HIF-1 inhibition decreases systemic vascular remodelling diseases by promoting apoptosis through a hexokinase 2-dependent mechanism
about
L-2-Oxothiazolidine-4-carboxylic acid or α-lipoic acid attenuates airway remodeling: involvement of nuclear factor-κB (NF-κB), nuclear factor erythroid 2p45-related factor-2 (Nrf2), and hypoxia-inducible factor (HIF)Prolonged mechanical stretch is associated with upregulation of hypoxia-inducible factors and reduced contraction in rat inferior vena cavaActivation of the pluripotency factor OCT4 in smooth muscle cells is atheroprotective.Hypoxia-inducible factor-1 activation in nonhypoxic conditions: the essential role of mitochondrial-derived reactive oxygen species.PDGF-induced vascular smooth muscle cell proliferation is associated with dysregulation of insulin receptor substrates.Middle cerebral artery alterations in a rat chronic hypoperfusion modelPKCε promotes cardiac mitochondrial and metabolic adaptation to chronic hypobaric hypoxia by GSK3β inhibition.Nickel-free stainless steel avoids neointima formation following coronary stent implantation.Hyperoxia synergizes with mutant bone morphogenic protein receptor 2 to cause metabolic stress, oxidant injury, and pulmonary hypertension.Mitochondria in lung diseases.The emergence of new therapeutic targets in pulmonary arterial hypertension: from now to the near future.Apoptotic cell death under hypoxia.Vascular remodeling process in pulmonary arterial hypertension, with focus on miR-204 and miR-126 (2013 Grover Conference series).Thyroid hormone and anti-apoptosis in tumor cells.Cross-talk between signaling and metabolism in the vasculature.Asiatic Acid Attenuates Myocardial Ischemia/Reperfusion Injury via Akt/GSK-3β/HIF-1α Signaling in Rat H9c2 Cardiomyocytes.RAGE-dependent activation of the oncoprotein Pim1 plays a critical role in systemic vascular remodeling processes.Increased aerobic glycolysis is important for the motility of activated VSMC and inhibited by indirubin-3'-monoxime.Role for Runt-related Transcription Factor 2 in Proliferative and Calcified Vascular Lesions in Pulmonary Arterial Hypertension.The effects of the 5-HT2A receptor antagonist sarpogrelate hydrochloride on chronic hypoxic pulmonary hypertension in rats.Assessment of HIF-1α expression and release following endothelial injury in-vitro and in-vivo.Advanced glycation end products accelerate calcification in VSMCs through HIF-1α/PDK4 activation and suppress glucose metabolism
P2860
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P2860
HIF-1 inhibition decreases systemic vascular remodelling diseases by promoting apoptosis through a hexokinase 2-dependent mechanism
description
2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2010
@ast
im Oktober 2010 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2010/10/01)
@sk
vědecký článek publikovaný v roce 2010
@cs
wetenschappelijk artikel (gepubliceerd op 2010/10/01)
@nl
наукова стаття, опублікована в жовтні 2010
@uk
مقالة علمية (نشرت في أكتوبر 2010)
@ar
name
HIF-1 inhibition decreases sys ...... xokinase 2-dependent mechanism
@ast
HIF-1 inhibition decreases sys ...... xokinase 2-dependent mechanism
@en
HIF-1 inhibition decreases sys ...... xokinase 2-dependent mechanism
@nl
type
label
HIF-1 inhibition decreases sys ...... xokinase 2-dependent mechanism
@ast
HIF-1 inhibition decreases sys ...... xokinase 2-dependent mechanism
@en
HIF-1 inhibition decreases sys ...... xokinase 2-dependent mechanism
@nl
prefLabel
HIF-1 inhibition decreases sys ...... xokinase 2-dependent mechanism
@ast
HIF-1 inhibition decreases sys ...... xokinase 2-dependent mechanism
@en
HIF-1 inhibition decreases sys ...... xokinase 2-dependent mechanism
@nl
P2093
P2860
P3181
P356
P1476
HIF-1 inhibition decreases sys ...... xokinase 2-dependent mechanism
@en
P2093
Caroline M Lambert
Geneviève A Robitaille
Mélanie Roy
Sébastien Bonnet
P2860
P304
P3181
P356
10.1093/CVR/CVQ152
P577
2010-05-24T00:00:00Z