Exit from pluripotency is gated by intracellular redistribution of the bHLH transcription factor Tfe3 - Wikidata - awgldk - TriplyDB

Exit from pluripotency is gated by intracellular redistribution of the bHLH transcription factor Tfe3

Exit from pluripotency is gated by intracellular redistribution of the bHLH transcription factor Tfe3

http://www.wikidata.org/entity/Q28594750

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From blastocyst to gastrula: gene regulatory networks of embryonic stem cells and early mouse embryogenesis Ex uno plures: molecular designs for embryonic pluripotency The role of pluripotency gene regulatory network components in mediating transitions between pluripotent cell states Folliculin regulates ampk-dependent autophagy and metabolic stress survival Expression and knockdown of zebrafish folliculin suggests requirement for embryonic brain morphogenesis.Mourning Dr. Alfred G. Knudson: the two-hit hypothesis, tumor suppressor genes, and the tuberous sclerosis complex TFEB at a glance Molecular basis of embryonic stem cell self-renewal: from signaling pathways to pluripotency network Beyond indigestion: emerging roles for lysosome-based signaling in human disease Genetic exploration of the exit from self-renewal using haploid embryonic stem cells Zeb2 Regulates Cell Fate at the Exit from Epiblast State in Mouse Embryonic Stem Cells Tracking the embryonic stem cell transition from ground state pluripotency.TFE3 regulates whole-body energy metabolism in cooperation with TFEB.Metabolic regulator Fnip1 is crucial for iNKT lymphocyte development.A mutation in the canine gene encoding folliculin-interacting protein 2 (FNIP2) associated with a unique disruption in spinal cord myelination.Topical rapamycin as a treatment for fibrofolliculomas in Birt-Hogg-Dubé syndrome: a double-blind placebo-controlled randomized split-face trial.In vitro differentiation of human embryonic stem cells into ovarian follicle-like cells.Otx2 and Oct4 drive early enhancer activation during embryonic stem cell transition from naive pluripotency.Stochastic simulation of notch signaling reveals novel factors that mediate the differentiation of neural stem cells.Resetting transcription factor control circuitry toward ground-state pluripotency in human.Folliculin (Flcn) inactivation leads to murine cardiac hypertrophy through mTORC1 deregulation Cellular differences in protein synthesis regulate tissue homeostasis.Foxd3 Promotes Exit from Naive Pluripotency through Enhancer Decommissioning and Inhibits Germline Specification Serum Inter-α-inhibitor activates the Yes tyrosine kinase and YAP/TEAD transcriptional complex in mouse embryonic stem cells.Folliculin regulates cell-cell adhesion, AMPK, and mTORC1 in a cell-type-specific manner in lung-derived cells.Pluripotent states of human embryonic stem cells Folliculin-interacting proteins Fnip1 and Fnip2 play critical roles in kidney tumor suppression in cooperation with Flcn.Distribution Analyzer, a methodology for identifying and clustering outlier conditions from single-cell distributions, and its application to a Nanog reporter RNAi screen.How are pluripotent cells captured in culture?Pluripotency and Epigenetic Factors in Mouse Embryonic Stem Cell Fate Regulation Hmga2 is necessary for Otx2-dependent exit of embryonic stem cells from the pluripotent ground state The roles of ERAS during cell lineage specification of mouse early embryonic development.Aberrant differentiation of Tsc2-deficient teratomas associated with activation of the mTORC1-TFE3 pathway Generation of Naivetropic Induced Pluripotent Stem Cells from Parkinson's Disease Patients for High-Efficiency Genetic Manipulation and Disease Modeling Clinical Features, Genetics and Potential Therapeutic Approaches for Birt-Hogg-Dubé Syndrome.A draft map of the mouse pluripotent stem cell spatial proteome Convergence of cMyc and β-catenin on Tcf7l1 enables endoderm specification.TFEB and TFE3 are novel components of the integrated stress response.AMPK governs lineage specification through Tfeb-dependent regulation of lysosomes.Pluripotency-associated miR-290/302 family of microRNAs promote the dismantling of naive pluripotency.

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P2860

Exit from pluripotency is gated by intracellular redistribution of the bHLH transcription factor Tfe3

http://www.wikidata.org/entity/Q28594750

description

2013 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2013 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

artículu científicu espublizáu en 2013

@ast

im April 2013 veröffentlichter wissenschaftlicher Artikel

@de

scientific journal article

@en

vedecký článok (publikovaný 2013/04/11)

@sk

vědecký článek publikovaný v roce 2013

@cs

wetenschappelijk artikel (gepubliceerd op 2013/04/11)

@nl

наукова стаття, опублікована у квітні 2013

@uk

مقالة علمية (نشرت في 11-4-2013)

@ar

name

Exit from pluripotency is gate ...... bHLH transcription factor Tfe3

@ast

Exit from pluripotency is gate ...... bHLH transcription factor Tfe3

@en

Exit from pluripotency is gate ...... bHLH transcription factor Tfe3

@nl

type

label

Exit from pluripotency is gate ...... bHLH transcription factor Tfe3

@ast

Exit from pluripotency is gate ...... bHLH transcription factor Tfe3

@en

Exit from pluripotency is gate ...... bHLH transcription factor Tfe3

@nl

prefLabel

Exit from pluripotency is gate ...... bHLH transcription factor Tfe3

@ast

Exit from pluripotency is gate ...... bHLH transcription factor Tfe3

@en

Exit from pluripotency is gate ...... bHLH transcription factor Tfe3

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J.CELL.2013.03.012

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Exit from pluripotency is gate ...... bHLH transcription factor Tfe3

@en

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Austin Smith

http://www.w3.org/2001/XMLSchema#string

Jennifer Nichols

http://www.w3.org/2001/XMLSchema#string

Joerg Betschinger

http://www.w3.org/2001/XMLSchema#string

Patrick J. Paddison

http://www.w3.org/2001/XMLSchema#string

Philip D. Corrin

http://www.w3.org/2001/XMLSchema#string

Sabine Dietmann

http://www.w3.org/2001/XMLSchema#string

P2860

Gene function in early mouse embryonic stem cell differentiation.

Folliculin encoded by the BHD gene interacts with a binding protein, FNIP1, and AMPK, and is involved in AMPK and mTOR signaling

Interaction of folliculin (Birt-Hogg-Dubé gene product) with a novel Fnip1-like (FnipL/Fnip2) protein

Identification and characterization of a novel folliculin-interacting protein FNIP2

Inactivation of the FLCN tumor suppressor gene induces TFE3 transcriptional activity by increasing its nuclear localization

Mitf and Tfe3, two members of the Mitf-Tfe family of bHLH-Zip transcription factors, have important but functionally redundant roles in osteoclast development.

mTOR signaling in growth control and disease

Tcf3: a transcriptional regulator of axis induction in the early embryo

Mutations in a novel gene lead to kidney tumors, lung wall defects, and benign tumors of the hair follicle in patients with the Birt-Hogg-Dubé syndrome

Transcriptional regulation of the melanoma prognostic marker melastatin (TRPM1) by MITF in melanocytes and melanoma

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335–347

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scholarly article

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156590

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10.1016/J.CELL.2013.03.012

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2

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236198700

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23582324

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Folliculin interacting protein 1

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3661979

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