Novel sulfated polysaccharides disrupt cathelicidins, inhibit RAGE and reduce cutaneous inflammation in a mouse model of rosacea - Wikidata - awgldk - TriplyDB

Novel sulfated polysaccharides disrupt cathelicidins, inhibit RAGE and reduce cutaneous inflammation in a mouse model of rosacea

Novel sulfated polysaccharides disrupt cathelicidins, inhibit RAGE and reduce cutaneous inflammation in a mouse model of rosacea

http://www.wikidata.org/entity/Q28742923

about

Rosacea: The cytokine and chemokine network Major pathophysiological correlations of rosacea: a complete clinical appraisal Prevention of anti-microbial peptide LL-37-induced apoptosis and ATP release in the urinary bladder by a modified glycosaminoglycan Recent advances in the understanding and management of rosacea.Isolevuglandin-type lipid aldehydes induce the inflammatory response of macrophages by modifying phosphatidylethanolamines and activating the receptor for advanced glycation endproducts.Distribution and expression of non-neuronal transient receptor potential (TRPV) ion channels in rosacea A Modified Glycosaminoglycan, GM-0111, Inhibits Molecular Signaling Involved in Periodontitis.Pharmacological control of receptor of advanced glycation end-products and its biological effects in psoriasis.Physiological relevance of LL-37 induced bladder inflammation and mast cells.Topical cathelicidin (LL-37) an innate immune peptide induces acute olfactory epithelium inflammation in a mouse model.DAMPs activating innate and adaptive immune responses in COPD.Endoplasmic reticulum stress: key promoter of rosacea pathogenesis.LL-37 causes cell death of human nasal epithelial cells, which is inhibited with a synthetic glycosaminoglycan.Targeting the Receptor for Advanced Glycation Endproducts (RAGE): A Medicinal Chemistry Perspective.Inhibition of the receptor for advanced glycation end-products (RAGE) protects from secondhand smoke (SHS)-induced intrauterine growth restriction IUGR in mice.A tunable hydrogel system for long-term release of cell-secreted cytokines and bioprinted in situ wound cell delivery.Prevention of sinonasal inflammation by a synthetic glycosaminoglycan.Sulfated hyaluronan attenuates inflammatory signaling pathways in macrophages involving induction of antioxidants.Recombinant erythroid differentiation regulator 1 inhibits both inflammation and angiogenesis in a mouse model of rosacea.A synthetic glycosaminoglycan reduces sinonasal inflammation in a murine model of chronic rhinosinusitis

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P2860

Novel sulfated polysaccharides disrupt cathelicidins, inhibit RAGE and reduce cutaneous inflammation in a mouse model of rosacea

http://www.wikidata.org/entity/Q28742923

description

2011 nî lūn-bûn

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2011 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի փետրվարին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Novel sulfated polysaccharides ...... on in a mouse model of rosacea

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Novel sulfated polysaccharides ...... on in a mouse model of rosacea

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Novel sulfated polysaccharides ...... on in a mouse model of rosacea

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Novel sulfated polysaccharides ...... on in a mouse model of rosacea

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JOURNAL.PONE.0016658

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Novel sulfated polysaccharides ...... on in a mouse model of rosacea

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Brian Argyle

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Gerald Krueger

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Glenn D Prestwich

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Jianxing Zhang

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Lindsi McCoard

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Narayanam V Rao

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Thomas P Kennedy

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William J Rusho

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Xiaoyu Xu

http://www.w3.org/2001/XMLSchema#string

P2860

Chemotactic activity of S100A7 (Psoriasin) is mediated by the receptor for advanced glycation end products and potentiates inflammation with highly homologous but functionally distinct S100A15

Heparin's anti-inflammatory effects require glucosamine 6-O-sulfation and are mediated by blockade of L- and P-selectins

Contaminated heparin associated with adverse clinical events and activation of the contact system

HMGB1: endogenous danger signaling

The 1.5 A Crystal Structure of Human Receptor for Advanced Glycation Endproducts (RAGE) Ectodomains Reveals Unique Features Determining Ligand Binding

Increased serine protease activity and cathelicidin promotes skin inflammation in rosacea

Autoactivation of human Hageman factor. Demonstration utilizing a synthetic substrate

Enhanced transdermal delivery of low molecular weight heparin by barrier perturbation.

Low anticoagulant heparin targets multiple sites of inflammation, suppresses heparin-induced thrombocytopenia, and inhibits interaction of RAGE with its ligands.

TLR4-mediated skin carcinogenesis is dependent on immune and radioresistant cells.

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49968044

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21347371

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polysaccharides

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