Sildenafil inhibits chronically hypoxic upregulation of canonical transient receptor potential expression in rat pulmonary arterial smooth muscle
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Proteomic analysis reveals that proteasome subunit beta 6 is involved in hypoxia-induced pulmonary vascular remodeling in ratsBone morphogenetic protein 4 enhances canonical transient receptor potential expression, store-operated Ca2+ entry, and basal [Ca2+]i in rat distal pulmonary arterial smooth muscle cellsNoggin inhibits hypoxia-induced proliferation by targeting store-operated calcium entry and transient receptor potential cation channels.Phosphodiesterase 5 inhibition ameliorates angiontensin II-induced podocyte dysmotility via the protein kinase G-mediated downregulation of TRPC6 activity.The Role of Transient Receptor Potential Channel 6 Channels in the Pulmonary VasculatureClassical transient receptor potential 1 and 6 contribute to hypoxic pulmonary hypertension through differential regulation of pulmonary vascular functions.Protein kinases modulate store-operated channels in pulmonary artery smooth muscle cells.New perspectives for the treatment of pulmonary hypertensionEffects of chronic exposure to cigarette smoke on canonical transient receptor potential expression in rat pulmonary arterial smooth muscle.Peroxisome proliferator-activated receptor γ inhibits pulmonary hypertension targeting store-operated calcium entryChronic hypoxia increases TRPC6 expression and basal intracellular Ca2+ concentration in rat distal pulmonary venous smooth muscleBMP4 increases the expression of TRPC and basal [Ca2+]i via the p38MAPK and ERK1/2 pathways independent of BMPRII in PASMCsUpregulation of canonical transient receptor potential channel in the pulmonary arterial smooth muscle of a chronic thromboembolic pulmonary hypertension rat model.Upregulation of osmo-mechanosensitive TRPV4 channel facilitates chronic hypoxia-induced myogenic tone and pulmonary hypertensionEnhanced store-operated Ca²+ entry and TRPC channel expression in pulmonary arteries of monocrotaline-induced pulmonary hypertensive rats.Hypoxia inducible factor-1-dependent up-regulation of BMP4 mediates hypoxia-induced increase of TRPC expression in PASMCs.Pathogenic role of store-operated and receptor-operated ca(2+) channels in pulmonary arterial hypertension.Sodium tanshinone IIA sulfonate inhibits canonical transient receptor potential expression in pulmonary arterial smooth muscle from pulmonary hypertensive rats.Vascular remodeling in pulmonary hypertension.Bone morphogenetic protein 2 decreases TRPC expression, store-operated Ca(2+) entry, and basal [Ca(2+)]i in rat distal pulmonary arterial smooth muscle cells.Sodium tanshinone IIA sulfonate inhibits hypoxia-induced enhancement of SOCE in pulmonary arterial smooth muscle cells via the PKG-PPAR-γ signaling axisAcute hypoxia activates store-operated Ca(2+) entry and increases intracellular Ca(2+) concentration in rat distal pulmonary venous smooth muscle cellsBMP4 increases canonical transient receptor potential protein expression by activating p38 MAPK and ERK1/2 signaling pathways in pulmonary arterial smooth muscle cellsSildenafil inhibits hypoxia-induced transient receptor potential canonical protein expression in pulmonary arterial smooth muscle via cGMP-PKG-PPARγ axisCanonical Transient Receptor Potential Channels and Their Link with Cardio/Cerebro-Vascular Diseases.Inhibition of phosphodiesterase-5 suppresses calcineurin/NFAT- mediated TRPC6 expression in pulmonary artery smooth muscle cellsChloroquine is a potent pulmonary vasodilator that attenuates hypoxia-induced pulmonary hypertension.Bortezomib alleviates experimental pulmonary hypertension by regulating intracellular calcium homeostasis in PASMCsHydrogen peroxide is a critical regulator of the hypoxia-induced alterations of store-operated Ca2+ entry into rat pulmonary arterial smooth muscle cells.Isolation, culture and identification of pulmonary arterial smooth muscle cells from rat distal pulmonary arteries.Role of thromboxane A₂-activated nonselective cation channels in hypoxic pulmonary vasoconstriction of rat.Sildenafil Prevents Podocyte Injury via PPAR-γ-Mediated TRPC6 Inhibition.NSFC spurs significant basic research progress of respiratory medicine in China.YM155, a selective survivin inhibitor, reverses chronic hypoxic pulmonary hypertension in rats via upregulating voltage-gated potassium channels.Development of a new method for the isolation and culture of pulmonary arterial endothelial cells from rat pulmonary arteries.
P2860
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P2860
Sildenafil inhibits chronically hypoxic upregulation of canonical transient receptor potential expression in rat pulmonary arterial smooth muscle
description
2010 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հունվարին հրատարակված գիտական հոդված
@hy
artikull shkencor
@sq
artículu científicu espublizáu en 2010
@ast
im Januar 2010 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
wetenschappelijk artikel
@nl
наукова стаття, опублікована в січні 2010
@uk
مقالة علمية (نشرت عام 2010)
@ar
name
Sildenafil inhibits chronicall ...... lmonary arterial smooth muscle
@ast
Sildenafil inhibits chronicall ...... lmonary arterial smooth muscle
@en
Sildenafil inhibits chronicall ...... lmonary arterial smooth muscle
@nl
type
label
Sildenafil inhibits chronicall ...... lmonary arterial smooth muscle
@ast
Sildenafil inhibits chronicall ...... lmonary arterial smooth muscle
@en
Sildenafil inhibits chronicall ...... lmonary arterial smooth muscle
@nl
prefLabel
Sildenafil inhibits chronicall ...... lmonary arterial smooth muscle
@ast
Sildenafil inhibits chronicall ...... lmonary arterial smooth muscle
@en
Sildenafil inhibits chronicall ...... lmonary arterial smooth muscle
@nl
P2093
P2860
P1476
Sildenafil inhibits chronicall ...... lmonary arterial smooth muscle
@en
P2093
Dandan Zhang
Gongyong Peng
Nanshan Zhong
P2860
P304
P356
10.1152/AJPCELL.00629.2008
P577
2010-01-01T00:00:00Z