The twin arginine translocation system is essential for virulence of Yersinia pseudotuberculosis.
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Comparative Genomic Analyses of Streptococcus pseudopneumoniae Provide Insight into Virulence and Commensalism DynamicsEnvironmental Regulation of Yersinia PathophysiologyStructure and function of cytidine monophosphate kinase from Yersinia pseudotuberculosis, essential for virulence but not for survivalBacterial Secretion Systems: An OverviewThe twin-arginine translocation pathway in α-proteobacteria is functionally preserved irrespective of genomic and regulatory divergenceIdentification of functional Tat signal sequences in Mycobacterium tuberculosis proteinsThe genome of Streptococcus mitis B6--what is a commensal?Pleiotropic effects of the twin-arginine translocation system on biofilm formation, colonization, and virulence in Vibrio cholerae.Abrogation of the twin arginine transport system in Salmonella enterica serovar Typhimurium leads to colonization defects during infection.A Yersinia pestis tat mutant is attenuated in bubonic and small-aerosol pneumonic challenge models of infection but not as attenuated by intranasal challengeSalmonella enterica serovar Enteritidis tatB and tatC mutants are impaired in Caco-2 cell invasion in vitro and show reduced systemic spread in chickens.Identification and evaluation of twin-arginine translocase inhibitorsFunctional characterization of Edwardsiella tarda twin-arginine translocation system and its potential use as biological containment in live attenuated vaccine of marine fish.Diversity and evolution of bacterial twin arginine translocase protein, TatC, reveals a protein secretion system that is evolving to fit its environmental nichePathogenesis of Y. enterocolitica and Y. pseudotuberculosis in Human Yersiniosis.Twin arginine translocation in Yersinia.Twin-arginine translocation system (tat) mutants of Salmonella are attenuated due to envelope defects, not respiratory defectsTranscriptome analysis of Vibrio parahaemolyticus in type III secretion system 1 inducing conditions.Twin-arginine translocation system in Helicobacter pylori: TatC, but not TatB, is essential for viability.Synthetic Cyclic Peptomers as Type III Secretion System Inhibitors.The Tat Substrate SufI Is Critical for the Ability of Yersinia pseudotuberculosis To Cause Systemic Infection.Transcriptomic and Phenotypic Analysis Reveals New Functions for the Tat Pathway in Yersinia pseudotuberculosis.The dual transcriptional regulator RovM regulates the expression of AR3- and T6SS4-dependent acid survival systems in response to nutritional status in Yersinia pseudotuberculosis.Genomic Comparisons Reveal Microevolutionary Differences in Mycobacterium abscessus Subspecies.Tat-exported peptidoglycan amidase-dependent cell division contributes to Salmonella Typhimurium fitness in the inflamed gutCitrobacter freundii fitness during bloodstream infection
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P2860
The twin arginine translocation system is essential for virulence of Yersinia pseudotuberculosis.
description
2006 nî lūn-bûn
@nan
2006 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի մարտին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
The twin arginine translocatio ...... f Yersinia pseudotuberculosis.
@ast
The twin arginine translocatio ...... f Yersinia pseudotuberculosis.
@en
type
label
The twin arginine translocatio ...... f Yersinia pseudotuberculosis.
@ast
The twin arginine translocatio ...... f Yersinia pseudotuberculosis.
@en
prefLabel
The twin arginine translocatio ...... f Yersinia pseudotuberculosis.
@ast
The twin arginine translocatio ...... f Yersinia pseudotuberculosis.
@en
P2093
P2860
P921
P1476
The twin arginine translocatio ...... f Yersinia pseudotuberculosis.
@en
P2093
Ake Forsberg
Jeanette E Bröms
Moa Lavander
Solveig K Ericsson
P2860
P304
P356
10.1128/IAI.74.3.1768-1776.2006
P407
P577
2006-03-01T00:00:00Z