IL-6 and Stat3 are required for survival of intestinal epithelial cells and development of colitis-associated cancer - Wikidata - awgldk - TriplyDB

IL-6 and Stat3 are required for survival of intestinal epithelial cells and development of colitis-associated cancer

IL-6 and Stat3 are required for survival of intestinal epithelial cells and development of colitis-associated cancer

http://www.wikidata.org/entity/Q29614594

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Persistently activated Stat3 maintains constitutive NF-kappaB activity in tumors.DNA damage-mediated induction of a chemoresistant niche A human colonic commensal promotes colon tumorigenesis via activation of T helper type 17 T cell responses An epigenetic switch involving NF-kappaB, Lin28, Let-7 MicroRNA, and IL6 links inflammation to cell transformation Potential role of chitinase 3-like-1 in inflammation-associated carcinogenic changes of epithelial cells Tumor self-seeding by circulating cancer cells Inflammation in gastric cancer: Interplay of the COX-2/prostaglandin E2 and Toll-like receptor/MyD88 pathways Inflammatory bowel disease, colorectal cancer and type 2 diabetes mellitus: The links Cytokine-Induced Modulation of Colorectal Cancer Integrating Immunologic Signaling Networks: The JAK/STAT Pathway in Colitis and Colitis-Associated Cancer Helicobacter pylori infection and gastric carcinoma: Not all the strains and patients are alike Diagnostic and Prognostic Value of Serum Interleukin-6 in Colorectal Cancer The Role of the Transcriptional Regulation of Stromal Cells in Chronic Inflammation The Hen or the Egg: Inflammatory Aspects of Murine MPN Models Why does obesity promote cancer? Epidemiology, biology, and open questions Chronic inflammation and the development of malignancy in the GI tract Tyro3, Axl, and Mertk receptor signaling in inflammatory bowel disease and colitis-associated cancer Nuclear factor of activated T cells - a transcription factor family as critical regulator in lung and colon cancer Targeting the tumor microenvironment: JAK-STAT3 signaling The gastrointestinal tumor microenvironment NLRP6 in infection and inflammation Tissue communication in regenerative inflammatory signaling: lessons from the fly gut The Yin and Yang of Toll-like receptors in cancer Sphingosine-1-phosphate in chronic intestinal inflammation and cancer Potential association between TLR4 and chitinase 3-like 1 (CHI3L1/YKL-40) signaling on colonic epithelial cells in inflammatory bowel disease and colitis-associated cancer Role of TH17 cytokines in the control of colorectal cancer STAT3 and sphingosine-1-phosphate in inflammation-associated colorectal cancer Therapeutic strategies for the clinical blockade of IL-6/gp130 signaling DNA Damage and Pulmonary Hypertension Multifocal epithelial tumors and field cancerization: stroma as a primary determinant Controversies and challenges in research on urogenital schistosomiasis-associated bladder cancer Role of interleukin-6 in Barrett's esophagus pathogenesis The wound healing, chronic fibrosis, and cancer progression triad Prevention of colitis-associated cancer: natural compounds that target the IL-6 soluble receptor The therapeutic value of targeting inflammation in gastrointestinal cancers The role of suppressors of cytokine signalling in human neoplasms Targeting aberrant colon cancer-specific DNA methylation with lipoteichoic acid-deficient Lactobacillus acidophilus Entamoeba lysyl-tRNA synthetase contains a cytokine-like domain with chemokine activity towards human endothelial cells AZD1480 blocks growth and tumorigenesis of RET- activated thyroid cancer cell lines Immunity, Inflammation, and Cancer

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IL-6 and Stat3 are required for survival of intestinal epithelial cells and development of colitis-associated cancer

http://www.wikidata.org/entity/Q29614594

description

2009 nî lūn-bûn

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2009 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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2009 թվականի փետրվարին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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IL-6 and Stat3 are required fo ...... t of colitis-associated cancer

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IL-6 and Stat3 are required fo ...... t of colitis-associated cancer

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IL-6 and Stat3 are required fo ...... t of colitis-associated cancer

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IL-6 and Stat3 are required fo ...... t of colitis-associated cancer

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IL-6 and Stat3 are required fo ...... t of colitis-associated cancer

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J.CCR.2009.01.001

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IL-6 and Stat3 are required fo ...... t of colitis-associated cancer

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Eliad Karin

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Guann-Yi Yu

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Hilde Cheroutre

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Janos Terzic

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Jürgen Scheller

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Lars Eckmann

http://www.w3.org/2001/XMLSchema#string

Michael Karin

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Sergei Grivennikov

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Sivakumar Vallabhapurapu

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P2860

Inflammation and cancer

IL-6 triggers malignant features in mammospheres from human ductal breast carcinoma and normal mammary gland

Mutations in the EGFR kinase domain mediate STAT3 activation via IL-6 production in human lung adenocarcinomas

The risk of colorectal cancer in ulcerative colitis: a meta-analysis

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

The interleukin-1 family: 10 years of discovery

TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells

Recognition of commensal microflora by toll-like receptors is required for intestinal homeostasis

NF-kappaB: linking inflammation and immunity to cancer development and progression

NF-kappaB functions as a tumour promoter in inflammation-associated cancer

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103-113

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scholarly article

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580382

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10.1016/J.CCR.2009.01.001

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2

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15

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Stefan Rose-John

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2009-02-01T00:00:00Z

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23967550

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19185845

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2667107

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