Breast cancer metastasis suppressor 1 (BRMS1) suppresses metastasis and correlates with improved patient survival in non-small cell lung cancer - Wikidata - awgldk - TriplyDB

Breast cancer metastasis suppressor 1 (BRMS1) suppresses metastasis and correlates with improved patient survival in non-small cell lung cancer

Breast cancer metastasis suppressor 1 (BRMS1) suppresses metastasis and correlates with improved patient survival in non-small cell lung cancer

http://www.wikidata.org/entity/Q30360186

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Metastasis suppressor genes Metastasis suppressor genes at the interface between the environment and tumor cell growth CK2α' Drives Lung Cancer Metastasis by Targeting BRMS1 Nuclear Export and Degradation.Loss of BRMS1 promotes a mesenchymal phenotype through NF-κB-dependent regulation of Twist1.Modelling and simulation of mutant alleles of breast cancer metastasis suppressor 1 (BRMS1) gene Combined proteasome and histone deacetylase inhibition attenuates epithelial-mesenchymal transition through E-cadherin in esophageal cancer cells Inhibition of breast cancer metastasis suppressor 1 promotes a mesenchymal phenotype in lung epithelial cells that express oncogenic K-RasV12 and loss of p53.BRMS1 suppresses lung cancer metastases through an E3 ligase function on histone acetyltransferase p300.Phosphorylation of RelA/p65 promotes DNMT-1 recruitment to chromatin and represses transcription of the tumor metastasis suppressor gene BRMS1.BRMS1 transcriptional repression correlates with CpG island methylation and advanced pathological stage in non-small cell lung cancer.A shift from nuclear to cytoplasmic breast cancer metastasis suppressor 1 expression is associated with highly proliferative estrogen receptor-negative breast cancers.BRMS1 suppresses glioma progression by regulating invasion, migration and adhesion of glioma cells.Protein Signatures in Human MDA-MB-231 Breast Cancer Cells Indicating a More Invasive Phenotype Following Knockdown of Human Endometase/Matrilysin-2 by siRNA.Cytoplasmic BRMS1 expression in malignant melanoma is associated with increased disease-free survival.Genomics screens for metastasis genes Low BRMS1 expression promotes nasopharyngeal carcinoma metastasis in vitro and in vivo and is associated with poor patient survival Homotypic gap junctional communication associated with metastasis suppression increases with PKA activity and is unaffected by PI3K inhibition Metastasis suppressors and the tumor microenvironment Unraveling the enigmatic complexities of BRMS1-mediated metastasis suppression Breast cancer metastasis suppressor 1 (BRMS1) attenuates TGF-β1-induced breast cancer cell aggressiveness through downregulating HIF-1α expression.Down-regulation of BRMS1 by DNA hypermethylation and its association with metastatic progression in triple-negative breast cancer Communication between host organism and cancer cells is transduced by systemic sphingosine kinase 1/sphingosine 1-phosphate signalling to regulate tumour metastasis.Loss of breast cancer metastasis suppressor 1 promotes ovarian cancer cell metastasis by increasing chemokine receptor 4 expression.Cyclin-dependent kinase-mediated phosphorylation of breast cancer metastasis suppressor 1 (BRMS1) affects cell migration.Ubiquitous Brms1 expression is critical for mammary carcinoma metastasis suppression via promotion of apoptosis.Breast cancer metastasis suppressor 1 modulates SIRT1-dependent p53 deacetylation through interacting with DBC1.Escin Ia suppresses the metastasis of triple-negative breast cancer by inhibiting epithelial-mesenchymal transition via down-regulating LOXL2 expression Breast cancer metastasis suppressor 1 coordinately regulates metastasis-associated microRNA expression Linking metastasis suppression with metastamiR regulation.Analysis of chromosomal aberration (1, 3, and 8) and association of microRNAs in uveal melanoma.Breast cancer metastasis suppressor-1 promoter methylation in cell-free DNA provides prognostic information in non-small cell lung cancer.Metastasis suppression by BRMS1 associated with SIN3 chromatin remodeling complexes.The role of TWIST1 in epithelial-mesenchymal transition and cancers.BRMS1 regulates apoptosis in non-small cell lung cancer cells.Effect of BRMS1 on tumorigenicity and metastasis of human rectal cancer.Breast cancer metastasis suppressor 1 regulates hepatocellular carcinoma cell apoptosis via suppressing osteopontin expression BRMS1 gene expression may be associated with clinico-pathological features of breast cancer.Gene promoter methylation and protein expression of BRMS1 in uterine cervix in relation to high-risk human papilloma virus infection and cancer.Cancer-specific promoters for expression-targeted gene therapy: ran, brms1 and mcm5.MicroRNA-423 enhances the invasiveness of hepatocellular carcinoma via regulation of BRMS1.

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Breast cancer metastasis suppressor 1 (BRMS1) suppresses metastasis and correlates with improved patient survival in non-small cell lung cancer

http://www.wikidata.org/entity/Q30360186

description

2008 nî lūn-bûn

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2008 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2008 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年論文

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2008年论文

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name

Breast cancer metastasis suppr ...... in non-small cell lung cancer

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Breast cancer metastasis suppr ...... in non-small cell lung cancer

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Breast cancer metastasis suppr ...... in non-small cell lung cancer

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Breast cancer metastasis suppr ...... in non-small cell lung cancer

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Breast cancer metastasis suppr ...... in non-small cell lung cancer

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Breast cancer metastasis suppr ...... in non-small cell lung cancer

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J.CANLET.2008.11.024

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Breast cancer metastasis suppr ...... in non-small cell lung cancer

@en

P2093

David R Jones

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Gina R Petroni

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Philip W Smith

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Suzanne A Siefert

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Yuan Liu

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P2860

Modulation of NF-kappaB-dependent transcription and cell survival by the SIRT1 deacetylase.

Breast cancer metastasis suppressor 1 functions as a corepressor by enhancing histone deacetylase 1-mediated deacetylation of RelA/p65 and promoting apoptosis.

Cancer statistics, 2008

Oesophageal squamous cell neoplasia in head and neck cancer patients: upregulation of COX-2 during carcinogenesis

Revisions in the International System for Staging Lung Cancer

Inhibition of NF-kappaB sensitizes non-small cell lung cancer cells to chemotherapy-induced apoptosis.

The relationship of BRMS1 and RhoGDI2 gene expression to metastatic potential in lineage related human bladder cancer cell lines.

Inhibition of phosphatidylinositol 3-kinase/Akt and histone deacetylase activity induces apoptosis in non-small cell lung cancer in vitro and in vivo.

Proteomics-based strategy to delineate the molecular mechanisms of the metastasis suppressor gene BRMS1.

Functional evidence for a novel human breast carcinoma metastasis suppressor, BRMS1, encoded at chromosome 11q13.

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196-203

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2

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276

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Christopher Moskaluk

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19111386

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4793277

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