MicroRNA-203 suppresses cell proliferation and migration by targeting BIRC5 and LASP1 in human triple-negative breast cancer cells. - Wikidata - awgldk - TriplyDB

MicroRNA-203 suppresses cell proliferation and migration by targeting BIRC5 and LASP1 in human triple-negative breast cancer cells.

MicroRNA-203 suppresses cell proliferation and migration by targeting BIRC5 and LASP1 in human triple-negative breast cancer cells.

http://www.wikidata.org/entity/Q30010065

about

MiRNAs and Other Epigenetic Changes as Biomarkers in Triple Negative Breast Cancer Functional regulation of phospholipase D expression in cancer and inflammation MicroRNAs and triple negative breast cancer Tumor suppressor microRNAs: a novel non-coding alliance against cancer MicroRNA-29a plays a suppressive role in non-small cell lung cancer cells via targeting LASP1 An update on the LIM and SH3 domain protein 1 (LASP1): a versatile structural, signaling, and biomarker protein Loss of tumor suppressor mir-203 mediates overexpression of LIM and SH3 Protein 1 (LASP1) in high-risk prostate cancer thereby increasing cell proliferation and migration.Tumor-suppressive microRNA-218 inhibits cancer cell migration and invasion via targeting of LASP1 in prostate cancer.MicroRNA-1258: An invasion and metastasis regulator that targets heparanase in gastric cancer Upregulation of the long non-coding RNA PVT1 promotes esophageal squamous cell carcinoma progression by acting as a molecular sponge of miR-203 and LASP1 miR-203 regulates progenitor cell proliferation during adult zebrafish retina regeneration.Regulation of the mRNA half-life in breast cancer Posttranscriptional deregulation of Src due to aberrant miR34a and miR203 contributes to gastric cancer development The inhibitory role of Mir-29 in growth of breast cancer cells microRNA-mediated survivin control of pluripotency.Association of survivin splice variants with prognosis and treatment of breast cancer.Combination therapy with local radiofrequency ablation and systemic vaccine enhances antitumor immunity and mediates local and distal tumor regression.miR-203 inhibits cell proliferation and migration of lung cancer cells by targeting PKCα miR-203 suppresses the proliferation and migration and promotes the apoptosis of lung cancer cells by targeting SRC.ZEB1-associated drug resistance in cancer cells is reversed by the class I HDAC inhibitor mocetinostat.Expression of microRNAs and isomiRs in the porcine endometrium: implications for gene regulation at the maternal-conceptus interface Low expression of microRNA-204 (miR-204) is associated with poor clinical outcome of acute myeloid leukemia (AML) patients.MicroRNA networks regulated by all-trans retinoic acid and Lapatinib control the growth, survival and motility of breast cancer cells Regulation of Cell Proliferation and Migration by miR-203 via GAS41/miR-10b Axis in Human Glioblastoma Cells.miR-429 mediates δ-tocotrienol-induced apoptosis in triple-negative breast cancer cells by targeting XIAP.Cantharidin modulates the E2F1/MCM7-miR-106b-93/p21-PTEN signaling axis in MCF-7 breast cancer cells.Expression of miR-203 is decreased and associated with the prognosis of melanoma patients.The role of microRNAs in breast cancer migration, invasion and metastasis TLR3 agonist and Sorafenib combinatorial therapy promotes immune activation and controls hepatocellular carcinoma progression miR-203 Functions as a Tumor Suppressor by Inhibiting Epithelial to Mesenchymal Transition in Ovarian Cancer Signaling between transforming growth factor β (TGF-β) and transcription factor SNAI2 represses expression of microRNA miR-203 to promote epithelial-mesenchymal transition and tumor metastasis Leukemia inhibitory factor promotes EMT through STAT3-dependent miR-21 induction Long-term exposure of MCF-12A normal human breast epithelial cells to ethanol induces epithelial mesenchymal transition and oncogenic features.The landscape of chromosomal aberrations in breast cancer mouse models reveals driver-specific routes to tumorigenesis.MicroRNA profiling of Sendai virus-infected A549 cells identifies miR-203 as an interferon-inducible regulator of IFIT1/ISG56.Epigenetic silencing of microRNA-203 is required for EMT and cancer stem cell properties.MicroRNA 203 Modulates Glioma Cell Migration via Robo1/ERK/MMP-9 Signaling.Can the microRNA expression profile help to identify novel targets for zoledronic acid in breast cancer?miR-203 facilitates tumor growth and metastasis by targeting fibroblast growth factor 2 in breast cancer miR-203 enhances let-7 biogenesis by targeting LIN28B to suppress tumor growth in lung cancer.

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P2860

MicroRNA-203 suppresses cell proliferation and migration by targeting BIRC5 and LASP1 in human triple-negative breast cancer cells.

http://www.wikidata.org/entity/Q30010065

description

2012 nî lūn-bûn

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2012 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2012 թվականի հունիսին հրատարակված գիտական հոդված

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2012年の論文

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MicroRNA-203 suppresses cell p ...... -negative breast cancer cells.

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MicroRNA-203 suppresses cell p ...... -negative breast cancer cells.

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type

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MicroRNA-203 suppresses cell p ...... -negative breast cancer cells.

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MicroRNA-203 suppresses cell p ...... -negative breast cancer cells.

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MicroRNA-203 suppresses cell p ...... -negative breast cancer cells.

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P2093

Chen Wang

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Chunyan Shen

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Xiangqian Zheng

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Yurong Shi

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P2860

Global cancer statistics

IAPs: more than just inhibitors of apoptosis proteins

A novel anti-apoptosis gene, survivin, expressed in cancer and lymphoma

Human microRNA genes are frequently located at fragile sites and genomic regions involved in cancers

MicroRNA expression profiles associated with prognosis and therapeutic outcome in colon adenocarcinoma

MicroRNA expression profiles classify human cancers

Inhibition of translational initiation by Let-7 MicroRNA in human cells

Genetic and epigenetic silencing of microRNA-203 enhances ABL1 and BCR-ABL1 oncogene expression

Anti-tumor activity of a novel compound-CDF is mediated by regulating miR-21, miR-200, and PTEN in pancreatic cancer

Micromanagers of gene expression: the potentially widespread influence of metazoan microRNAs

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