The interaction of caveolin 3 protein with the potassium inward rectifier channel Kir2.1: physiology and pathology related to long qt syndrome 9 (LQT9)
about
Protein assemblies of sodium and inward rectifier potassium channels control cardiac excitability and arrhythmogenesisIon channel macromolecular complexes in cardiomyocytes: roles in sudden cardiac deathMicrodomain-specific localization of functional ion channels in cardiomyocytes: an emerging concept of local regulation and remodelling.Identification and functional analysis of a new putative caveolin-3 variant found in a patient with sudden unexplained deathDiseases caused by mutations in Nav1.5 interacting proteinsNav1.5 N-terminal domain binding to α1-syntrophin increases membrane density of human Kir2.1, Kir2.2 and Nav1.5 channels.IK1-enhanced human-induced pluripotent stem cell-derived cardiomyocytes: an improved cardiomyocyte model to investigate inherited arrhythmia syndromesApplication of fluorescence resonance energy transfer in protein studiesGenetic Discoveries in Atrial Fibrillation and Implications for Clinical Practice.Histone Deacetylase Inhibitors Prolong Cardiac Repolarization through Transcriptional Mechanisms.Rare variants in genes encoding the cardiac sodium channel and associated compounds and their impact on outcome of catheter ablation of atrial fibrillation.Potential of caveolae in the therapy of cardiovascular and neurological diseases.Inward Rectifier Potassium Channels (Kir2.x) and Caveolin-3 Domain-Specific Interaction: Implications for Purkinje Cell-Dependent Ventricular Arrhythmias.Perivascular adipose tissue and the dynamic regulation of Kv 7 and Kir channels: Implications for resistant hypertension.The expression of the rare caveolin-3 variant T78M alters cardiac ion channels function and membrane excitability.Simulation of the effects of moderate stimulation/inhibition of the β1-adrenergic signaling system and its components in mouse ventricular myocytes.Ion Channel Disorders and Sudden Cardiac Death.Voltage-Gated Sodium Channel β1/β1B Subunits Regulate Cardiac Physiology and Pathophysiology.Golgin-97 Targets Ectopically Expressed Inward Rectifying Potassium Channel, Kir2.1, to the -Golgi Network in COS-7 Cells
P2860
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P2860
The interaction of caveolin 3 protein with the potassium inward rectifier channel Kir2.1: physiology and pathology related to long qt syndrome 9 (LQT9)
description
2013 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի հունիսին հրատարակված գիտական հոդված
@hy
article publié dans la revue scientifique Journal of Biological Chemistry
@fr
artículu científicu espublizáu en 2013
@ast
im Juni 2013 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2013/06/14)
@sk
vědecký článek publikovaný v roce 2013
@cs
wetenschappelijk artikel (gepubliceerd op 2013/06/14)
@nl
наукова стаття, опублікована в червні 2013
@uk
name
The interaction of caveolin 3 ...... d to long qt syndrome 9 (LQT9)
@ast
The interaction of caveolin 3 ...... d to long qt syndrome 9 (LQT9)
@en
The interaction of caveolin 3 ...... d to long qt syndrome 9 (LQT9)
@nl
type
label
The interaction of caveolin 3 ...... d to long qt syndrome 9 (LQT9)
@ast
The interaction of caveolin 3 ...... d to long qt syndrome 9 (LQT9)
@en
The interaction of caveolin 3 ...... d to long qt syndrome 9 (LQT9)
@nl
prefLabel
The interaction of caveolin 3 ...... d to long qt syndrome 9 (LQT9)
@ast
The interaction of caveolin 3 ...... d to long qt syndrome 9 (LQT9)
@en
The interaction of caveolin 3 ...... d to long qt syndrome 9 (LQT9)
@nl
P2093
P2860
P356
P1476
The interaction of caveolin 3 ...... d to long qt syndrome 9 (LQT9)
@en
P2093
Amanda L. Vega
Bi-Hua Tan
Chunhua Song
Jonathan C. Makielski
Lee L. Eckhardt
Ravi Vaidyanathan
Stuart Berger
P2860
P304
17472–17480
P356
10.1074/JBC.M112.435370
P407
P577
2013-06-14T00:00:00Z