Functional and physical interactions between formyl-peptide-receptors and scavenger receptor MARCO and their involvement in amyloid beta 1-42-induced signal transduction in glial cells.
about
Formyl peptide receptors from immune and vomeronasal system exhibit distinct agonist propertiesMechanisms of Aβ Clearance and Degradation by Glial CellsCalcium-Sensing Receptors of Human Neural Cells Play Crucial Roles in Alzheimer's DiseaseAmyloid-β Receptors: The Good, the Bad, and the Prion ProteinInnate immunity in Alzheimer's disease: a complex affairInvolvement of formyl peptide receptors in receptor for advanced glycation end products (RAGE)--and amyloid beta 1-42-induced signal transduction in glial cellsDeficiency of formyl peptide receptor 1 and 2 is associated with increased inflammation and enhanced liver injury after LPS-stimulation.The formyl peptide receptor like-1 and scavenger receptor MARCO are involved in glial cell activation in bacterial meningitis.Distinct signaling cascades elicited by different formyl peptide receptor 2 (FPR2) agonistsAnnexin A1: a central player in the anti-inflammatory and neuroprotective role of microglia.Active immunization against complement factor C5a: a new therapeutic approach for Alzheimer's disease3-(1H-indol-3-yl)-2-[3-(4-nitrophenyl)ureido]propanamide enantiomers with human formyl-peptide receptor agonist activity: molecular modeling of chiral recognition by FPR2Identifying N-linked glycan moiety and motifs in the cysteine-rich domain critical for N-glycosylation and intracellular trafficking of SR-AI and MARCO.Fucoidan is a novel platelet agonist for the C-type lectin-like receptor 2 (CLEC-2)Expression and function of psoriasin (S100A7) and koebnerisin (S100A15) in the brain.Microglial scavenger receptors and their roles in the pathogenesis of Alzheimer's disease.Microglia, neuroinflammation, and beta-amyloid protein in Alzheimer's disease.Microglial Aβ receptors in Alzheimer's disease.The anti-inflammatory Annexin A1 induces the clearance and degradation of the amyloid-β peptide.Proinflammatory stimulants promote the expression of a promiscuous G protein-coupled receptor, mFPR2, in microvascular endothelial cells.Microglial activation depends on beta-amyloid conformation: role of the formylpeptide receptor 2.FPR2/ALX receptor expression and internalization are critical for lipoxin A4 and annexin-derived peptide-stimulated phagocytosis.Lack of formyl peptide receptor 1 and 2 leads to more severe inflammation and higher mortality in mice with of pneumococcal meningitis.Age Influences Microglial Activation After Cuprizone-Induced Demyelination
P2860
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P248
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P2860
Functional and physical interactions between formyl-peptide-receptors and scavenger receptor MARCO and their involvement in amyloid beta 1-42-induced signal transduction in glial cells.
description
2010 nî lūn-bûn
@nan
2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Functional and physical intera ...... l transduction in glial cells.
@ast
Functional and physical intera ...... l transduction in glial cells.
@en
type
label
Functional and physical intera ...... l transduction in glial cells.
@ast
Functional and physical intera ...... l transduction in glial cells.
@en
prefLabel
Functional and physical intera ...... l transduction in glial cells.
@ast
Functional and physical intera ...... l transduction in glial cells.
@en
P2093
P921
P1476
Functional and physical intera ...... l transduction in glial cells.
@en
P2093
Christoph J Wruck
Lars-Ove Brandenburg
Maximilian Konrad
Ralph Lucius
Thomas Koch
Thomas Pufe
P304
P356
10.1111/J.1471-4159.2010.06637.X
P407
P577
2010-02-05T00:00:00Z