The effect of mood stabilizer lithium on expression and activity of glutathione s-transferase isoenzymes
about
Mitochondrial modulators for bipolar disorder: A pathophysiologically informed paradigm for new drug developmentThe role of lithium in the treatment of bipolar disorder: convergent evidence for neurotrophic effects as a unifying hypothesisOxidative damage to RNA but not DNA in the hippocampus of patients with major mental illness.Impaired mitochondrial function in psychiatric disorders.The relationship between oxidative stress and post-translational modification of the dopamine transporter in bipolar disorder.Mitochondria and the central nervous system: searching for a pathophysiological basis of psychiatric disorders.Lithium protects against oxidative stress-mediated cell death in α-synuclein-overexpressing in vitro and in vivo models of Parkinson's disease.Effect of Long-term In Vitro Lithium Exposure on mRNA Levels of Claudin-3, CYP1A1, ABCG2 and GSTM3 Genes in the hCMEC/D3 Human Brain Endothelial Cell Line.Glutathione-mediated effects of lithium in decreasing protein oxidation induced by mitochondrial complex I dysfunction.Neuroprotective effects of chronic exposure of SH-SY5Y to low lithium concentration involve glycolysis stimulation, extracellular pyruvate accumulation and resistance to oxidative stress.Targeting oxidative stress in the hypothalamus: the effect of transcription factor STAT3 knockdown on endogenous antioxidants-mediated appetite control.Effects of lithium and lamotrigine on oxidative-nitrosative stress and spatial learning deficit after global cerebral ischemia.Investigating the protective effect of lithium against high glucose-induced neurotoxicity in PC12 cells: involvements of ROS, JNK and P38 MAPKs, and apoptotic mitochondria pathway.Decreased global methylation in patients with bipolar disorder who respond to lithium.Prefrontal cortex glutathione S-transferase levels in patients with bipolar disorder, major depression and schizophrenia.Mitochondrial dysfunction and lipid peroxidation in rat frontal cortex by chronic NMDA administration can be partially prevented by lithium treatment.Glutathione mediated reductive activation and mitochondrial dysfunction play key roles in lithium induced oxidative stress and cytotoxicity in liver.Toxicity of valproic acid in isolated rat liver mitochondria.Lithium reduces the effects of rotenone-induced complex I dysfunction on DNA methylation and hydroxymethylation in rat cortical primary neurons.
P2860
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P2860
The effect of mood stabilizer lithium on expression and activity of glutathione s-transferase isoenzymes
description
2008 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի հունվարին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2008
@ast
im Januar 2008 veröffentlichter wissenschaftlicher Artikel
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scientific journal article
@en
vedecký článok (publikovaný 2008/01/24)
@sk
vědecký článek publikovaný v roce 2008
@cs
wetenschappelijk artikel (gepubliceerd op 2008/01/24)
@nl
наукова стаття, опублікована в січні 2008
@uk
مقالة علمية (نشرت في 24-1-2008)
@ar
name
The effect of mood stabilizer ...... hione s-transferase isoenzymes
@ast
The effect of mood stabilizer ...... hione s-transferase isoenzymes
@en
The effect of mood stabilizer ...... hione s-transferase isoenzymes
@nl
type
label
The effect of mood stabilizer ...... hione s-transferase isoenzymes
@ast
The effect of mood stabilizer ...... hione s-transferase isoenzymes
@en
The effect of mood stabilizer ...... hione s-transferase isoenzymes
@nl
prefLabel
The effect of mood stabilizer ...... hione s-transferase isoenzymes
@ast
The effect of mood stabilizer ...... hione s-transferase isoenzymes
@en
The effect of mood stabilizer ...... hione s-transferase isoenzymes
@nl
P2093
P1433
P1476
The effect of mood stabilizer ...... hione s-transferase isoenzymes
@en
P2093
P304
P356
10.1016/J.NEUROSCIENCE.2007.10.041
P407
P577
2008-01-24T00:00:00Z