Mice deficient for Rb are nonviable and show defects in neurogenesis and haematopoiesis
about
The amyloid precursor protein-binding protein APP-BP1 drives the cell cycle through the S-M checkpoint and causes apoptosis in neuronsCells degrade a novel inhibitor of differentiation with E1A-like properties upon exiting the cell cyclep38 phosphorylates Rb on Ser567 by a novel, cell cycle-independent mechanism that triggers Rb-Hdm2 interaction and apoptosisAssociation of human Pur alpha with the retinoblastoma protein, Rb, regulates binding to the single-stranded DNA Pur alpha recognition elementTargeted disruption of the ubiquitous CNC-bZIP transcription factor, Nrf-1, results in anemia and embryonic lethality in mice.E2F-6, a member of the E2F family that can behave as a transcriptional repressorA thyroid hormone receptor coactivator negatively regulated by the retinoblastoma proteinCharacterization of a novel 350-kilodalton nuclear phosphoprotein that is specifically involved in mitotic-phase progressionRNA polymerase III transcription factor IIIB is a target for repression by pocket proteins p107 and p130.Fli-1, an Ets-related transcription factor, regulates erythropoietin-induced erythroid proliferation and differentiation: evidence for direct transcriptional repression of the Rb gene during differentiation.Negative regulation of DNA replication by the retinoblastoma protein is mediated by its association with MCM7.Nab1, a corepressor of NGFI-A (Egr-1), contains an active transcriptional repression domainExplant-induced reactivation of herpes simplex virus occurs in neurons expressing nuclear cdk2 and cdk4Germ line transmission of the Cdk4(R24C) mutation facilitates tumorigenesis and escape from cellular senescence.A pentamer transcriptional complex including tal-1 and retinoblastoma protein downmodulates c-kit expression in normal erythroblastsArrest of G(1)-S progression by the p53-inducible gene PC3 is Rb dependent and relies on the inhibition of cyclin D1 transcriptionCDC25A phosphatase is a target of E2F and is required for efficient E2F-induced S phaseMDMX: a novel p53-binding protein with some functional properties of MDM2miR-17~92 cooperates with RB pathway mutations to promote retinoblastomaTumor suppressors: enhancers or suppressors of regeneration?E2F activity is regulated by cell cycle-dependent changes in subcellular localizationInteraction of D-type cyclins with a novel myb-like transcription factor, DMP1A new member of the hsp90 family of molecular chaperones interacts with the retinoblastoma protein during mitosis and after heat shockPostnatal neuronal proliferation in mice lacking Ink4d and Kip1 inhibitors of cyclin-dependent kinasesDifferential gene expression of p27Kip1 and Rb knockout pituitary tumors associated with altered growth and angiogenesisRINT-1 serves as a tumor suppressor and maintains Golgi dynamics and centrosome integrity for cell survivalThe retinoblastoma family: twins or distant cousins?Mouse models of liver cancer: Progress and recommendationsRebuilding cancer metastasis in the mouseUnderstanding pRb: toward the necessary development of targeted treatments for retinoblastomaA Mouse Model for Imprinting of the Human Retinoblastoma GeneEpigenetic regulation of histone H3 serine 10 phosphorylation status by HCF-1 proteins in C. elegans and mammalian cellsThe phosphate transporter PiT1 (Slc20a1) revealed as a new essential gene for mouse liver developmentCritical role of the Rb family in myoblast survival and fusionTranscriptional control of stem cell fate by E2Fs and pocket proteinsPhysical and functional interactions of neuronal growth suppressor necdin with p53BAD/BCL-[X(L)] heterodimerization leads to bypass of G0/G1 arrestAntioncogenes and human cancer.Synergistic role of E1A-binding proteins and tissue-specific transcription factors in differentiationNecdin, a postmitotic neuron-specific growth suppressor, interacts with viral transforming proteins and cellular transcription factor E2F1
P2860
Q22253351-CB562FE8-76CB-4CF9-BD2B-7DFD3EB3788EQ24290503-E192C36F-8EF5-4F2F-8CFD-8226A419CD57Q24301099-729E5556-880B-4D37-B12D-AC5A06DE687CQ24315375-0F72829D-9783-419B-A313-0473A5F829D6Q24317348-720E80DD-B47B-479A-B822-45B211312BBEQ24317400-A3843ED5-D9F3-4B3E-991C-787C4A30D9F5Q24322646-2BFD1070-BC1B-4C8E-A52A-ED95E2E681FCQ24336046-FDFE2BF8-B095-46ED-9EE9-3DD939955727Q24517706-93349A1D-9E0C-4196-840E-9D1EE15F84D9Q24517781-B0786B77-BF10-47B1-8BF2-D245EE3D9F5EQ24523723-5ED2E374-0850-4029-AB3C-0160008CC9A6Q24530146-AF4541DD-9CBE-488D-A0F1-9D2760B69E81Q24537780-9F6F20F8-D353-44D5-A220-2FBCBCA0D8B3Q24540051-69B6B5C6-F979-41F1-886F-3DFD6857BBD1Q24553092-14BEB17A-A856-46B6-8248-7B324934F694Q24554293-E5560B33-D710-4C19-97EB-2565E95DFCEAQ24554436-7BD197EA-A4E6-42DE-9181-129D6CA158F7Q24561971-0805088F-3FB4-4E68-A00E-5E8456421792Q24606181-B9114CA8-B419-4FC0-B951-A30E9948BBA5Q24618747-00EA601D-D5C4-43B3-B511-3B057161CCDDQ24643965-F9AB9C55-B0D4-4E02-8623-C4979E8FB07DQ24647887-0198B1C3-EF6B-47B2-967B-5EA97673818DQ24648784-54806044-3DF1-4211-A9ED-CA1C3B3FF0C1Q24649057-F1AC6A75-720F-4BC4-A48D-0102130BCC7FQ24675863-2016E150-20A4-47B7-8BBD-60D140B2BE4CQ24675915-25D7A26D-9AC4-450C-8CA1-37C9779CD98DQ24792893-6BDAB07C-3922-40CA-A04F-71D84B490006Q26799788-D82F0AEA-2443-4FDB-97D0-BA9D9C142D46Q26864450-C98947FB-CB1C-4542-A502-6DBCD9C089E0Q27011911-CCAB651C-FD64-4A6D-BD20-1B0CE330B7B3Q27301081-0C8171EF-AED2-490C-B4AC-081C93335E36Q27302346-DDF7F7A3-8586-442B-BEEC-9EEF35937D47Q27334753-315FD702-0F5F-465F-9A61-87C2A425EDEBQ27349918-04D6A491-43D0-4753-9F7A-71F699D6ABECQ28082523-B0A813D8-EB88-433A-88EF-5F37198F2F41Q28144785-37A78DBF-D7FE-4E73-A6EB-264AAED54891Q28211754-53B4AB30-A6E0-4956-862E-861E95C199F3Q28237050-7F8B9C08-504B-47CB-9678-74532AD5D67EQ28255533-3922C8E5-4492-4E98-B640-831B0F956E07Q28258359-423C17CD-F9BE-4126-8F23-66979A0E02F5
P2860
Mice deficient for Rb are nonviable and show defects in neurogenesis and haematopoiesis
description
1992 nî lūn-bûn
@nan
1992 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
1992 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
1992年の論文
@ja
1992年論文
@yue
1992年論文
@zh-hant
1992年論文
@zh-hk
1992年論文
@zh-mo
1992年論文
@zh-tw
1992年论文
@wuu
name
Mice deficient for Rb are nonviable and show defects in neurogenesis and haematopoiesis
@ast
Mice deficient for Rb are nonviable and show defects in neurogenesis and haematopoiesis
@en
type
label
Mice deficient for Rb are nonviable and show defects in neurogenesis and haematopoiesis
@ast
Mice deficient for Rb are nonviable and show defects in neurogenesis and haematopoiesis
@en
prefLabel
Mice deficient for Rb are nonviable and show defects in neurogenesis and haematopoiesis
@ast
Mice deficient for Rb are nonviable and show defects in neurogenesis and haematopoiesis
@en
P2093
P3181
P356
P1433
P1476
Mice deficient for Rb are nonviable and show defects in neurogenesis and haematopoiesis
@en
P2093
P2888
P304
P3181
P356
10.1038/359288A0
P407
P577
1992-09-24T00:00:00Z
P6179
1037982331