Altered properties of quantal neurotransmitter release at endplates of mice lacking P/Q-type Ca2+ channels - Wikidata - awgldk - TriplyDB

Altered properties of quantal neurotransmitter release at endplates of mice lacking P/Q-type Ca2+ channels

Altered properties of quantal neurotransmitter release at endplates of mice lacking P/Q-type Ca2+ channels

http://www.wikidata.org/entity/Q28594746

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Muscle channelopathies and critical points in functional and genetic studies.The Physiology, Pathology, and Pharmacology of Voltage-Gated Calcium Channels and Their Future Therapeutic Potential Gamma-band deficiency and abnormal thalamocortical activity in P/Q-type channel mutant mice Presynaptic BK channel localization is dependent on the hierarchical organization of alpha-catulin and dystrobrevin and fine-tuned by CaV2 calcium channels.Calcium channels and synaptic transmission in familial hemiplegic migraine type 1 animal models.Role of exercise in maintaining the integrity of the neuromuscular junction.Functional neuromuscular junctions formed by embryonic stem cell-derived motor neurons.Active zone protein Bassoon co-localizes with presynaptic calcium channel, modifies channel function, and recovers from aging related loss by exercise.Synaptic Pathophysiology and Treatment of Lambert-Eaton Myasthenic Syndrome.Age-dependent contribution of P/Q- and R-type Ca2+ channels to neuromuscular transmission in lethargic mice.Altered neurotransmitter release machinery in mice deficient for the deubiquitinating enzyme Usp14.Age and gender-dependent alternative splicing of P/Q-type calcium channel EF-hand.Increased asynchronous release and aberrant calcium channel activation in amyloid precursor protein deficient neuromuscular synapses.Altered short-term synaptic plasticity and reduced muscle strength in mice with impaired regulation of presynaptic CaV2.1 Ca2+ channels The P/Q-type voltage-dependent calcium channel: a therapeutic target in spinocerebellar ataxia type 6.Defects in Neuromuscular Transmission May Underlie Motor Dysfunction in Spinal and Bulbar Muscular Atrophy Recovery of mouse neuromuscular junctions from single and repeated injections of botulinum neurotoxin A Evaluation of a novel calcium channel agonist for therapeutic potential in Lambert-Eaton myasthenic syndrome Calcium channels link the muscle-derived synapse organizer laminin β2 to Bassoon and CAST/Erc2 to organize presynaptic active zones.Active zone density is conserved during synaptic growth but impaired in aged mice.Presynaptic homeostasis at CNS nerve terminals compensates for lack of a key Ca2+ entry pathway.Tick holocyclotoxins trigger host paralysis by presynaptic inhibition.Cav2-type calcium channels encoded by cac regulate AP-independent neurotransmitter release at cholinergic synapses in adult Drosophila brain.Regulation of glutamatergic and GABAergic neurotransmission in the chick nucleus laminaris: role of N-type calcium channels.Molecular mechanism of active zone organization at vertebrate neuromuscular junctions.Acid-sensing ion channels 1a (ASIC1a) inhibit neuromuscular transmission in female mice.Transsynaptic channelosomes: non-conducting roles of ion channels in synapse formation Active zones of mammalian neuromuscular junctions: formation, density, and aging.Organization and function of transmitter release sites at the neuromuscular junction.T-type channel-mediated neurotransmitter release.The Role of Calcium Channels in Epilepsy.Synaptic Vesicle Proteins and Active Zone Plasticity.The role of laminins in the organization and function of neuromuscular junctions.Amyotrophic lateral sclerosis-immunoglobulins selectively interact with neuromuscular junctions expressing P/Q-type calcium channels.Synaptic gain-of-function effects of mutant Cav2.1 channels in a mouse model of familial hemiplegic migraine are due to increased basal [Ca2+]i Muscle Contraction Regulates BDNF/TrkB Signaling to Modulate Synaptic Function through Presynaptic cPKCα and cPKCβI.Severe and progressive neurotransmitter release aberrations in familial hemiplegic migraine type 1 Cacna1a S218L knock-in mice.Developmental changes in release properties of the CA3-CA1 glutamate synapse in rat hippocampus.Presynaptic N-type and P/Q-type Ca2+ channels mediating synaptic transmission at the calyx of Held of mice.Dominant-negative effects of human P/Q-type Ca2+ channel mutations associated with episodic ataxia type 2.

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Altered properties of quantal neurotransmitter release at endplates of mice lacking P/Q-type Ca2+ channels

http://www.wikidata.org/entity/Q28594746

description

2003 թուականի Մարտին հրատարակուած գիտական յօդուած

@hyw

2003 թվականի մարտին հրատարակված գիտական հոդված

@hy

article publié dans les Procee ...... f the United States of America

@fr

artículu científicu espublizáu en 2003

@ast

im März 2003 veröffentlichter wissenschaftlicher Artikel

@de

scientific journal article

@en

vedecký článok (publikovaný 2003/03/18)

@sk

vědecký článek publikovaný v roce 2003

@cs

wetenschappelijk artikel (gepubliceerd op 2003/03/18)

@nl

наукова стаття, опублікована в березні 2003

@uk

name

Altered properties of quantal ...... lacking P/Q-type Ca2+ channels

@ast

Altered properties of quantal ...... lacking P/Q-type Ca2+ channels

@en

Altered properties of quantal ...... lacking P/Q-type Ca2+ channels

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type

label

Altered properties of quantal ...... lacking P/Q-type Ca2+ channels

@ast

Altered properties of quantal ...... lacking P/Q-type Ca2+ channels

@en

Altered properties of quantal ...... lacking P/Q-type Ca2+ channels

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prefLabel

Altered properties of quantal ...... lacking P/Q-type Ca2+ channels

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Altered properties of quantal ...... lacking P/Q-type Ca2+ channels

@en

Altered properties of quantal ...... lacking P/Q-type Ca2+ channels

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PNAS.0437991100

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Proceedings of the National Academy of Sciences of the United States of America

P1476

Altered properties of quantal ...... lacking P/Q-type Ca2+ channels

@en

P2093

Francisco J Urbano

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Hee-Sup Shin

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Kisun Jun

http://www.w3.org/2001/XMLSchema#string

Richard W Tsien

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P2860

Synaptic vesicle exocytosis captured by quick freezing and correlated with quantal transmitter release

Development of the vertebrate neuromuscular junction

Induction, assembly, maturation and maintenance of a postsynaptic apparatus

alpha(1E) subunits form the pore of three cerebellar R-type calcium channels with different pharmacological and permeation properties

Dystonia and cerebellar atrophy in Cacna1a null mice lacking P/Q calcium channel activity

Functional disorders of the sympathetic nervous system in mice lacking the alpha 1B subunit (Cav 2.2) of N-type calcium channels

Abnormal transmitter release at neuromuscular junctions of mice carrying the tottering alpha(1A) Ca(2+) channel mutation

Distinct phenotypes of mutant mice lacking agrin, MuSK, or rapsyn

Ablation of P/Q-type Ca(2+) channel currents, altered synaptic transmission, and progressive ataxia in mice lacking the alpha(1A)-subunit

The architecture of active zone material at the frog's neuromuscular junction.

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3491-3496

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scholarly article

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172760

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10.1073/PNAS.0437991100

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6

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100

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Erika Piedras-renteria

Osvaldo D Uchitel

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2003-03-06T00:00:00Z

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8050902

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12624181

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neurotransmitter

Calcium channel, voltage-dependent, P/Q type, alpha 1A subunit

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152320

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