Spinal CCL2 pronociceptive action is no longer effective in CCR2 receptor antagonist-treated rats
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Pathological and protective roles of glia in chronic painNeuroendocrine immunoregulation in multiple sclerosisNOV/CCN3 attenuates inflammatory pain through regulation of matrix metalloproteinases-2 and -9Visualization of chemokine receptor activation in transgenic mice reveals peripheral activation of CCR2 receptors in states of neuropathic pain.Ketamine does not produce relief of neuropathic pain in mice lacking the β-common receptor (CD131).Contribution of the chemokine (C-C motif) ligand 2 (CCL2) to mechanical hypersensitivity after surgical incision in rats.Pathological pain and the neuroimmune interface.Insights into the regulation of chemokine receptors by molecular signaling pathways: functional roles in neuropathic painSuppression and regression of choroidal neovascularization in mice by a novel CCR2 antagonist, INCB3344Exploring the neuroimmunopharmacology of opioids: an integrative review of mechanisms of central immune signaling and their implications for opioid analgesia.Streptozotocin-induced early thermal hyperalgesia is independent of glycemic state of rats: role of transient receptor potential vanilloid 1(TRPV1) and inflammatory mediatorsSciatic nerve injury induces functional pro-nociceptive chemokine receptors in bladder-associated primary afferent neurons in the ratPannexin-1 Up-regulation in the Dorsal Root Ganglion Contributes to Neuropathic Pain Development.Chemokine CCL2 and its receptor CCR2 in the medullary dorsal horn are involved in trigeminal neuropathic pain.Human axonal survival of motor neuron (a-SMN) protein stimulates axon growth, cell motility, C-C motif ligand 2 (CCL2), and insulin-like growth factor-1 (IGF1) production.CCL2 and CCL3 are essential mediators of pelvic pain in experimental autoimmune prostatitisPain in experimental autoimmune encephalitis: a comparative study between different mouse models.Upregulation of nuclear factor of activated T-cells by nerve injury contributes to development of neuropathic pain.Expression and function of CCL2/CCR2 in rat micturition reflexes and somatic sensitivity with urinary bladder inflammationPaeoniflorin and Albiflorin Attenuate Neuropathic Pain via MAPK Pathway in Chronic Constriction Injury Rats.Functional inhibition of chemokine receptor CCR2 by dicer-substrate-siRNA prevents pain developmentJNK-induced MCP-1 production in spinal cord astrocytes contributes to central sensitization and neuropathic pain.Chemokines and pain mechanisms.Chemokine action in the nervous systemInfluence of the vanilloid receptor TRPV1 on the activation of spinal cord glia in mouse models of pain.Chemokine contribution to neuropathic pain: respective induction of CXCL1 and CXCR2 in spinal cord astrocytes and neurons.The role(s) of cytokines/chemokines in urinary bladder inflammation and dysfunction.Chemokines, neuronal-glial interactions, and central processing of neuropathic pain.Neurochemokines: a menage a trois providing new insights on the functions of chemokines in the central nervous system.Neuropathic pain and cytokines: current perspectives.Peroxisome proliferator-activated receptor agonists modulate neuropathic pain: a link to chemokines?Opioid and chemokine receptor crosstalk: a promising target for pain therapy?The therapeutic potential of targeting chemokine signalling in the treatment of chronic pain.Involvement of spinal chemokine CCL2 in the hyperalgesia evoked by bone cancer in mice: a role for astroglia and microglia.Chemokines in neuron-glial cell interaction and pathogenesis of neuropathic pain.CCL2 released at tumoral level contributes to the hyperalgesia evoked by intratibial inoculation of NCTC 2472 but not B16-F10 cells in mice.PKC-NF-κB are involved in CCL2-induced Nav1.8 expression and channel function in dorsal root ganglion neuronsBlocking spinal CCR2 with AZ889 reversed hyperalgesia in a model of neuropathic pain.Minocycline, a microglial inhibitor, blocks spinal CCL2-induced heat hyperalgesia and augmentation of glutamatergic transmission in substantia gelatinosa neurons.Interferon-gamma potentiates NMDA receptor signaling in spinal dorsal horn neurons via microglia-neuron interaction.
P2860
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P2860
Spinal CCL2 pronociceptive action is no longer effective in CCR2 receptor antagonist-treated rats
description
2008 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2008
@ast
scientific journal article
@en
vedecký článok (publikovaný 2008/07/01)
@sk
vědecký článek publikovaný v roce 2008
@cs
wetenschappelijk artikel (gepubliceerd op 2008/07/01)
@nl
наукова стаття, опублікована в липні 2008
@uk
مقالة علمية (نشرت في يوليو 2008)
@ar
name
Spinal CCL2 pronociceptive act ...... ceptor antagonist-treated rats
@ast
Spinal CCL2 pronociceptive act ...... ceptor antagonist-treated rats
@en
Spinal CCL2 pronociceptive act ...... ceptor antagonist-treated rats
@nl
type
label
Spinal CCL2 pronociceptive act ...... ceptor antagonist-treated rats
@ast
Spinal CCL2 pronociceptive act ...... ceptor antagonist-treated rats
@en
Spinal CCL2 pronociceptive act ...... ceptor antagonist-treated rats
@nl
prefLabel
Spinal CCL2 pronociceptive act ...... ceptor antagonist-treated rats
@ast
Spinal CCL2 pronociceptive act ...... ceptor antagonist-treated rats
@en
Spinal CCL2 pronociceptive act ...... ceptor antagonist-treated rats
@nl
P2093
P2860
P50
P3181
P1476
Spinal CCL2 pronociceptive act ...... ceptor antagonist-treated rats
@en
P2093
Annie Mauborgne
Blandine Pommier
Marc-André Dansereau
Michel Pohl
Patricia Mechighel
Patrick Kitabgi
Philippe Sarret
Romain-Daniel Gosselin
P2860
P304
P3181
P356
10.1111/J.1471-4159.2008.05429.X
P407
P577
2008-04-17T00:00:00Z