Autophagy is required for exercise training-induced skeletal muscle adaptation and improvement of physical performance - Wikidata - awgldk - TriplyDB

Autophagy is required for exercise training-induced skeletal muscle adaptation and improvement of physical performance

Autophagy is required for exercise training-induced skeletal muscle adaptation and improvement of physical performance

http://www.wikidata.org/entity/Q30410425

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Cachexia in chronic obstructive pulmonary disease: new insights and therapeutic perspective The beneficial role of proteolysis in skeletal muscle growth and stress adaptation Mitochondrial Quality Control and Muscle Mass Maintenance You're only as old as your arteries: translational strategies for preserving vascular endothelial function with aging Autophagy is essential to support skeletal muscle plasticity in response to endurance exercise Autophagy-associated atrophy and metabolic remodeling of the mouse diaphragm after short-term intermittent hypoxia Disrupted autophagy undermines skeletal muscle adaptation and integrity Mitochondria Initiate and Regulate Sarcopenia Ulk1-mediated autophagy plays an essential role in mitochondrial remodeling and functional regeneration of skeletal muscle.Molecular mechanisms for mitochondrial adaptation to exercise training in skeletal muscle.Exercise prevents maternal high-fat diet-induced hypermethylation of the Pgc-1α gene and age-dependent metabolic dysfunction in the offspring.Effect of electrical stimulation-induced resistance exercise on mitochondrial fission and fusion proteins in rat skeletal muscle.Activating Autophagy by Aerobic Exercise in Mice.The Nuclear Receptor, Nor-1, Induces the Physiological Responses Associated With Exercise.Protein kinase C-alpha suppresses autophagy and induces neural tube defects via miR-129-2 in diabetic pregnancy.Electrical stimulation counteracts muscle decline in seniors.Heat shock response and autophagy--cooperation and control.Can endurance exercise preconditioning prevention disuse muscle atrophy?Role of PGC-1α during acute exercise-induced autophagy and mitophagy in skeletal muscle Autophagic signaling and proteolytic enzyme activity in cardiac and skeletal muscle of spontaneously hypertensive rats following chronic aerobic exercise Autophagy is not required to sustain exercise and PRKAA1/AMPK activity but is important to prevent mitochondrial damage during physical activity.Induction and adaptation of chaperone-assisted selective autophagy CASA in response to resistance exercise in human skeletal muscle.A novel mechanism of autophagic cell death in dystrophic muscle regulated by P2RX7 receptor large-pore formation and HSP90.Mitochondrial quality control, promoted by PGC-1α, is dysregulated by Western diet-induced obesity and partially restored by moderate physical activity in mice.Endurance exercise and selective breeding for longevity extend Drosophila healthspan by overlapping mechanisms.Denervation-Induced Activation of the Standard Proteasome and Immunoproteasome Effects of long-term resistance exercise training on autophagy in rat skeletal muscle of chloroquine-induced sporadic inclusion body myositis.Skeletal muscle metabolic adaptations to endurance exercise training are attainable in mice with simvastatin treatment PGC-1α promotes exercise-induced autophagy in mouse skeletal muscle.The regulation of autophagy during exercise in skeletal muscle The emerging role of skeletal muscle oxidative metabolism as a biological target and cellular regulator of cancer-induced muscle wasting Effects of aerobic training on markers of autophagy in the elderly.Voluntary resistance wheel exercise from mid-life prevents sarcopenia and increases markers of mitochondrial function and autophagy in muscles of old male and female C57BL/6J mice.Autophagy and mitophagy in the myocardium: therapeutic potential and concerns.Skeletal muscle, autophagy, and physical activity: the ménage à trois of metabolic regulation in health and disease.Autophagic cellular responses to physical exercise in skeletal muscle.The many roles of PGC-1α in muscle--recent developments Autophagy--a key player in cellular and body metabolism.Mitochondria, muscle health, and exercise with advancing age.Autophagy as a Potential Target for Sarcopenia.

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Autophagy is required for exercise training-induced skeletal muscle adaptation and improvement of physical performance

http://www.wikidata.org/entity/Q30410425

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2013 nî lūn-bûn

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2013 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2013 թվականի հունիսին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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Autophagy is required for exer ...... vement of physical performance

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Autophagy is required for exer ...... vement of physical performance

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Autophagy is required for exer ...... vement of physical performance

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P2093

David S Breen

http://www.w3.org/2001/XMLSchema#string

Mei Zhang

http://www.w3.org/2001/XMLSchema#string

Mitsuharu Okutsu

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Nicholas P Greene

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Rhianna C Laker

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Vitor A Lira

http://www.w3.org/2001/XMLSchema#string

Zhen Yan

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P2860

LC3, a mammalian homologue of yeast Apg8p, is localized in autophagosome membranes after processing

Exercise-induced BCL2-regulated autophagy is required for muscle glucose homeostasis

Promotion of tumorigenesis by heterozygous disruption of the beclin 1 autophagy gene

Physical exercise stimulates autophagy in normal skeletal muscles but is detrimental for collagen VI-deficient muscles

Autophagy fights disease through cellular self-digestion

Development by self-digestion: molecular mechanisms and biological functions of autophagy

Transcriptional co-activator PGC-1 alpha drives the formation of slow-twitch muscle fibres

Diet, lifestyle, and the risk of type 2 diabetes mellitus in women

HDAC6 controls autophagosome maturation essential for ubiquitin-selective quality-control autophagy

PGC-1alpha plays a functional role in exercise-induced mitochondrial biogenesis and angiogenesis but not fiber-type transformation in mouse skeletal muscle.

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10.1096/FJ.13-228486

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10

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27

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245029090

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23825228

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4046188

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