GWAS of cerebrospinal fluid tau levels identifies risk variants for Alzheimer's disease. - Wikidata - awgldk - TriplyDB

GWAS of cerebrospinal fluid tau levels identifies risk variants for Alzheimer's disease.

GWAS of cerebrospinal fluid tau levels identifies risk variants for Alzheimer's disease.

http://www.wikidata.org/entity/Q29417137

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Meta-analysis of 74,046 individuals identifies 11 new susceptibility loci for Alzheimer's disease Rare coding variants in the phospholipase D3 gene confer risk for Alzheimer's disease Targeting innate immunity for neurodegenerative disorders of the central nervous system Genes associated with Alzheimer's disease: an overview and current status Expanding the Repertoire of Biomarkers for Alzheimer's Disease: Targeted and Non-targeted Approaches Genetic studies of quantitative MCI and AD phenotypes in ADNI: Progress, opportunities, and plans Genetic analysis of quantitative phenotypes in AD and MCI: imaging, cognition and biomarkers Genomic variants, genes, and pathways of Alzheimer's disease: An overview.Meta-analysis of the rs2075650 polymorphism and risk of Alzheimer disease.A rare mutation in UNC5C predisposes to late-onset Alzheimer's disease and increases neuronal cell death.Cognitive decline and brain pathology in aging--need for a dimensional, lifespan and systems vulnerability view.2014 Update of the Alzheimer's Disease Neuroimaging Initiative: A review of papers published since its inception.Leveraging existing data sets to generate new insights into Alzheimer's disease biology in specific patient subsets.Meta-Analysis of Transcriptome Data Related to Hippocampus Biopsies and iPSC-Derived Neuronal Cells from Alzheimer's Disease Patients Reveals an Association with FOXA1 and FOXA2 Gene Regulatory Networks.Principal component of explained variance: An efficient and optimal data dimension reduction framework for association studies.Neurodegenerative disease mutations in TREM2 reveal a functional surface and distinct loss-of-function mechanisms Genome-wide significant risk factors for Alzheimer's disease: role in progression to dementia due to Alzheimer's disease among subjects with mild cognitive impairment Genome-wide, high-content siRNA screening identifies the Alzheimer's genetic risk factor FERMT2 as a major modulator of APP metabolism.Alzheimer's disease risk genes and the age-at-onset phenotype.Multisystem component phenotypes of bipolar disorder for genetic investigations of extended pedigrees.Sex modifies the APOE-related risk of developing Alzheimer disease TRANSCRIPTION FACTOR GLI-SIMILAR 3 (GLIS3): IMPLICATIONS FOR THE DEVELOPMENT OF CONGENITAL HYPOTHYROIDISM.Genetic modification of the relationship between phosphorylated tau and neurodegeneration.Genome-wide association study of CSF levels of 59 alzheimer's disease candidate proteins: significant associations with proteins involved in amyloid processing and inflammation Alzheimer's disease risk genes and mechanisms of disease pathogenesis SUCLG2 identified as both a determinator of CSF Aβ1-42 levels and an attenuator of cognitive decline in Alzheimer's disease.Adaptors for disorders of the brain? The cancer signaling proteins NEDD9, CASS4, and PTK2B in Alzheimer's disease.Epigenomics of Alzheimer's disease.Apolipoprotein E: structure and function in lipid metabolism, neurobiology, and Alzheimer's diseases.Rare coding variants in PLCG2, ABI3, and TREM2 implicate microglial-mediated innate immunity in Alzheimer's disease.Amyloid accumulation is a late event in sporadic Alzheimer's disease-like pathology in nontransgenic rats.A genome-wide association meta-analysis of plasma Aβ peptides concentrations in the elderly.A Genetic Variant (COMT) Coding Dopaminergic Activity Predicts Personality Traits in Healthy Elderly.TREM2 Protein Expression Changes Correlate with Alzheimer's Disease Neurodegenerative Pathologies in Post-Mortem Temporal Cortices FASTKD2 is associated with memory and hippocampal structure in older adults.A pilot study to assess effects of long-term inhalation of airborne particulate matter on early Alzheimer-like changes in the mouse brain.A TREM1 variant alters the accumulation of Alzheimer-related amyloid pathology.The triggering receptor expressed on myeloid cells 2 (TREM2) is associated with enhanced inflammation, neuropathological lesions and increased risk for Alzheimer's dementia More evidence for association of a rare TREM2 mutation (R47H) with Alzheimer's disease risk.Variations in the FRA10AC1 Fragile Site and 15q21 Are Associated with Cerebrospinal Fluid Aβ1-42 Level.

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GWAS of cerebrospinal fluid tau levels identifies risk variants for Alzheimer's disease.

http://www.wikidata.org/entity/Q29417137

description

2013 nî lūn-bûn

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2013 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2013 թվականի ապրիլին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

GWAS of cerebrospinal fluid tau levels identifies risk variants for Alzheimer's disease.

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GWAS of cerebrospinal fluid tau levels identifies risk variants for Alzheimer's disease.

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type

label

GWAS of cerebrospinal fluid tau levels identifies risk variants for Alzheimer's disease.

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GWAS of cerebrospinal fluid tau levels identifies risk variants for Alzheimer's disease.

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altLabel

GWAS of Cerebrospinal Fluid Tau Levels Identifies Risk Variants for Alzheimer’s Disease

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prefLabel

GWAS of cerebrospinal fluid tau levels identifies risk variants for Alzheimer's disease.

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GWAS of cerebrospinal fluid tau levels identifies risk variants for Alzheimer's disease.

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J.NEURON.2013.02.026

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GWAS of cerebrospinal fluid tau levels identifies risk variants for Alzheimer's disease.

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Alzheimer Disease Genetic Consortium (ADGC)

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Amanda T Jeng

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Amy Gerrish

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David Carrell

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David McKean

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Douglas Galasko

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Elaine R Peskind

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GERAD Consortium

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Gerard D Schellenberg

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John S K Kauwe

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P2860

SNPs associated with cerebrospinal fluid phospho-tau levels influence rate of decline in Alzheimer's disease

TREM2 Variants in Alzheimer's Disease

Clinical diagnosis of Alzheimer's disease: report of the NINCDS-ADRDA Work Group under the auspices of Department of Health and Human Services Task Force on Alzheimer's Disease

The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics

Principal components analysis corrects for stratification in genome-wide association studies

Interleukin-1 receptor accessory protein organizes neuronal synaptogenesis as a cell adhesion molecule

Genetic evidence implicates the immune system and cholesterol metabolism in the aetiology of Alzheimer's disease

Common variants at MS4A4/MS4A6E, CD2AP, CD33 and EPHA1 are associated with late-onset Alzheimer's disease.

Genome-wide association study of CSF biomarkers Abeta1-42, t-tau, and p-tau181p in the ADNI cohort

Common variants at ABCA7, MS4A6A/MS4A4E, EPHA1, CD33 and CD2AP are associated with Alzheimer's disease

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78

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Margaret A. Pericak-Vance

Lindsay A. Farrer

Bruno A Benitez

Carlos Cruchaga

Alzheimer's Disease Neuroimaging Initiative

Celeste M Karch

David M. Holtzman

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2013-04-04T00:00:00Z

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