The role of TLR2 in vivo following challenge with Staphylococcus aureus and prototypic ligands
about
Evasion of Neutrophil Killing by Staphylococcus aureusRole of redox signaling in neuroinflammation and neurodegenerative diseasesNADPH oxidase-mediated redox signal contributes to lipoteichoic acid-induced MMP-9 upregulation in brain astrocytesLipoteichoic acid induces matrix metalloproteinase-9 expression via transactivation of PDGF receptors and NF-kappaB activation in rat brain astrocytesMast cells augment adaptive immunity by orchestrating dendritic cell trafficking through infected tissues.Vav1 is essential for mechanotactic crawling and migration of neutrophils out of the inflamed microvasculatureMast cell activation is characterized by upregulation of a functional anaphylatoxin C5a receptorFunctional basis for complement evasion by staphylococcal superantigen-like 7.Calmodulin kinase II-dependent transactivation of PDGF receptors mediates astrocytic MMP-9 expression and cell motility induced by lipoteichoic acidNeutrophil-derived IL-1β is sufficient for abscess formation in immunity against Staphylococcus aureus in miceBDCA1-positive dendritic cells (DCs) represent a unique human myeloid DC subset that induces innate and adaptive immune responses to Staphylococcus aureus Infection.A narrow repertoire of transcriptional modules responsive to pyogenic bacteria is impaired in patients carrying loss-of-function mutations in MYD88 or IRAK4Contribution of toll-like receptor 2 to the innate response against Staphylococcus aureus infection in mice.Plasticity in mast cell responses during bacterial infections.The Role of TLR2 in Infection and ImmunityToll-like receptor 2 ligand pretreatment attenuates retinal microglial inflammatory response but enhances phagocytic activity toward Staphylococcus aureus.Deciphering mechanisms of staphylococcal biofilm evasion of host immunity.Mast cell deficiency in Kit(W-sh) mice does not impair antibody-mediated arthritisStaphylococcal protein Ecb impairs complement receptor-1 mediated recognition of opsonized bacteria.Identification of single nucleotide polymorphisms in bovine CARD15 and their associations with health and production traits in Canadian Holsteins.Regulation of TLR2-mediated tolerance and cross-tolerance through IRAK4 modulation by miR-132 and miR-212.Immunopathogenesis of Staphylococcus aureus pulmonary infection.Pretreatment with the Gram-positive bacterial cell wall molecule peptidoglycan improves bacterial clearance and decreases inflammation and mortality in mice challenged with Pseudomonas aeruginosa.Pretreatment with the Gram-positive bacterial cell wall molecule peptidoglycan improves bacterial clearance and decreases inflammation and mortality in mice challenged with Staphylococcus aureus.Modulation of Stat-1 in Human Macrophages Infected with Different Species of Intracellular Pathogenic Bacteria.Mice that exclusively express TLR4 on endothelial cells can efficiently clear a lethal systemic Gram-negative bacterial infection.Ephedrine hydrochloride inhibits PGN-induced inflammatory responses by promoting IL-10 production and decreasing proinflammatory cytokine secretion via the PI3K/Akt/GSK3β pathway.The function of TLR2 during staphylococcal diseases.Toll-like receptor 2-mediated MAPKs and NF-κB activation requires the GNAO1-dependent pathway in human mast cells.Impact of thrombosis on pulmonary endothelial injury and repair following sepsis.Human peritoneal mesothelial cells respond to bacterial ligands through a specific subset of Toll-like receptors.The Staphylococcus aureus lipoprotein SitC colocalizes with Toll-like receptor 2 (TLR2) in murine keratinocytes and elicits intracellular TLR2 accumulation.Modulation of the local neutrophil response by a novel hyaluronic acid-binding peptide reduces bacterial burden during staphylococcal wound infection.Interferon-γ enhances both the anti-bacterial and the pro-inflammatory response of human mast cells to Staphylococcus aureus.Protective or deleterious role of scavenger receptors SR-A and CD36 on host resistance to Staphylococcus aureus depends on the site of infectionToll-like receptor 2 has a prominent but nonessential role in innate immunity to Staphylococcus aureus pneumonia.Neutrophil evasion strategies by Streptococcus pneumoniae and Staphylococcus aureus.Ephedrine hydrochloride protects mice from staphylococcus aureus-induced peritonitis.
P2860
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P2860
The role of TLR2 in vivo following challenge with Staphylococcus aureus and prototypic ligands
description
2006 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2006
@ast
im Dezember 2006 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2006/12/01)
@sk
vědecký článek publikovaný v roce 2006
@cs
wetenschappelijk artikel (gepubliceerd op 2006/12/01)
@nl
наукова стаття, опублікована в грудні 2006
@uk
مقالة علمية (نشرت في ديسمبر 2006)
@ar
name
The role of TLR2 in vivo follo ...... aureus and prototypic ligands
@ast
The role of TLR2 in vivo follo ...... aureus and prototypic ligands
@en
The role of TLR2 in vivo follo ...... aureus and prototypic ligands
@nl
type
label
The role of TLR2 in vivo follo ...... aureus and prototypic ligands
@ast
The role of TLR2 in vivo follo ...... aureus and prototypic ligands
@en
The role of TLR2 in vivo follo ...... aureus and prototypic ligands
@nl
prefLabel
The role of TLR2 in vivo follo ...... aureus and prototypic ligands
@ast
The role of TLR2 in vivo follo ...... aureus and prototypic ligands
@en
The role of TLR2 in vivo follo ...... aureus and prototypic ligands
@nl
P921
P3181
P1476
The role of TLR2 in vivo follo ...... aureus and prototypic ligands
@en
P2093
Paul Kubes
Sarah C. Mullaly
P304
P3181
P356
10.4049/JIMMUNOL.177.11.8154
P407
P577
2006-12-01T00:00:00Z