Hypertension produced by placental ischemia in pregnant rats is associated with increased soluble endoglin expression
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Increased plasma soluble endoglin levels as an indicator of cardiovascular alterations in hypertensive and diabetic patientsTherapeutic potential of statins and the induction of heme oxygenase-1 in preeclampsiaThe renal circulation in normal pregnancy and preeclampsia: is there a place for relaxin?Angiotensin receptor agonistic autoantibodies and hypertension: preeclampsia and beyondA Comprehensive Review of Hypertension in PregnancyBioactive factors in uteroplacental and systemic circulation link placental ischemia to generalized vascular dysfunction in hypertensive pregnancy and preeclampsiaMolecular and vascular targets in the pathogenesis and management of the hypertension associated with preeclampsia.Effects of hypoxia-inducible factor-1alpha overexpression in pregnant mice: possible implications for preeclampsia and intrauterine growth restrictionPreeclampsia, of mice and women.Altered matrix metalloproteinase-2 and -9 expression/activity links placental ischemia and anti-angiogenic sFlt-1 to uteroplacental and vascular remodeling and collagen deposition in hypertensive pregnancy.Recombinant vascular endothelial growth factor 121 infusion lowers blood pressure and improves renal function in rats with placentalischemia-induced hypertension.Endoglin in liver fibrogenesis: Bridging basic science and clinical practiceThe role of inflammation in the pathology of preeclampsiaPlasma soluble endoglin concentration in pre-eclampsia is associated with an increased impedance to flow in the maternal and fetal circulations.Recent insights into the pathophysiology of preeclampsia.Heterozygous deficiency of endoglin decreases insulin and hepatic triglyceride levels during high fat dietAngiogenic factors and preeclampsia.Endothelin type A receptor antagonist attenuates placental ischemia-induced hypertension and uterine vascular resistance.Role of reactive oxygen species during hypertension in response to chronic antiangiogenic factor (sFlt-1) excess in pregnant rats.Full-length human placental sFlt-1-e15a isoform induces distinct maternal phenotypes of preeclampsia in mice.Recombinant vascular endothelial growth factor 121 attenuates autoantibody-induced features of pre-eclampsia in pregnant mice.Activating autoantibodies to the angiotensin II type I receptor play an important role in mediating hypertension in response to adoptive transfer of CD4+ T lymphocytes from placental ischemic ratsIs preeclampsia an autoimmune disease?Endoglin regulates PI3-kinase/Akt trafficking and signaling to alter endothelial capillary stability during angiogenesis.Late-onset preeclampsia is associated with an imbalance of angiogenic and anti-angiogenic factors in patients with and without placental lesions consistent with maternal underperfusion.A model of preeclampsia in rats: the reduced uterine perfusion pressure (RUPP) model.Genetic, immune and vasoactive factors in the vascular dysfunction associated with hypertension in pregnancy.Placental ischemia increases seizure susceptibility and cerebrospinal fluid cytokinesPlacental ischemia impairs middle cerebral artery myogenic responses in the pregnant rat.Placental ischemia-induced increases in brain water content and cerebrovascular permeability: role of TNF-αImpaired autophagy by soluble endoglin, under physiological hypoxia in early pregnant period, is involved in poor placentation in preeclampsia.Protein Kinase C Inhibitors as Modulators of Vascular Function and their Application in Vascular DiseaseA critical role of interleukin-10 in modulating hypoxia-induced preeclampsia-like disease in mice.Endoglin: a critical mediator of cardiovascular healthMechanisms of Endothelial Dysfunction in Hypertensive Pregnancy and Preeclampsia.Identifying immune mechanisms mediating the hypertension during preeclampsiaSildenafil attenuates placental ischemia-induced hypertension.Recent advances in the understanding of the pathophysiology of preeclampsia.Oxidative stress contributes to soluble fms-like tyrosine kinase-1 induced vascular dysfunction in pregnant ratsPlacental Ischemia and Resultant Phenotype in Animal Models of Preeclampsia.
P2860
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P2860
Hypertension produced by placental ischemia in pregnant rats is associated with increased soluble endoglin expression
description
2009 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2009
@ast
im Februar 2009 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2009/02/01)
@sk
vědecký článek publikovaný v roce 2009
@cs
wetenschappelijk artikel (gepubliceerd op 2009/02/01)
@nl
наукова стаття, опублікована в лютому 2009
@uk
مقالة علمية (نشرت في فبراير 2009)
@ar
name
Hypertension produced by place ...... ed soluble endoglin expression
@ast
Hypertension produced by place ...... ed soluble endoglin expression
@en
Hypertension produced by place ...... ed soluble endoglin expression
@nl
type
label
Hypertension produced by place ...... ed soluble endoglin expression
@ast
Hypertension produced by place ...... ed soluble endoglin expression
@en
Hypertension produced by place ...... ed soluble endoglin expression
@nl
prefLabel
Hypertension produced by place ...... ed soluble endoglin expression
@ast
Hypertension produced by place ...... ed soluble endoglin expression
@en
Hypertension produced by place ...... ed soluble endoglin expression
@nl
P2093
P2860
P1433
P1476
Hypertension produced by place ...... ed soluble endoglin expression
@en
P2093
Jeffrey S. Gilbert
Joey P. Granger
Marietta Arany
Sara A. B. Gilbert
P2860
P304
P356
10.1161/HYPERTENSIONAHA.108.123513
P407
P577
2009-02-01T00:00:00Z