MARCO is the major binding receptor for unopsonized particles and bacteria on human alveolar macrophages - Wikidata - awgldk - TriplyDB

MARCO is the major binding receptor for unopsonized particles and bacteria on human alveolar macrophages

MARCO is the major binding receptor for unopsonized particles and bacteria on human alveolar macrophages

http://www.wikidata.org/entity/Q28677922

about

Tempering allorecognition to induce transplant tolerance with chemically modified apoptotic donor cells Therapeutic inflammatory monocyte modulation using immune-modifying microparticles Silica binding and toxicity in alveolar macrophages Contrasting macrophage activation by fine and ultrafine titanium dioxide particles is associated with different uptake mechanisms Characterization of a newly developed chicken 44K Agilent microarray.Heterogeneity in macrophage phagocytosis of Staphylococcus aureus strains: high-throughput scanning cytometry-based analysis Targeting MARCO can lead to enhanced dendritic cell motility and anti-melanoma activity Evaluation of the relationship between MARCO and CD36 single-nucleotide polymorphisms and susceptibility to pulmonary tuberculosis in a Chinese Han population.Role of lysosomes in silica-induced inflammasome activation and inflammation in absence of MARCO.A subpopulation of CD163-positive macrophages is classically activated in psoriasis.Scavenger receptors and their potential as therapeutic targets in the treatment of cardiovascular disease Scavenger receptor-A (CD204): a two-edged sword in health and disease VtaA8 and VtaA9 from Haemophilus parasuis delay phagocytosis by alveolar macrophages.Determinants of host susceptibility to murine respiratory syncytial virus (RSV) disease identify a role for the innate immunity scavenger receptor MARCO gene in human infants Examination of MARCO activity on dendritic cell phenotype and function using a gene knockout mouse Macrophage immunoregulatory pathways in tuberculosis.The scavenger receptor MARCO modulates TLR-induced responses in dendritic cells.MARCOing monocytes for elimination.Functional consequences for primary human alveolar macrophages following treatment with long, but not short, multiwalled carbon nanotubes.Protection against inhaled oxidants through scavenging of oxidized lipids by macrophage receptors MARCO and SR-AI/II In vivo uptake of inhaled particles by airway phagocytes is enhanced in patients with mild asthma compared with normal volunteers.MARCO regulates early inflammatory responses against influenza: a useful macrophage function with adverse outcome Allergic Lung Inflammation Reduces Tissue Invasion and Enhances Survival from Pulmonary Pneumococcal Infection in Mice, Which Correlates with Increased Expression of Transforming Growth Factor β1 and SiglecF(low) Alveolar Macrophages.Alternative activation of macrophages and pulmonary fibrosis are modulated by scavenger receptor, macrophage receptor with collagenous structure.TRPV4 Mechanosensitive Ion Channel Regulates Lipopolysaccharide-Stimulated Macrophage Phagocytosis.MARCO is required for TLR2- and Nod2-mediated responses to Streptococcus pneumoniae and clearance of pneumococcal colonization in the murine nasopharynx.The biodegradability of electrospun Dextran/PLGA scaffold in a fibroblast/macrophage co-culture.Characterization of immortalized MARCO and SR-AI/II-deficient murine alveolar macrophage cell lines Signaling pathways required for macrophage scavenger receptor-mediated phagocytosis: analysis by scanning cytometry Role of macrophage receptor with collagenous structure in innate immune tolerance.HSV-1 exploits the innate immune scavenger receptor MARCO to enhance epithelial adsorption and infection.The macrophage scavenger receptor SR-AI/II and lung defense against pneumococci and particles Role of scavenger receptor a family in lung inflammation from exposure to environmental particles.Differential binding of inorganic particles to MARCO Exploiting apoptosis for therapeutic tolerance induction.Divergence of macrophage phagocytic and antimicrobial programs in leprosy Roles of oxidative stress in signaling and inflammation induced by particulate matter.Biomarkers in the diagnosis of aspiration syndromes.A mechanistic review of silica-induced inhalation toxicity.Immunomodulators targeting MARCO expression improve resistance to postinfluenza bacterial pneumonia.

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P2860

MARCO is the major binding receptor for unopsonized particles and bacteria on human alveolar macrophages

http://www.wikidata.org/entity/Q28677922

description

2005 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2005 թվականի նոյեմբերին հրատարակված գիտական հոդված

@hy

artículu científicu espublizáu en 2005

@ast

im November 2005 veröffentlichter wissenschaftlicher Artikel

@de

scientific journal article

@en

vedecký článok (publikovaný 2005/11/01)

@sk

vědecký článek publikovaný v roce 2005

@cs

wetenschappelijk artikel (gepubliceerd op 2005/11/01)

@nl

наукова стаття, опублікована в листопаді 2005

@uk

مقالة علمية (نشرت في نوفمبر 2005)

@ar

name

MARCO is the major binding rec ...... on human alveolar macrophages

@ast

MARCO is the major binding rec ...... on human alveolar macrophages

@en

MARCO is the major binding rec ...... on human alveolar macrophages

@nl

type

label

MARCO is the major binding rec ...... on human alveolar macrophages

@ast

MARCO is the major binding rec ...... on human alveolar macrophages

@en

MARCO is the major binding rec ...... on human alveolar macrophages

@nl

prefLabel

MARCO is the major binding rec ...... on human alveolar macrophages

@ast

MARCO is the major binding rec ...... on human alveolar macrophages

@en

MARCO is the major binding rec ...... on human alveolar macrophages

@nl

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JIMMUNOL.175.9.6058

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Journal of Immunology

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MARCO is the major binding rec ...... on human alveolar macrophages

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P2093

Aiyappa Palecanda

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Amy Imrich

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Henry Koziel

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Karl Tryggvason

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Lester Kobzik

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Marko Sankala

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Mohamed S. Arredouani

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Motohiro Takeya

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Sara O. Vargas

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Timo Pikkarainen

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6058–6064

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scholarly article

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4306128

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10.4049/JIMMUNOL.175.9.6058

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2005-11-01T00:00:00Z

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