Calmodulin protects against alcohol-induced pancreatic trypsinogen activation elicited via Ca2+ release through IP3 receptors
about
Alcoholic pancreatitis: New insights into the pathogenesis and treatmentReview article: pancreatic renin-angiotensin systems in health and diseasePhysiology and pathophysiology of bicarbonate secretion by pancreatic duct epitheliumThe role of Ca2+ in the pathophysiology of pancreatitisFatty acid ethyl ester synthase inhibition ameliorates ethanol-induced Ca2+-dependent mitochondrial dysfunction and acute pancreatitisPathobiology of acute pancreatitis: focus on intracellular calcium and calmodulin.The ryanodine receptor is expressed in human pancreatic acinar cells and contributes to acinar cell injuryMitochondrial function and malfunction in the pathophysiology of pancreatitis.IP3 receptor type 2 deficiency is associated with a secretory defect in the pancreatic acinar cell and an accumulation of zymogen granules.Calcium signaling of pancreatic acinar cells in the pathogenesis of pancreatitis.Src Dependent Pancreatic Acinar Injury Can Be Initiated Independent of an Increase in Cytosolic Calcium.Bile acids induce pancreatic acinar cell injury and pancreatitis by activating calcineurin.Ca2+ release-activated Ca2+ channel blockade as a potential tool in antipancreatitis therapyRyanodine receptors contribute to bile acid-induced pathological calcium signaling and pancreatitis in mice.Pancreatic acinar cell nuclear factor κB activation because of bile acid exposure is dependent on calcineurin.Cluster of differentiation 38 (CD38) mediates bile acid-induced acinar cell injury and pancreatitis through cyclic ADP-ribose and intracellular calcium releaseEffects of oxidative alcohol metabolism on the mitochondrial permeability transition pore and necrosis in a mouse model of alcoholic pancreatitis.Caffeine protects against experimental acute pancreatitis by inhibition of inositol 1,4,5-trisphosphate receptor-mediated Ca2+ release.Cellular geography of IP3 receptors, STIM and Orai: a lesson from secretory epithelial cells.Treating acute pancreatitis: what's new?BH3 mimetic-elicited Ca2+ signals in pancreatic acinar cells are dependent on Bax and can be reduced by Ca2+-like peptides.
P2860
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P2860
Calmodulin protects against alcohol-induced pancreatic trypsinogen activation elicited via Ca2+ release through IP3 receptors
description
2011 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
article publié dans les Procee ...... f the United States of America
@fr
artículu científicu espublizáu en 2011
@ast
im April 2011 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2011/04/05)
@sk
vědecký článek publikovaný v roce 2011
@cs
wetenschappelijk artikel (gepubliceerd op 2011/04/05)
@nl
наукова стаття, опублікована у квітні 2011
@uk
name
Calmodulin protects against al ...... release through IP3 receptors
@ast
Calmodulin protects against al ...... release through IP3 receptors
@en
Calmodulin protects against al ...... release through IP3 receptors
@nl
type
label
Calmodulin protects against al ...... release through IP3 receptors
@ast
Calmodulin protects against al ...... release through IP3 receptors
@en
Calmodulin protects against al ...... release through IP3 receptors
@nl
prefLabel
Calmodulin protects against al ...... release through IP3 receptors
@ast
Calmodulin protects against al ...... release through IP3 receptors
@en
Calmodulin protects against al ...... release through IP3 receptors
@nl
P2093
P2860
P356
P1476
Calmodulin protects against al ...... release through IP3 receptors
@en
P2093
Etsuko Ebisui
György Lur
Julia V Gerasimenko
Katsuhiko Mikoshiba
Ole H Petersen
Oleg V Gerasimenko
Pawel Ferdek
P2860
P304
P356
10.1073/PNAS.1016534108
P407
P577
2011-03-21T00:00:00Z