Upregulation of sodium channel Nav1.3 and functional involvement in neuronal hyperexcitability associated with central neuropathic pain after spinal cord injury
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Mechanisms of chronic central neuropathic pain after spinal cord injuryNeuropathic pain develops normally in mice lacking both Na(v)1.7 and Na(v)1.8.Ion channels as drug targets in central nervous system disordersStructure and function of μ-conotoxins, peptide-based sodium channel blockers with analgesic activityFunction of microglia and macrophages in secondary damage after spinal cord injuryThoracic 9 Spinal Transection-Induced Model of Muscle Spasticity in the Rat: A Systematic Electrophysiological and Histopathological CharacterizationFood-Derived Natural Compounds for Pain Relief in Neuropathic PainRoles of Voltage-Gated Tetrodotoxin-Sensitive Sodium Channels NaV1.3 and NaV1.7 in Diabetes and Painful Diabetic NeuropathyGlobal Nav1.7 knockout mice recapitulate the phenotype of human congenital indifference to painOtotrauma induces sodium channel plasticity in auditory afferent neuronsAlterations in the spontaneous discharge patterns of single units in the dorsal cochlear nucleus following intense sound exposure.Nonassociative learning as gated neural integrator and differentiator in stimulus-response pathways.Acrolein involvement in sensory and behavioral hypersensitivity following spinal cord injury in the ratSynergetic action of domain II and IV underlies persistent current generation in Nav1.3 as revealed by a tarantula toxin.Blockage of the upregulation of voltage-gated sodium channel nav1.3 improves outcomes after experimental traumatic brain injury.Fatty acid amide hydrolase (FAAH) inhibitors exert pharmacological effects, but lack antinociceptive efficacy in rats with neuropathic spinal cord injury pain.MiR-30b Attenuates Neuropathic Pain by Regulating Voltage-Gated Sodium Channel Nav1.3 in Rats.Bioactive marine drugs and marine biomaterials for brain diseasesGene network-based analysis identifies two potential subtypes of small intestinal neuroendocrine tumors.Spinal cord injuries containing asymmetrical damage in the ventrolateral funiculus is associated with a higher incidence of at-level allodyniaEarly afferent activity from the facet joint after painful trauma to its capsule potentiates neuronal excitability and glutamate signaling in the spinal cord.Interactions of disulfide-deficient selenocysteine analogs of μ-conotoxin BuIIIB with the α-subunit of the voltage-gated sodium channel subtype 1.3.Vascular endothelial growth factor and spinal cord injury pain.Regulatory effects of intermittent noxious stimulation on spinal cord injury-sensitive microRNAs and their presumptive targets following spinal cord contusionTetrodotoxin (TTX) as a therapeutic agent for pain.Pretreatment with intrathecal amitriptyline potentiates anti-hyperalgesic effects of post-injury intra-peritoneal amitriptyline following spinal nerve ligationCalcium channel alpha-2-delta-1 protein upregulation in dorsal spinal cord mediates spinal cord injury-induced neuropathic pain states.Voltage-gated sodium channel modulation by scorpion alpha-toxins.Are voltage-gated sodium channels on the dorsal root ganglion involved in the development of neuropathic pain?The roles of sodium channels in nociception: Implications for mechanisms of painExpansion of voltage-dependent Na+ channel gene family in early tetrapods coincided with the emergence of terrestriality and increased brain complexityThe animal model of spinal cord injury as an experimental pain model.Subarachnoid Transplant of the Human Neuronal hNT2.19 Serotonergic Cell Line Attenuates Behavioral Hypersensitivity without Affecting Motor Dysfunction after Severe Contusive Spinal Cord InjuryNerve injury induces robust allodynia and ectopic discharges in Nav1.3 null mutant mice.Intermittent noxious stimulation following spinal cord contusion injury impairs locomotor recovery and reduces spinal brain-derived neurotrophic factor-tropomyosin-receptor kinase signaling in adult rats.Dissection of voltage-gated sodium channels in developing cochlear sensory epithelia.Functional gene expression differences between inbred alcohol-preferring and -non-preferring rats in five brain regionsExpression of voltage-gated sodium channel Nav1.3 is associated with severity of traumatic brain injury in adult ratsVirus-mediated shRNA knockdown of Na(v)1.3 in rat dorsal root ganglion attenuates nerve injury-induced neuropathic painProgressive inflammation-mediated neurodegeneration after traumatic brain or spinal cord injury.
P2860
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P2860
Upregulation of sodium channel Nav1.3 and functional involvement in neuronal hyperexcitability associated with central neuropathic pain after spinal cord injury
description
2003 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2003
@ast
im Oktober 2003 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2003/10/01)
@sk
vědecký článek publikovaný v roce 2003
@cs
wetenschappelijk artikel (gepubliceerd op 2003/10/01)
@nl
наукова стаття, опублікована в жовтні 2003
@uk
مقالة علمية (نشرت في أكتوبر 2003)
@ar
name
Upregulation of sodium channel ...... pain after spinal cord injury
@ast
Upregulation of sodium channel ...... pain after spinal cord injury
@en
Upregulation of sodium channel ...... pain after spinal cord injury
@nl
type
label
Upregulation of sodium channel ...... pain after spinal cord injury
@ast
Upregulation of sodium channel ...... pain after spinal cord injury
@en
Upregulation of sodium channel ...... pain after spinal cord injury
@nl
prefLabel
Upregulation of sodium channel ...... pain after spinal cord injury
@ast
Upregulation of sodium channel ...... pain after spinal cord injury
@en
Upregulation of sodium channel ...... pain after spinal cord injury
@nl
P2093
P3181
P1476
Upregulation of sodium channel ...... pain after spinal cord injury
@en
P2093
Bryan C Hains
Carl Y Saab
Joel A Black
Joshua P Klein
Matthew J Craner
Stephen G Waxman
P304
P3181
P356
10.1523/JNEUROSCI.23-26-08881.2003
P407
P577
2003-10-01T00:00:00Z