P478
P577
P698