Synergistic stimulation of type I interferons during influenza virus coinfection promotes Streptococcus pneumoniae colonization in mice. - Wikidata - awgldk - TriplyDB

Synergistic stimulation of type I interferons during influenza virus coinfection promotes Streptococcus pneumoniae colonization in mice.

Synergistic stimulation of type I interferons during influenza virus coinfection promotes Streptococcus pneumoniae colonization in mice.

http://www.wikidata.org/entity/Q30405962

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Harnessing Mechanistic Knowledge on Beneficial Versus Deleterious IFN-I Effects to Design Innovative Immunotherapies Targeting Cytokine Activity to Specific Cell Types Type I interferon protects against pneumococcal invasive disease by inhibiting bacterial transmigration across the lung Mechanisms of Bacterial Colonization of the Respiratory Tract Influenza and Bacterial Superinfection: Illuminating the Immunologic Mechanisms of Disease Viral-bacterial interactions-therapeutic implications Differential effects of type I and II interferons on myeloid cells and resistance to intracellular bacterial infections Co-infection subverts mucosal immunity in the upper respiratory tract Digging through the Obstruction: Insight into the Epithelial Cell Response to Respiratory Virus Infection in Patients with Cystic Fibrosis Disease-promoting effects of type I interferons in viral, bacterial, and coinfections The immunology of influenza virus-associated bacterial pneumonia Postviral Complications: Bacterial Pneumonia Limited Efficacy of Antibacterial Vaccination Against Secondary Serotype 3 Pneumococcal Pneumonia Following Influenza Infection.Time and dose-dependent risk of pneumococcal pneumonia following influenza: a model for within-host interaction between influenza and Streptococcus pneumoniae Influenza vaccines to control influenza-associated bacterial infection: where do we stand?Influenza-associated bacterial pneumonia; managing and controlling infection on two fronts.Reconstruction of the 1918 influenza virus: unexpected rewards from the past Kinetics of coinfection with influenza A virus and Streptococcus pneumoniae.Identifying the interaction between influenza and pneumococcal pneumonia using incidence data.Type I interferon signaling regulates activation of the absent in melanoma 2 inflammasome during Streptococcus pneumoniae infection.Characterization of inflammatory responses during intranasal colonization with Streptococcus pneumoniae.Influenza promotes pneumococcal growth during coinfection by providing host sialylated substrates as a nutrient source.Pneumolysin expression by streptococcus pneumoniae protects colonized mice from influenza virus-induced disease Type I interferons in bacterial infections: taming of myeloid cells and possible implications for autoimmunity.Coinfection with Streptococcus pneumoniae modulates the B cell response to influenza virus Development of primary invasive pneumococcal disease caused by serotype 1 pneumococci is driven by early increased type I interferon response in the lung Current concepts in host-microbe interaction leading to pneumococcal pneumonia Coinfection with Streptococcus pneumoniae negatively modulates the size and composition of the ongoing influenza-specific CD8⁺ T cell response Reprogramming of murine macrophages through TLR2 confers viral resistance via TRAF3-mediated, enhanced interferon production.The methyltransferase Setdb2 mediates virus-induced susceptibility to bacterial superinfection.Lethal coinfection of influenza virus and Streptococcus pneumoniae lowers antibody response to influenza virus in lung and reduces numbers of germinal center B cells, T follicular helper cells, and plasma cells in mediastinal lymph Node.Expression of suppressor of cytokine signaling 1 (SOCS1) impairs viral clearance and exacerbates lung injury during influenza infection.The administration of intranasal live attenuated influenza vaccine induces changes in the nasal microbiota and nasal epithelium gene expression profiles Type I Interferon Induced Epigenetic Regulation of Macrophages Suppresses Innate and Adaptive Immunity in Acute Respiratory Viral Infection Viral Inhibition of Bacterial Phagocytosis by Human Macrophages: Redundant Role of CD36.Pulmonary immunostimulation with MALP-2 in influenza virus-infected mice increases survival after pneumococcal superinfection Impact of Type I and III Interferons on Respiratory Superinfections Due to Multidrug-Resistant Pathogens.Type I interferon induction during influenza virus infection increases susceptibility to secondary Streptococcus pneumoniae infection by negative regulation of γδ T cells Intranasal vaccination with replication-defective adenovirus type 5 encoding influenza virus hemagglutinin elicits protective immunity to homologous challenge and partial protection to heterologous challenge in pigs.Immune modulation by group B Streptococcus influences host susceptibility to urinary tract infection by uropathogenic Escherichia coli.Modulation of nasopharyngeal innate defenses by viral coinfection predisposes individuals to experimental pneumococcal carriage

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P2860

Synergistic stimulation of type I interferons during influenza virus coinfection promotes Streptococcus pneumoniae colonization in mice.

http://www.wikidata.org/entity/Q30405962

description

2011 nî lūn-bûn

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2011 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի օգոստոսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Synergistic stimulation of typ ...... eumoniae colonization in mice.

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Synergistic stimulation of typ ...... eumoniae colonization in mice.

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Synergistic stimulation of typ ...... eumoniae colonization in mice.

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Synergistic stimulation of typ ...... eumoniae colonization in mice.

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Synergistic stimulation of typ ...... eumoniae colonization in mice.

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Kimberly M Davis

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Shigeki Nakamura

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P2860

Functional role of type I and type II interferons in antiviral defense

Predominant role of bacterial pneumonia as a cause of death in pandemic influenza: implications for pandemic influenza preparedness

Virus interference. I. The interferon

Insights into the interaction between influenza virus and pneumococcus

Growth of H5N1 influenza A viruses in the upper respiratory tracts of mice.

Bacterial ligands generated in a phagosome are targets of the cytosolic innate immune system.

NOD2, RIP2 and IRF5 play a critical role in the type I interferon response to Mycobacterium tuberculosis.

Streptococcus pneumoniae nasopharyngeal colonization induces type I interferons and interferon-induced gene expression.

Intranasal Poly-IC treatment exacerbates tuberculosis in mice through the pulmonary recruitment of a pathogen-permissive monocyte/macrophage population.

Recognition of pneumolysin by Toll-like receptor 4 confers resistance to pneumococcal infection.

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3657-3665

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10.1172/JCI57762

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9

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Jeffrey N Weiser

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2011-08-15T00:00:00Z

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