Salmonella enterica serovar Typhimurium periplasmic superoxide dismutase SodCI is a member of the PhoPQ regulon and is induced in macrophages

Salmonella enterica serovar Typhimurium periplasmic superoxide dismutase SodCI is a member of the PhoPQ regulon and is induced in macrophages

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http://www.wikidata.org/entity/Q28766733
The TolC protein of Legionella pneumophila plays a major role in multi-drug resistance and the early steps of host invasionStructural properties of periplasmic SodCI that correlate with virulence in Salmonella enterica serovar TyphimuriumNitric oxide antagonizes the acid tolerance response that protects Salmonella against innate gastric defenses.Protecting against antimicrobial effectors in the phagosome allows SodCII to contribute to virulence in Salmonella enterica serovar TyphimuriumThe broadly conserved regulator PhoP links pathogen virulence and membrane potential in Escherichia coliEither periplasmic tethering or protease resistance is sufficient to allow a SodC to protect Salmonella enterica serovar Typhimurium from phagocytic superoxide.Periplasmic superoxide dismutase SodCI of Salmonella binds peptidoglycan to remain tethered within the periplasm.Regulatory and structural differences in the Cu,Zn-superoxide dismutases of Salmonella enterica and their significance for virulence.Probing the ArcA regulon under aerobic/ROS conditions in Salmonella enterica serovar Typhimurium.Protective Immunity Elicited by Oral Immunization of Mice with Salmonella enterica Serovar Typhimurium Braun Lipoprotein (Lpp) and Acetyltransferase (MsbB) Mutants.Codependent and independent effects of nitric oxide-mediated suppression of PhoPQ and Salmonella pathogenicity island 2 on intracellular Salmonella enterica serovar typhimurium survival.Mechanisms used by virulent Salmonella to impair dendritic cell function and evade adaptive immunity.FliZ regulates expression of the Salmonella pathogenicity island 1 invasion locus by controlling HilD protein activity in Salmonella enterica serovar typhimurium.Magnesium homeostasis protects Salmonella against nitrooxidative stress.Cytoplasmic Copper Detoxification in Salmonella Can Contribute to SodC Metalation but Is Dispensable during Systemic Infection.Hotspot mutations and ColE1 plasmids contribute to the fitness of Salmonella Heidelberg in poultry litter
P2860

Salmonella enterica serovar Typhimurium periplasmic superoxide dismutase SodCI is a member of the PhoPQ regulon and is induced in macrophages

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http://www.wikidata.org/entity/Q28766733
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2006 nî lūn-bûn
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2006 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
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2006 թվականի նոյեմբերին հրատարակված գիտական հոդված
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2006年の論文
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2006年論文
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2006年論文
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2006年論文
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2006年論文
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2006年論文
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2006年论文
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Salmonella enterica serovar Ty ...... and is induced in macrophages
@ast
Salmonella enterica serovar Ty ...... and is induced in macrophages
@en
Salmonella enterica serovar Ty ...... and is induced in macrophages
@nl
type
label
Salmonella enterica serovar Ty ...... and is induced in macrophages
@ast
Salmonella enterica serovar Ty ...... and is induced in macrophages
@en
Salmonella enterica serovar Ty ...... and is induced in macrophages
@nl
prefLabel
Salmonella enterica serovar Ty ...... and is induced in macrophages
@ast
Salmonella enterica serovar Ty ...... and is induced in macrophages
@en
Salmonella enterica serovar Ty ...... and is induced in macrophages
@nl
P1433
P1476
P2093
P2860
P304
P31
P3181
P356
P407
P433
P478
P577
P698
P921
P932
P3181
P356
P1433
P1476
Salmonella enterica serovar Ty ...... and is induced in macrophages
@en
P2093
P2860
P304
P31
P3181
P356
P407
P433
P478
P577
P698
P921
P932