Tubular kidney injury molecule-1 in protein-overload nephropathy - Wikidata - awgldk - TriplyDB

Tubular kidney injury molecule-1 in protein-overload nephropathy

Tubular kidney injury molecule-1 in protein-overload nephropathy

http://www.wikidata.org/entity/Q28572259

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Emerging biomarkers and metabolomics for assessing toxic nephropathy and acute kidney injury (AKI) in neonatology Increased susceptibility of aging kidney to ischemic injury: identification of candidate genes changed during aging, but corrected by caloric restriction Tissue Kim-1 and urinary clusterin as early indicators of cisplatin-induced acute kidney injury in rats Kidney injury molecule-1 is an early noninvasive indicator for donor brain death-induced injury prior to kidney transplantation Age-related differences in susceptibility to cisplatin-induced renal toxicity.The metabolic profile of a rat model of chronic kidney disease.Identification of tubular heparan sulfate as a docking platform for the alternative complement component properdin in proteinuric renal disease Kidney injury molecule-1 outperforms traditional biomarkers of kidney injury in preclinical biomarker qualification studies.The effect of progressive glomerular disease on megalin-mediated endocytosis in the kidney Kidney injury molecule-1 expression in transplant biopsies is a sensitive measure of cell injury.Blood kidney injury molecule-1 is a biomarker of acute and chronic kidney injury and predicts progression to ESRD in type I diabetes Factor h and properdin recognize different epitopes on renal tubular epithelial heparan sulfate.Fenofibrate attenuates tubulointerstitial fibrosis and inflammation through suppression of nuclear factor-κB and transforming growth factor-β1/Smad3 in diabetic nephropathy.Effect of treatment on urinary kidney injury molecule-1 in IgA nephropathy Glomerular expression of kidney injury molecule-1 and podocytopenia in diabetic glomerulopathy.Effect of renin-angiotensin-aldosterone system inhibition, dietary sodium restriction, and/or diuretics on urinary kidney injury molecule 1 excretion in nondiabetic proteinuric kidney disease: a post hoc analysis of a randomized controlled trial Effects of long-acting erythropoietin analog darbepoetin-α on adriamycin-induced chronic nephropathy.Desferrithiocin analogue iron chelators: iron clearing efficiency, tissue distribution, and renal toxicity Early Markers of Tubulointerstitial Fibrosis in Children With Idiopathic Nephrotic Syndrome: Preliminary Report.Human Alpha-1-Antitrypsin (hAAT) therapy reduces renal dysfunction and acute tubular necrosis in a murine model of bilateral kidney ischemia-reperfusion injury.Mechanistic biomarkers for cytotoxic acute kidney injury.Peritubular ischemia contributes more to tubular damage than proteinuria in immune-mediated glomerulonephritis.Cell therapy with human renal cell cultures containing erythropoietin-positive cells improves chronic kidney injury.Differential effects of sex steroids in young and aged female mRen2.Lewis rats: a model of estrogen and salt-sensitive hypertension.3-MCPD 1-Palmitate Induced Tubular Cell Apoptosis In Vivo via JNK/p53 Pathways.Mammalian Target of Rapamycin Mediates Kidney Injury Molecule 1-Dependent Tubule Injury in a Surrogate Model.Chronic epithelial kidney injury molecule-1 expression causes murine kidney fibrosis.Reduction of proteinuria in adriamycin-induced nephropathy is associated with reduction of renal kidney injury molecule (Kim-1) over time Kidney Injury Molecule-1 (KIM-1): a specific and sensitive biomarker of kidney injury.Wnt4 is a novel biomarker for the early detection of kidney tubular injury after ischemia/reperfusion injury A rapid urine test for early detection of kidney injury.Comparison of kidney injury molecule-1 and other nephrotoxicity biomarkers in urine and kidney following acute exposure to gentamicin, mercury, and chromium.High urinary excretion of kidney injury molecule-1 is an independent predictor of graft loss in renal transplant recipients.Kidney injury molecule-1 is an early biomarker of cadmium nephrotoxicity Renal dysfunction in acute heart failure: epidemiology, mechanisms and assessment.Review and discussion of tubular biomarkers in the diagnosis and management of diabetic nephropathy.Kidney injury molecule-1: more than just an injury marker of tubular epithelial cells?The effect of antiepileptic drugs on the kidney function and structure.Urinary biomarkers in the early detection and follow-up of tubular injury in childhood urolithiasis.Renal neutrophil gelatinase-associated lipocalin and kidney injury molecule-1 expression in children with acute kidney injury and Henoch-Schönlein purpura nephritis.

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P2860

Tubular kidney injury molecule-1 in protein-overload nephropathy

http://www.wikidata.org/entity/Q28572259

description

2006 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2006 թվականի օգոստոսին հրատարակված գիտական հոդված

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artículu científicu espublizáu en 2006

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im August 2006 veröffentlichter wissenschaftlicher Artikel

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scientific journal article

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vedecký článok (publikovaný 2006/08/01)

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vědecký článek publikovaný v roce 2006

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wetenschappelijk artikel (gepubliceerd op 2006/08/01)

@nl

наукова стаття, опублікована в серпні 2006

@uk

مقالة علمية (نشرت في أغسطس 2006)

@ar

name

Tubular kidney injury molecule-1 in protein-overload nephropathy

@ast

Tubular kidney injury molecule-1 in protein-overload nephropathy

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Tubular kidney injury molecule-1 in protein-overload nephropathy

@nl

type

label

Tubular kidney injury molecule-1 in protein-overload nephropathy

@ast

Tubular kidney injury molecule-1 in protein-overload nephropathy

@en

Tubular kidney injury molecule-1 in protein-overload nephropathy

@nl

prefLabel

Tubular kidney injury molecule-1 in protein-overload nephropathy

@ast

Tubular kidney injury molecule-1 in protein-overload nephropathy

@en

Tubular kidney injury molecule-1 in protein-overload nephropathy

@nl

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American Journal of Physiology - Renal Physiology

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Tubular kidney injury molecule-1 in protein-overload nephropathy

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Coen A. Stegeman

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Jeffrey Damman

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Joseph V. Bonventre

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Mirjan M. van Timmeren

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Stephan J. L. Bakker

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Theo A. Schuurs

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Veronique Bailly

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Vishal S. Vaidya

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F456–464

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scholarly article

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10.1152/AJPRENAL.00403.2005

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2

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