Adaptive mechanisms that preserve cardiac function in mice without myoglobin
about
skNAC, a Smyd1-interacting transcription factor, is involved in cardiac development and skeletal muscle growth and regenerationSimulating evolution by gene duplication of protein features that require multiple amino acid residuesAdaptation of the myoglobin knockout mouse to hypoxic stressAbnormal cardiac development in the absence of heart glycogenIn vivo microscopy reveals extensive embedding of capillaries within the sarcolemma of skeletal muscle fibersComparative genetic analysis: the utility of mouse genetic systems for studying human monogenic disease.Ontogeny of globin expression in zebrafish (Danio rerio).Hypoxia-induced left ventricular dysfunction in myoglobin-deficient mice.Regulation of myoglobin expressionNeonatal hyper- and hypothyroidism alter the myoglobin gene expression program in adulthood.Cardiac metabolic pathways affected in the mouse model of barth syndrome.Hypoxia reprograms calcium signaling and regulates myoglobin expression.Skeletal myofiber VEGF regulates contraction-induced perfusion and exercise capacity but not muscle capillarity in adult mice.Disruption of nesprin-1 produces an Emery Dreifuss muscular dystrophy-like phenotype in mice.Cytoglobin is a stress-responsive hemoprotein expressed in the developing and adult brain.Myoglobin's old and new clothes: from molecular structure to function in living cells.Ischemia-induced copper loss and suppression of angiogenesis in the pathogenesis of myocardial infarction.Highly athletic terrestrial mammals: horses and dogs.Regulation of cellular gas exchange, oxygen sensing, and metabolic control.Remodeling and dedifferentiation of adult cardiomyocytes during disease and regeneration.Myoglobin and the regulation of mitochondrial respiratory chain complex IV.Ankyrin Repeat Domain 1 Protein: A Functionally Pleiotropic Protein with Cardiac Biomarker Potential.Muscle-specific VEGF deficiency greatly reduces exercise endurance in mice.Myoglobin-deficient mice activate a distinct cardiac gene expression program in response to isoproterenol-induced hypertrophy.Enzyme activity and myoglobin concentration in rat myocardium and skeletal muscles after passive intermittent simulated altitude exposure.HIC2 regulates isoform switching during maturation of the cardiovascular system.Skeletal muscle glucose uptake during treadmill exercise in neuronal nitric oxide synthase-μ knockout mice.
P2860
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P2860
Adaptive mechanisms that preserve cardiac function in mice without myoglobin
description
2001 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2001
@ast
im April 2001 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2001/04/13)
@sk
vědecký článek publikovaný v roce 2001
@cs
wetenschappelijk artikel (gepubliceerd op 2001/04/13)
@nl
наукова стаття, опублікована у квітні 2001
@uk
مقالة علمية (نشرت في 13-4-2001)
@ar
name
Adaptive mechanisms that preserve cardiac function in mice without myoglobin
@ast
Adaptive mechanisms that preserve cardiac function in mice without myoglobin
@en
Adaptive mechanisms that preserve cardiac function in mice without myoglobin
@nl
type
label
Adaptive mechanisms that preserve cardiac function in mice without myoglobin
@ast
Adaptive mechanisms that preserve cardiac function in mice without myoglobin
@en
Adaptive mechanisms that preserve cardiac function in mice without myoglobin
@nl
prefLabel
Adaptive mechanisms that preserve cardiac function in mice without myoglobin
@ast
Adaptive mechanisms that preserve cardiac function in mice without myoglobin
@en
Adaptive mechanisms that preserve cardiac function in mice without myoglobin
@nl
P2093
P356
P1433
P1476
Adaptive mechanisms that preserve cardiac function in mice without myoglobin
@en
P2093
A. P. Meeson
D. J. Garry
J. Elterman
J. M. DiMaio
J. M. Shelton
K. Hutcheson
N. Radford
P. P. Mammen
R. S. Williams
P304
P356
10.1161/HH0701.089753
P577
2001-04-13T00:00:00Z