Persistent nicotine treatment potentiates amplification of the dihydrofolate reductase gene in rat lung epithelial cells as a consequence of Ras activation

Persistent nicotine treatment potentiates amplification of the dihydrofolate reductase gene in rat lung epithelial cells as a consequence of Ras activation

Item
http://www.wikidata.org/entity/Q28580057
Nicotine-mediated cell proliferation and tumor progression in smoking-related cancersNicotine overrides DNA damage-induced G1/S restriction in lung cellsDifferential sensitization of different prostate cancer cells to apoptosis.Life-long programming implications of exposure to tobacco smoking and nicotine before and soon after birth: evidence for altered lung developmentSensitization of epithelial growth factor receptors by nicotine exposure to promote breast cancer cell growthTobacco smoking: patterns, health consequences for adults, and the long-term health of the offspring.The pathobiological impact of cigarette smoke on pancreatic cancer development (review).Nicotine and lung cancer.Nicotine, through upregulating pro-survival signaling, cooperates with NNK to promote transformationNicotine promotes mammary tumor migration via a signaling cascade involving protein kinase C and CDC42.Emerging role of MAP kinase pathways as therapeutic targets in COPD.Are nicotine replacement therapy, varenicline or bupropion options for pregnant mothers to quit smoking? Effects on the respiratory system of the offspring.Rosiglitazone inhibits alpha4 nicotinic acetylcholine receptor expression in human lung carcinoma cells through peroxisome proliferator-activated receptor gamma-independent signals.Modulation of intracellular signaling pathways to induce apoptosis in prostate cancer cells.Tomato juice protects the lungs of the offspring of female rats exposed to nicotine during gestation and lactation.[Roles of nicotine and nicotinic acetylcholine receptors (nAChR) in carcinogenesis and development of lung cancer].Nicotine-induced memory impairment by increasing brain oxidative stress
P2860

Persistent nicotine treatment potentiates amplification of the dihydrofolate reductase gene in rat lung epithelial cells as a consequence of Ras activation

Item
http://www.wikidata.org/entity/Q28580057
description
2005 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
article publié dans la revue scientifique Journal of Biological Chemistry
@fr
artículu científicu espublizáu en 2005
@ast
im August 2005 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2005/08/26)
@sk
vědecký článek publikovaný v roce 2005
@cs
wetenschappelijk artikel (gepubliceerd op 2005/08/26)
@nl
наукова стаття, опублікована в серпні 2005
@uk
name
Persistent nicotine treatment ...... consequence of Ras activation
@ast
Persistent nicotine treatment ...... consequence of Ras activation
@en
Persistent nicotine treatment ...... consequence of Ras activation
@nl
type
label
Persistent nicotine treatment ...... consequence of Ras activation
@ast
Persistent nicotine treatment ...... consequence of Ras activation
@en
Persistent nicotine treatment ...... consequence of Ras activation
@nl
prefLabel
Persistent nicotine treatment ...... consequence of Ras activation
@ast
Persistent nicotine treatment ...... consequence of Ras activation
@en
Persistent nicotine treatment ...... consequence of Ras activation
@nl
P1433
P1476
P2093
P2860
P304
P31
P356
P407
P433
P478
P577
P698
P356
P1433
P1476
Persistent nicotine treatment ...... consequence of Ras activation
@en
P2093
P2860
P304
P31
P356
P407
P433
P478
P577
P698