Positive- and negative-acting Kruppel-like transcription factors bind a transforming growth factor beta control element required for expression of the smooth muscle cell differentiation marker SM22alpha in vivo.
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Proteasomal degradation of the KLF5 transcription factor through a ubiquitin-independent pathwayKLF5 Interacts with p53 in regulating survivin expression in acute lymphoblastic leukemiaSynergistic activation of the rat laminin gamma1 chain promoter by the gut-enriched Kruppel-like factor (GKLF/KLF4) and Sp1Opposing effects of Krüppel-like factor 4 (gut-enriched Krüppel-like factor) and Krüppel-like factor 5 (intestinal-enriched Krüppel-like factor) on the promoter of the Krüppel-like factor 4 gene.Mammalian Krüppel-like factors in health and diseasesOverexpression of Krüppel-like factor 4 in the human colon cancer cell line RKO leads to reduced tumorigenecityMicroRNA-145, a novel smooth muscle cell phenotypic marker and modulator, controls vascular neointimal lesion formationBiology of fibroblasts and myofibroblastsIntestinal-enriched Krüppel-like factor (Krüppel-like factor 5) is a positive regulator of cellular proliferationPhosphorylation of Kruppel-like factor 5 (KLF5/IKLF) at the CBP interaction region enhances its transactivation functionSp1- and Krüppel-like transcription factorsEndothelial Plasticity: Shifting Phenotypes through Force FeedbackThe Role of Krüppel-like Factor 4 in Renal FibrosisThe double life of KLF5: Opposing roles in regulation of gene-expression, cellular function, and transformationKruppel-like factor 4 (KLF4) represses histidine decarboxylase gene expression through an upstream Sp1 site and downstream gastrin responsive elementsHuman Kruppel-like factor 5 is a target of the E3 ubiquitin ligase WWP1 for proteolysis in epithelial cellsUniaxial mechanical strain modulates the differentiation of neural crest stem cells into smooth muscle lineage on micropatterned surfacesThe SWI/SNF chromatin remodeling complex regulates myocardin-induced smooth muscle-specific gene expressionLentivirus Live Cell Array for Quantitative Assessment of Gene and Pathway Activation during Myogenic Differentiation of Mesenchymal Stem CellsPIAS1 activates the expression of smooth muscle cell differentiation marker genes by interacting with serum response factor and class I basic helix-loop-helix proteins.Kruppel-like factor 4, Elk-1, and histone deacetylases cooperatively suppress smooth muscle cell differentiation markers in response to oxidized phospholipids.Gut-enriched Krüppel-like factor interaction with Smad3 inhibits myofibroblast differentiationWhole animal knockout of smooth muscle alpha-actin does not alter excisional wound healing or the fibroblast-to-myofibroblast transitionCooperative binding of KLF4, pELK-1, and HDAC2 to a G/C repressor element in the SM22α promoter mediates transcriptional silencing during SMC phenotypic switching in vivo.PIAS1 mediates TGFbeta-induced SM alpha-actin gene expression through inhibition of KLF4 function-expression by protein sumoylationSmooth and cardiac muscle-selective knock-out of Kruppel-like factor 4 causes postnatal death and growth retardation.Platelet-derived growth factor-BB and Ets-1 transcription factor negatively regulate transcription of multiple smooth muscle cell differentiation marker genes.5' CArG degeneracy in smooth muscle alpha-actin is required for injury-induced gene suppression in vivo.Sp1-dependent activation of KLF4 is required for PDGF-BB-induced phenotypic modulation of smooth muscle.Oxidized phospholipids induce type VIII collagen expression and vascular smooth muscle cell migration.Control of SRF binding to CArG box chromatin regulates smooth muscle gene expression in vivo.Conditional deletion of Krüppel-like factor 4 delays downregulation of smooth muscle cell differentiation markers but accelerates neointimal formation following vascular injuryMultiple repressor pathways contribute to phenotypic switching of vascular smooth muscle cells.Prostate cancer invasion and metastasis: insights from mining genomic data.The zinc finger transcription factor transforming growth factor beta-inducible early gene-1 confers myeloid-specific activation of the leukocyte integrin CD11d promoter.Association of the actin-binding protein transgelin with lymph node metastasis in human colorectal cancerKlf5 Mediates Odontoblastic Differentiation through Regulating Dentin-Specific Extracellular Matrix Gene Expression during Mouse Tooth DevelopmentMicroRNA expression in human airway smooth muscle cells: role of miR-25 in regulation of airway smooth muscle phenotypeSm22α transcription occurs at the early onset of the cardiovascular system and the intron 1 is dispensable for its transcription in smooth muscle cells during mouse development.Sp1 and krüppel-like factor family of transcription factors in cell growth regulation and cancer.
P2860
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P2860
Positive- and negative-acting Kruppel-like transcription factors bind a transforming growth factor beta control element required for expression of the smooth muscle cell differentiation marker SM22alpha in vivo.
description
2000 nî lūn-bûn
@nan
2000 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
Positive- and negative-acting ...... tion marker SM22alpha in vivo.
@ast
Positive- and negative-acting ...... tion marker SM22alpha in vivo.
@en
type
label
Positive- and negative-acting ...... tion marker SM22alpha in vivo.
@ast
Positive- and negative-acting ...... tion marker SM22alpha in vivo.
@en
prefLabel
Positive- and negative-acting ...... tion marker SM22alpha in vivo.
@ast
Positive- and negative-acting ...... tion marker SM22alpha in vivo.
@en
P2093
P2860
P356
P1476
Positive- and negative-acting ...... ation marker SM22alpha in vivo
@en
P2093
P2860
P304
37798-37806
P356
10.1074/JBC.M006323200
P407
P577
2000-12-01T00:00:00Z