A molecular platform in neurons regulates inflammation after spinal cord injury
about
P2X7-dependent release of interleukin-1beta and nociception in the spinal cord following lipopolysaccharideFrom mechanosensitivity to inflammatory responses: new players in the pathology of glaucomaThe role of inflammasome in Alzheimer's diseaseAcid-sensing ion channel 1a contributes to the effect of extracellular acidosis on NLRP1 inflammasome activation in cortical neuronsGenetic ablation of Pannexin1 protects retinal neurons from ischemic injuryEmerging Roles for the Immune System in Traumatic Brain InjuryThe adaptor ASC has extracellular and 'prionoid' activities that propagate inflammationThe nucleic acid-sensing inflammasomesTherapeutic neutralization of the NLRP1 inflammasome reduces the innate immune response and improves histopathology after traumatic brain injuryThe pannexin 1 channel activates the inflammasome in neurons and astrocytesNeurogenic Inflammation – The Peripheral Nervous System’s Role in Host Defense and ImmunopathologyThe far-reaching scope of neuroinflammation after traumatic brain injury.Dealing with Danger in the CNS: The Response of the Immune System to Injury.Forelimb locomotor assessment scale (FLAS): novel assessment of forelimb dysfunction after cervical spinal cord injury.Rapid inflammasome activation in microglia contributes to brain disease in HIV/AIDSAmyloid-β induces NLRP1-dependent neuronal pyroptosis in models of Alzheimer's disease.Beneficial effects of secretory leukocyte protease inhibitor after spinal cord injury.Susceptibility to anthrax lethal toxin-induced rat death is controlled by a single chromosome 10 locus that includes rNlrp1Constitutively active inflammasome in human melanoma cells mediating autoinflammation via caspase-1 processing and secretion of interleukin-1betaPediatric spinal cord injury in infant piglets: description of a new large animal model and review of the literature.Chronic glucocorticoid exposure activates BK-NLRP1 signal involving in hippocampal neuron damage.Heightened inflammasome activation is linked to age-related cognitive impairment in Fischer 344 ratsInterleukin 18 in the CNS.Cerebrospinal fluid inflammatory cytokines and chemokines in naturally occurring canine spinal cord injury.Initiation and progression of axonopathy in experimental autoimmune encephalomyelitis.Lectin-like ox-LDL receptor-1 (LOX-1)-Toll-like receptor 4 (TLR4) interaction and autophagy in CATH.a differentiated cells exposed to angiotensin II.Anti-CD11d monoclonal antibody treatment for rat spinal cord compression injury.Nlrp3-inflammasome activation in non-myeloid-derived cells aggravates diabetic nephropathyP2X4 assembles with P2X7 and pannexin-1 in gingival epithelial cells and modulates ATP-induced reactive oxygen species production and inflammasome activationTNF-Α may mediate inflammasome activation in the absence of bacterial infection in more than one way.Deep tissue injury in development of pressure ulcers: a decrease of inflammasome activation and changes in human skin morphology in response to aging and mechanical load.Genome-wide gene expression profiling of stress response in a spinal cord clip compression injury modelAnti-inflammatory treatments during the chronic phase of spinal cord injury improve locomotor function in adult mice.AIM2 and NLRC4 inflammasomes contribute with ASC to acute brain injury independently of NLRP3.NLRP3 inflammasome contributes to inflammation after intracerebral hemorrhage.Isoflurane/nitrous oxide anesthesia induces increases in NMDA receptor subunit NR2B protein expression in the aged rat brainNLRP3 Inflammasome Is Expressed and Functional in Mouse Brain Microglia but Not in Astrocytes.Astrogliosis involves activation of retinoic acid-inducible gene-like signaling in the innate immune response after spinal cord injury.Neuropathic Pain Phenotype Does Not Involve the NLRP3 Inflammasome and Its End Product Interleukin-1β in the Mice Spared Nerve Injury Model.Activation of the nuclear factor E2-related factor 2/antioxidant response element pathway is neuroprotective after spinal cord injury
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A molecular platform in neurons regulates inflammation after spinal cord injury
description
2008 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի մարտին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2008
@ast
im März 2008 veröffentlichter wissenschaftlicher Artikel
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scientific journal article
@en
vedecký článok (publikovaný 2008/03/26)
@sk
vědecký článek publikovaný v roce 2008
@cs
wetenschappelijk artikel (gepubliceerd op 2008/03/26)
@nl
наукова стаття, опублікована в березні 2008
@uk
مقالة علمية (نشرت في 26-3-2008)
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name
A molecular platform in neurons regulates inflammation after spinal cord injury
@ast
A molecular platform in neurons regulates inflammation after spinal cord injury
@en
A molecular platform in neurons regulates inflammation after spinal cord injury
@nl
type
label
A molecular platform in neurons regulates inflammation after spinal cord injury
@ast
A molecular platform in neurons regulates inflammation after spinal cord injury
@en
A molecular platform in neurons regulates inflammation after spinal cord injury
@nl
prefLabel
A molecular platform in neurons regulates inflammation after spinal cord injury
@ast
A molecular platform in neurons regulates inflammation after spinal cord injury
@en
A molecular platform in neurons regulates inflammation after spinal cord injury
@nl
P2093
P3181
P1476
A molecular platform in neurons regulates inflammation after spinal cord injury
@en
P2093
Alex E Marcillo
George Lotocki
Robert W Keane
W Dalton Dietrich
P304
P3181
P356
10.1523/JNEUROSCI.0157-08.2008
P407
P577
2008-03-26T00:00:00Z