Defects in dermal Vγ4 γ δ T cells result in delayed wound healing in diabetic mice
about
IL-15 Enhances Activation and IGF-1 Production of Dendritic Epidermal T Cells to Promote Wound Healing in Diabetic Mice.Dermal Vγ4+T cells enhance the IMQ-induced psoriasis-like skin inflammatidon in re-challenged mice.Vγ4 T Cells Inhibit the Pro-healing Functions of Dendritic Epidermal T Cells to Delay Skin Wound Closure Through IL-17A.Functions of Vγ4 T Cells and Dendritic Epidermal T Cells on Skin Wound Healing.
P2860
Defects in dermal Vγ4 γ δ T cells result in delayed wound healing in diabetic mice
description
2016 nî lūn-bûn
@nan
2016 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2016 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
name
Defects in dermal Vγ4 γ δ T cells result in delayed wound healing in diabetic mice
@ast
Defects in dermal Vγ4 γ δ T cells result in delayed wound healing in diabetic mice
@en
type
label
Defects in dermal Vγ4 γ δ T cells result in delayed wound healing in diabetic mice
@ast
Defects in dermal Vγ4 γ δ T cells result in delayed wound healing in diabetic mice
@en
prefLabel
Defects in dermal Vγ4 γ δ T cells result in delayed wound healing in diabetic mice
@ast
Defects in dermal Vγ4 γ δ T cells result in delayed wound healing in diabetic mice
@en
P2093
P2860
P1476
Defects in dermal Vγ4 γ δ T cells result in delayed wound healing in diabetic mice
@en
P2093
Guangping Liang
Jingling Zhao
Jinming Tang
Shaohai Qi
Weifeng He
P2860
P304
P407
P50
P577
2016-06-15T00:00:00Z