Breast cancer molecular signatures as determined by SAGE: correlation with lymph node status. - Wikidata - awgldk - TriplyDB

Breast cancer molecular signatures as determined by SAGE: correlation with lymph node status.

Breast cancer molecular signatures as determined by SAGE: correlation with lymph node status.

http://www.wikidata.org/entity/Q30410300

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Large-scale data integration framework provides a comprehensive view on glioblastoma multiforme Dissecting the Potential Interplay of DEK Functions in Inflammation and Cancer Epigenetic changes induced by air toxics: formaldehyde exposure alters miRNA expression profiles in human lung cells Unraveling 50-Year-Old Clues Linking Neurodegeneration and Cancer to Cycad Toxins: Are microRNAs Common Mediators?The DEK oncogene is a target of steroid hormone receptor signaling in breast cancer A gene expression classifier of node-positive colorectal cancer Predicting sulfotyrosine sites using the random forest algorithm with significantly improved prediction accuracy.Robust estimation of the expected survival probabilities from high-dimensional Cox models with biomarker-by-treatment interactions in randomized clinical trials.Genomic alterations of primary tumor and blood in invasive ductal carcinoma of breast Control of tumorigenesis and chemoresistance by the DEK oncogene Differential gene expression in primary breast tumors associated with lymph node metastasis.Serological analysis of SEREX-defined medullary breast carcinoma-associated antigens.Exploring molecular links between lymph node invasion and cancer prognosis in human breast cancer.Mammary differentiation induces expression of Tristetraprolin, a tumor suppressor AU-rich mRNA-binding protein.DEK over expression as an independent biomarker for poor prognosis in colorectal cancer.Identification by array comparative genomic hybridization of a new amplicon on chromosome 17q highly recurrent in BRCA1 mutated triple negative breast cancer.Aberrant expression of maternal Plk1 and Dctn3 results in the developmental failure of human in-vivo- and in-vitro-matured oocytes.The human DEK oncogene stimulates β-catenin signaling, invasion and mammosphere formation in breast cancer.Molecular signatures of lymph node status by intrinsic subtype: gene expression analysis of primary breast tumors from patients with and without metastatic lymph nodes.Quantitative analysis of energy metabolic pathways in MCF-7 breast cancer cells by selected reaction monitoring assay.Future directions and treatment strategies for head and neck squamous cell carcinomas Overexpression of DEK is an indicator of poor prognosis in patients with gastric adenocarcinoma.DEK proto-oncogene expression interferes with the normal epithelial differentiation program.Overexpression of the cellular DEK protein promotes epithelial transformation in vitro and in vivo Overexpressed vs mutated Kras in murine fibroblasts: a molecular phenotyping study.Identification of gene expression signature in estrogen receptor positive breast carcinoma.Candidate SNP Markers of Chronopathologies Are Predicted by a Significant Change in the Affinity of TATA-Binding Protein for Human Gene Promoters.The role of DEK protein in hepatocellular carcinoma for progression and prognosis.The DEK oncogene activates VEGF expression and promotes tumor angiogenesis and growth in HIF-1α-dependent and -independent manners.Lung cancer gene expression database analysis incorporating prior knowledge with support vector machine-based classification method.TIS11 family proteins and their roles in posttranscriptional gene regulation.Gene signature distinguishes patients with chronic ulcerative colitis harboring remote neoplastic lesions.Rhomboid domain containing 2 (RHBDD2): a novel cancer-related gene over-expressed in breast cancer.Ductal carcinoma in situ of the breast: morphological and molecular features implicated in progression.Analysis of molecular markers as predictive factors of lymph node involvement in breast carcinoma.Identification of signaling pathways modulated by RHBDD2 in breast cancer cells: a link to the unfolded protein response.Integrating breast cancer genetics into clinical practice.The DEK oncoprotein and its emerging roles in gene regulation.RNA binding proteins as regulators of immune cell biology.Ror2 Signaling and Its Relevance in Breast Cancer Progression.

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Breast cancer molecular signatures as determined by SAGE: correlation with lymph node status.

http://www.wikidata.org/entity/Q30410300

description

2007 nî lūn-bûn

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2007 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2007 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

Breast cancer molecular signat ...... lation with lymph node status.

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Breast cancer molecular signat ...... lation with lymph node status.

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Breast cancer molecular signat ...... lation with lymph node status.

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Breast cancer molecular signat ...... lation with lymph node status.

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Breast cancer molecular signat ...... lation with lymph node status.

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Molecular Cancer Research

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Breast cancer molecular signat ...... elation with lymph node status

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Aysegul Sahin

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Hongxia Sun

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Jeffrey A Drake

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Maria I Nunez

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Steve Horvath

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Tao Shi

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Yuhui Hu

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P2860

Characterization of the p22 subunit of dynactin reveals the localization of cytoplasmic dynein and dynactin to the midbody of dividing cells

D53 (TPD52L1) is a cell cycle-regulated protein maximally expressed at the G2-M transition in breast cancer cells

Identifying biological themes within lists of genes with EASE

Early mitotic degradation of the homeoprotein HOXC10 is potentially linked to cell cycle progression

DAVID: Database for Annotation, Visualization, and Integrated Discovery

Gene expression patterns of breast carcinomas distinguish tumor subclasses with clinical implications

Gene expression profiling predicts clinical outcome of breast cancer

A gene-expression signature as a predictor of survival in breast cancer

Molecular portraits of human breast tumours

Repeated observation of breast tumor subtypes in independent gene expression data sets

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881-890

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10.1158/1541-7786.MCR-07-0055

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Martín C. Abba

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