Infections of People with Complement Deficiencies and Patients Who Have Undergone Splenectomy
about
Atypical hemolytic uremic syndromeMeningococcal VaccinationsBacterial Infections Following Splenectomy for Malignant and Nonmalignant Hematologic DiseasesMembrane attack by complement: the assembly and biology of terminal complement complexesPrevention of infections during primary immunodeficiencyImportance of circulating antibodies in protection against meningococcal diseaseThe spleen in local and systemic regulation of immunityStructural basis for sialic acid-mediated self-recognition by complement factor HA systematic review of eculizumab for atypical haemolytic uraemic syndrome (aHUS)Resistance of Neisseria gonorrhoeae to neutrophils.Use of eculizumab for atypical haemolytic uraemic syndrome and C3 glomerulopathies.[Management of patients with EHEC/HUS. Lessons and perspectives from clinical infectious disease specialists].Eculizumab in the treatment of atypical hemolytic uremic syndrome in an infant leads to cessation of peritoneal dialysis and improvement of severe hypertension.Inhibition of the alternative pathway of nonhuman infant complement by porin B2 contributes to virulence of Neisseria meningitidis in the infant rat modelThe Alpha-Tocopherol Form of Vitamin E Boosts Elastase Activity of Human PMNs and Their Ability to Kill Streptococcus pneumoniae.Complement factor P is a ligand for the natural killer cell-activating receptor NKp46.Current evidence on the discontinuation of eculizumab in patients with atypical haemolytic uraemic syndrome.Genomic analyses of pneumococci from children with sickle cell disease expose host-specific bacterial adaptations and deficits in current interventions.Role of macrophage sialoadhesin in host defense against the sialylated pathogen group B Streptococcus.Plasmodium falciparum malaria and invasive bacterial co-infection in young African children: the dysfunctional spleen hypothesis.Meningococcal disease and the complement system.Lower airway colonization and inflammatory response in COPD: a focus on Haemophilus influenzaeFunctional analyses of complement convertases using C3 and C5-depleted sera.Non-operative management and immune function after splenic injury.Fusion protein comprising factor H domains 6 and 7 and human IgG1 Fc as an antibacterial immunotherapeuticArcA-regulated glycosyltransferase lic2B promotes complement evasion and pathogenesis of nontypeable Haemophilus influenzae.Naturally acquired antibodies against Haemophilus influenzae type a in Aboriginal adults, CanadaDeficiency in mannose-binding lectin-associated serine protease-2 does not increase susceptibility to Trypanosoma cruzi infectionA tick mannose-binding lectin inhibitor interferes with the vertebrate complement cascade to enhance transmission of the lyme disease agent.Susceptibility to invasive meningococcal disease: polymorphism of complement system genes and Neisseria meningitidis factor H binding proteinImmunodeficiency among children with recurrent invasive pneumococcal disease.Properdin is critical for antibody-dependent bactericidal activity against Neisseria gonorrhoeae that recruit C4b-binding protein.Alternative Pathway Inhibition by Exogenous Factor H Fails to Attenuate Inflammation and Vascular Leakage in Experimental Pneumococcal Sepsis in Mice.The relative roles of factor H binding protein, neisserial surface protein A, and lipooligosaccharide sialylation in regulation of the alternative pathway of complement on meningococciThe new multicomponent vaccine against meningococcal serogroup B, 4CMenB: immunological, functional and structural characterization of the antigens.Antibody to reduction modifiable protein increases the bacterial burden and the duration of gonococcal infection in a mouse model.Small-molecule factor D inhibitors targeting the alternative complement pathway.A novel mutation in the complement component 3 gene in a patient with selective IgA deficiency.Defining the Binding Region in Factor H to Develop a Therapeutic Factor H-Fc Fusion Protein against Non-Typeable Haemophilus influenzaeHepatitis C virus suppresses C9 complement synthesis and impairs membrane attack complex function.
P2860
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P2860
Infections of People with Complement Deficiencies and Patients Who Have Undergone Splenectomy
description
2010 nî lūn-bûn
@nan
2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Infections of People with Comp ...... Who Have Undergone Splenectomy
@ast
Infections of People with Comp ...... Who Have Undergone Splenectomy
@en
type
label
Infections of People with Comp ...... Who Have Undergone Splenectomy
@ast
Infections of People with Comp ...... Who Have Undergone Splenectomy
@en
prefLabel
Infections of People with Comp ...... Who Have Undergone Splenectomy
@ast
Infections of People with Comp ...... Who Have Undergone Splenectomy
@en
P2093
P2860
P3181
P356
P1476
Infections of People with Comp ...... Who Have Undergone Splenectomy
@en
P2093
P2860
P304
P3181
P356
10.1128/CMR.00048-09
P407
P577
2010-10-01T00:00:00Z