CD25+CD4+ cells contribute to Th2 polarization during helminth infection by suppressing Th1 response development
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Regulatory T cell suppressive potency dictates the balance between bacterial proliferation and clearance during persistent Salmonella infectionCurrent status of vaccines for schistosomiasisParasitic worms and inflammatory diseasesTregs in infection and vaccinology: heroes or traitors?Mechanisms underlying the induction of regulatory T cells and its relevance in the adaptive immune response in parasitic infectionsProtection and pathology during parasite infection: IL-10 strikes the balanceModulation of specific and allergy-related immune responses by helminthsInhibition of Granulomatous Inflammation and Prophylactic Treatment of Schistosomiasis with a Combination of Edelfosine and PraziquantelMetabolic and Phenotypic Differences between Mice Producing a Werner Syndrome Helicase Mutant Protein and Wrn Null MiceS. mansoni bolsters anti-viral immunity in the murine respiratory tractThe beneficial helminth parasite?Cytokine responses to Schistosoma haematobium in a Zimbabwean population: contrasting profiles for IFN-gamma, IL-4, IL-5 and IL-10 with ageTaenia crassiceps infection attenuates multiple low-dose streptozotocin-induced diabetes.Chronic helminth infections protect against allergic diseases by active regulatory processesImmune modulation by Schistosoma mansoni antigens in NOD mice: effects on both innate and adaptive immune systems.Elimination of helminth infection restores HIV-1C vaccine-specific T cell responses independent of helminth-induced IL-10Host cytokine production, lymphoproliferation, and antibody responses during the course of Ancylostoma ceylanicum infection in the Golden Syrian hamster.Regulatory and activated T cells in human Schistosoma haematobium infections.Impact of Schistosoma japonicum infection on collagen-induced arthritis in DBA/1 mice: a murine model of human rheumatoid arthritisHeligmosomoides polygyrus abrogates antigen-specific gut injury in a murine model of inflammatory bowel disease.IL-10 and TGF-beta redundantly protect against severe liver injury and mortality during acute schistosomiasisHelminth-primed dendritic cells alter the host response to enteric bacterial infectionRole of return migration in the emergence of multiple sclerosis in the French West Indies.Helicobacter infection and gastric neoplasia.Protection of Tregs, suppression of Th1 and Th17 cells, and amelioration of experimental allergic encephalomyelitis by a physically-modified saline.Worms and allergy.Regulatory T-cell expansion during chronic viral infection is dependent on endogenous retroviral superantigens.Mechanisms of disease: the hygiene hypothesis revisited.Modulation of innate antigen-presenting cell function by pre-patent schistosome infectionCinnamon ameliorates experimental allergic encephalomyelitis in mice via regulatory T cells: implications for multiple sclerosis therapy.The novel CD4+CD25+ regulatory T cell effector molecule fibrinogen-like protein 2 contributes to the outcome of murine fulminant viral hepatitis.NOD dendritic cells stimulated with Lactobacilli preferentially produce IL-10 versus IL-12 and decrease diabetes incidenceCCR8 is expressed by antigen-elicited, IL-10-producing CD4+CD25+ T cells, which regulate Th2-mediated granuloma formation in miceMycobacterium bovis BCG infection severely delays Trichuris muris expulsion and co-infection suppresses immune responsiveness to both pathogens.Preexisting helminth infection induces inhibition of innate pulmonary anti-tuberculosis defense by engaging the IL-4 receptor pathway.Exposure, infection, systemic cytokine levels and antibody responses in young children concurrently exposed to schistosomiasis and malaria.Helminth-induced CD19+CD23hi B cells modulate experimental allergic and autoimmune inflammation.T regulatory cells: aid or hindrance in the clearance of disease?Helminth Infection and Commensal Microbiota Drive Early IL-10 Production in the Skin by CD4+ T Cells That Are Functionally Suppressive.Helminthic infection and the risk of neurologic disease progression in HTLV-1
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P2860
CD25+CD4+ cells contribute to Th2 polarization during helminth infection by suppressing Th1 response development
description
2004 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2004
@ast
im Juli 2004 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2004/07/15)
@sk
vědecký článek publikovaný v roce 2004
@cs
wetenschappelijk artikel (gepubliceerd op 2004/07/15)
@nl
наукова стаття, опублікована в липні 2004
@uk
مقالة علمية (نشرت في 15-7-2004)
@ar
name
CD25+CD4+ cells contribute to ...... ssing Th1 response development
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CD25+CD4+ cells contribute to ...... ssing Th1 response development
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CD25+CD4+ cells contribute to ...... ssing Th1 response development
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type
label
CD25+CD4+ cells contribute to ...... ssing Th1 response development
@ast
CD25+CD4+ cells contribute to ...... ssing Th1 response development
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CD25+CD4+ cells contribute to ...... ssing Th1 response development
@nl
prefLabel
CD25+CD4+ cells contribute to ...... ssing Th1 response development
@ast
CD25+CD4+ cells contribute to ...... ssing Th1 response development
@en
CD25+CD4+ cells contribute to ...... ssing Th1 response development
@nl
P921
P3181
P1476
CD25+CD4+ cells contribute to ...... ssing Th1 response development
@en
P2093
Amy S. McKee
Edward J. Pearce
P304
P3181
P356
10.4049/JIMMUNOL.173.2.1224
P407
P5008
P577
2004-07-15T00:00:00Z