DNA methylation: a mechanism linking environmental chemical exposures to risk of autism spectrum disorders? - Wikidata - awgldk - TriplyDB

DNA methylation: a mechanism linking environmental chemical exposures to risk of autism spectrum disorders?

DNA methylation: a mechanism linking environmental chemical exposures to risk of autism spectrum disorders?

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Piecing together a different picture: A host of new studies on autism have begun decoding the longstanding puzzle of its causes.Drinking Water and the Developing Brain.Associations of endocrine stress-related gene polymorphisms with risk of autism spectrum disorders: Evidence from an integrated meta-analysis.Environmental Induction of Neurodevelopmental Disorders.Microbiome, inflammation, epigenetic alterations, and mental diseases.Special Issue Introduction: Role of Epigenetic Gene Regulation in Brain Function Prenatal influences on temperament development: The role of environmental epigenetics.Genetics and epigenetics of autism: A Review.Are endocrine disrupting compounds environmental risk factors for autism spectrum disorder?Environmental Enrichment Improves Cognitive Deficits, AD Hallmarks and Epigenetic Alterations Presented in 5xFAD Mouse Model

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DNA methylation: a mechanism linking environmental chemical exposures to risk of autism spectrum disorders?

http://www.wikidata.org/entity/Q28833595

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2016 nî lūn-bûn

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2016 թուականի Մարտին հրատարակուած գիտական յօդուած

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Environmental Epigenetics

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Kimberly P Keil

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Pamela J Lein

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P2860

Why are autism spectrum conditions more prevalent in males?

DNA methyltransferases Dnmt3a and Dnmt3b are essential for de novo methylation and mammalian development

Epigenetic regulation of gene expression: how the genome integrates intrinsic and environmental signals

Lead and PCBs as risk factors for attention deficit/hyperactivity disorder

Advances in autism

Advances in autism genetics: on the threshold of a new neurobiology

Rett syndrome is caused by mutations in X-linked MECP2, encoding methyl-CpG-binding protein 2

Conversion of 5-methylcytosine to 5-hydroxymethylcytosine in mammalian DNA by MLL partner TET1

Developmental exposure to estradiol and bisphenol A increases susceptibility to prostate carcinogenesis and epigenetically regulates phosphodiesterase type 4 variant 4

The genetic and neurobiologic compass points toward common signaling dysfunctions in autism spectrum disorders

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Autism spectrum

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