Regulation of CD18 expression on neutrophils in response to fluid shear stress
about
LFA-1 and Mac-1 define characteristically different intralumenal crawling and emigration patterns for monocytes and neutrophils in situ.Vav1 is essential for mechanotactic crawling and migration of neutrophils out of the inflamed microvasculatureReceptor cleavage reduces the fluid shear response in neutrophils of the spontaneously hypertensive ratShear-induced resistance to neutrophil activation via the formyl peptide receptorReceptor cleavage and P-selectin-dependent reduction of leukocyte adhesion in the spontaneously hypertensive rat.Shear-induced capping of L-selectin on the neutrophil surface during centrifugationCleavage and reduced CD36 ectodomain density on heart and spleen macrophages in the spontaneously hypertensive rat.Two-dimensional kinetics of beta 2-integrin and ICAM-1 bindings between neutrophils and melanoma cells in a shear flow.Biomechanical regulation of cell orientation and fateFocal adhesion kinase regulation of mechanotransduction and its impact on endothelial cell functions.Cell friction.The autodigestion hypothesis: Proteolytic receptor cleavage in rheological and cardiovascular cell dysfunction1.G protein-coupled receptors serve as mechanosensors for fluid shear stress in neutrophils.Shear stress during early embryonic stem cell differentiation promotes hematopoietic and endothelial phenotypes.Flow cytometry as a diagnostic tool for identifying total hip arthroplasty loosening and differentiating between septic and aseptic cases.Hydrodynamic shear rate regulates melanoma-leukocyte aggregation, melanoma adhesion to the endothelium, and subsequent extravasationVascular receptors as new substrates for matrix metalloproteinases in hypertension and other inflammatory states.Control of neutrophil pseudopods by fluid shear: role of Rho family GTPases.Characterization of the shear stress regulation of CD18 surface expression by HL60-derived neutrophil-like cells.Toll-like receptor signaling mechanisms in hostile neutrophils. Focus on "Bone marrow MyD88 signaling modulates neutrophil function and ischemic myocardial injury".Fluid shear-induced cathepsin B release in the control of Mac1-dependent neutrophil adhesion.Pathophysiological Mechanisms of Chronic Venous Disease and Implications for Venoactive Drug Therapy.
P2860
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P2860
Regulation of CD18 expression on neutrophils in response to fluid shear stress
description
2003 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
article publié dans les Procee ...... f the United States of America
@fr
artículu científicu espublizáu en 2003
@ast
im November 2003 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2003/11/11)
@sk
vědecký článek publikovaný v roce 2003
@cs
wetenschappelijk artikel (gepubliceerd op 2003/11/11)
@nl
наукова стаття, опублікована в листопаді 2003
@uk
name
Regulation of CD18 expression on neutrophils in response to fluid shear stress
@ast
Regulation of CD18 expression on neutrophils in response to fluid shear stress
@en
Regulation of CD18 expression on neutrophils in response to fluid shear stress
@nl
type
label
Regulation of CD18 expression on neutrophils in response to fluid shear stress
@ast
Regulation of CD18 expression on neutrophils in response to fluid shear stress
@en
Regulation of CD18 expression on neutrophils in response to fluid shear stress
@nl
prefLabel
Regulation of CD18 expression on neutrophils in response to fluid shear stress
@ast
Regulation of CD18 expression on neutrophils in response to fluid shear stress
@en
Regulation of CD18 expression on neutrophils in response to fluid shear stress
@nl
P2860
P356
P1476
Regulation of CD18 expression on neutrophils in response to fluid shear stress
@en
P2093
Geert W. Schmid-Schönbein
Shunichi Fukuda
P2860
P304
13152–13157
P356
10.1073/PNAS.2336130100
P407
P577
2003-11-11T00:00:00Z