Tyrosine kinase and phosphatidylinositol 3-kinase activation are required for cyclic adenosine 3',5'-monophosphate-dependent potentiation of deoxyribonucleic acid synthesis induced by insulin-like growth factor-I in FRTL-5 cells
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53BP2S, interacting with insulin receptor substrates, modulates insulin signalingEffects of tyrosine kinase inhibitors on cell death induced by sodium fluoride and pertussis toxin in the pancreatic beta-cell line, RINm5FPhosphatidylinositol 3-Kinase-Associated Protein (PI3KAP)/XB130 Crosslinks Actin Filaments through Its Actin Binding and Multimerization Properties In Vitro and Enhances Endocytosis in HEK293 CellsPhosphatidylinositol 3-kinase-binding protein, PI3KAP/XB130, is required for cAMP-induced amplification of IGF mitogenic activity in FRTL-5 thyroid cellsIdentification of microRNAs that mediate thyroid cell growth induced by TSHParaquat-induced oxidative stress represses phosphatidylinositol 3-kinase activities leading to impaired glucose uptake in 3T3-L1 adipocytes.Tumor necrosis factor (TNF)-α-induced repression of GKAP42 protein levels through cGMP-dependent kinase (cGK)-Iα causes insulin resistance in 3T3-L1 adipocytesPhosphatidylinositol 3-kinase (PI3K) activity bound to insulin-like growth factor-I (IGF-I) receptor, which is continuously sustained by IGF-I stimulation, is required for IGF-I-induced cell proliferation.Mechanism by which cAMP activates PI3-kinase and increases bile acid secretion in WIF-B9 cells.The association between thyroid autoantibodies in serum and abnormal function and structure of the thyroid.Growth hormone inhibition of glucose uptake in adipocytes occurs without affecting GLUT4 translocation through an insulin receptor substrate-2-phosphatidylinositol 3-kinase-dependent pathway.TSH signaling and cell survival in 3T3-L1 preadipocytes.ERK phosphorylation is not increased in papillary thyroid carcinomas with BRAF(V600E) mutation compared to that of corresponding normal thyroid tissues.The Emerging Roles of Thyroglobulin
P2860
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P2860
Tyrosine kinase and phosphatidylinositol 3-kinase activation are required for cyclic adenosine 3',5'-monophosphate-dependent potentiation of deoxyribonucleic acid synthesis induced by insulin-like growth factor-I in FRTL-5 cells
description
2000 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2000
@ast
im Juli 2000 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2000/07/01)
@sk
vědecký článek publikovaný v roce 2000
@cs
wetenschappelijk artikel (gepubliceerd op 2000/07/01)
@nl
наукова стаття, опублікована в липні 2000
@uk
مقالة علمية (نشرت في يوليو 2000)
@ar
name
Tyrosine kinase and phosphatid ...... rowth factor-I in FRTL-5 cells
@ast
Tyrosine kinase and phosphatid ...... rowth factor-I in FRTL-5 cells
@en
Tyrosine kinase and phosphatid ...... rowth factor-I in FRTL-5 cells
@nl
type
label
Tyrosine kinase and phosphatid ...... rowth factor-I in FRTL-5 cells
@ast
Tyrosine kinase and phosphatid ...... rowth factor-I in FRTL-5 cells
@en
Tyrosine kinase and phosphatid ...... rowth factor-I in FRTL-5 cells
@nl
prefLabel
Tyrosine kinase and phosphatid ...... rowth factor-I in FRTL-5 cells
@ast
Tyrosine kinase and phosphatid ...... rowth factor-I in FRTL-5 cells
@en
Tyrosine kinase and phosphatid ...... rowth factor-I in FRTL-5 cells
@nl
P2093
P3181
P1433
P1476
Tyrosine kinase and phosphatid ...... rowth factor-I in FRTL-5 cells
@en
P2093
H. Sakamoto
K. Umesaki
M. Akahori
S. I. Takahashi
T. Nedachi
P304
P3181
P356
10.1210/EN.141.7.2429
10.1210/ENDO.141.7.7539
P407
P577
2000-07-01T00:00:00Z