The heat-stable antigen determines pathogenicity of self-reactive T cells in experimental autoimmune encephalomyelitis
about
CD24-Siglec G/10 discriminates danger- from pathogen-associated molecular patternsCD24 polymorphisms affect risk and progression of chronic hepatitis B virus infectionA dinucleotide deletion in CD24 confers protection against autoimmune diseasesCD24 controls expansion and persistence of autoreactive T cells in the central nervous system during experimental autoimmune encephalomyelitisCD24 expression on T cells is required for optimal T cell proliferation in lymphopenic hostMassive and destructive T cell response to homeostatic cue in CD24-deficient lymphopenic hostsDeletion of CD24 impairs development of heat shock protein gp96-driven autoimmune disease through expansion of myeloid-derived suppressor cells.CD24 is a genetic modifier for risk and progression of multiple sclerosis.Leptin surge precedes onset of autoimmune encephalomyelitis and correlates with development of pathogenic T cell responses.CD24 on thymic APCs regulates negative selection of myelin antigen-specific T lymphocytes.A hypermorphic SP1-binding CD24 variant associates with risk and progression of multiple sclerosis.CD24 expression does not affect dopamine neuronal survival in a mouse model of Parkinson's disease.Natural ligands for CD33-related Siglecs?CD24 and Siglec-10 selectively repress tissue damage-induced immune responses.CD24 enrichment protects while its loss increases susceptibility of juvenile chondrocytes towards inflammationCD24: from A to Z.Association between the CD24 Ala57Val polymorphism and risk for multiple sclerosis and systemic lupus erythematosus: a meta-analysis.CD24: from a Hematopoietic Differentiation Antigen to a Genetic Risk Factor for Multiple Autoimmune Diseases.CD24 aggravates acute liver injury in autoimmune hepatitis by promoting IFN-γ production by CD4(+) T cells.CD24 Ala57Val polymorphism is associated with spontaneous viral clearance in the HCV-infected Chinese population.Elevated cerebral cortical CD24 levels in patients and mice with traumatic brain injury: a potential negative role in nuclear factor κb/inflammatory factor pathway.Interleukin-27 Gene Therapy Prevents the Development of Autoimmune Encephalomyelitis but Fails to Attenuate Established Inflammation due to the Expansion of CD11b+Gr-1+ Myeloid Cells.
P2860
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P2860
The heat-stable antigen determines pathogenicity of self-reactive T cells in experimental autoimmune encephalomyelitis
description
2000 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի մայիսին հրատարակված գիտական հոդված
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artículu científicu espublizáu en 2000
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im Mai 2000 veröffentlichter wissenschaftlicher Artikel
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scientific journal article
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vedecký článok (publikovaný 2000/05/01)
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vědecký článek publikovaný v roce 2000
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wetenschappelijk artikel (gepubliceerd op 2000/05/01)
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наукова стаття, опублікована в травні 2000
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مقالة علمية (نشرت في مايو 2000)
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name
The heat-stable antigen determ ...... l autoimmune encephalomyelitis
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The heat-stable antigen determ ...... l autoimmune encephalomyelitis
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The heat-stable antigen determ ...... l autoimmune encephalomyelitis
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type
label
The heat-stable antigen determ ...... l autoimmune encephalomyelitis
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The heat-stable antigen determ ...... l autoimmune encephalomyelitis
@en
The heat-stable antigen determ ...... l autoimmune encephalomyelitis
@nl
prefLabel
The heat-stable antigen determ ...... l autoimmune encephalomyelitis
@ast
The heat-stable antigen determ ...... l autoimmune encephalomyelitis
@en
The heat-stable antigen determ ...... l autoimmune encephalomyelitis
@nl
P2093
P2860
P921
P356
P1476
The heat-stable antigen determ ...... l autoimmune encephalomyelitis
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P2093
P2860
P304
P356
10.1172/JCI9012
P407
P577
2000-05-01T00:00:00Z