TASK-3 channel deletion in mice recapitulates low-renin essential hypertension. - Wikidata - awgldk - TriplyDB

TASK-3 channel deletion in mice recapitulates low-renin essential hypertension.

TASK-3 channel deletion in mice recapitulates low-renin essential hypertension.

http://www.wikidata.org/entity/Q30419556

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Primary Aldosteronism: Changing Definitions and New Concepts of Physiology and Pathophysiology Both Inside and Outside the Kidney.KCNK3 Variants Are Associated With Hyperaldosteronism and Hypertension.Variations in the potassium channel genes KCNK3 and KCNK9 in relation to blood pressure and aldosterone production: an exploratory study Minireview: aldosterone biosynthesis: electrically gated for our protection Minireview: potassium channels and aldosterone dysregulation: is primary aldosteronism a potassium channelopathy?Molecular aspects of structure, gating, and physiology of pH-sensitive background K2P and Kir K+-transport channels.Two-pore domain potassium channels in the adrenal cortex.Molecular and Cellular Mechanisms of Aldosterone Producing Adenoma Development Potassium channels related to primary aldosteronism: Expression similarities and differences between human and rat adrenals.Breathing Stimulant Compounds Inhibit TASK-3 Potassium Channel Function Likely by Binding at a Common Site in the Channel Pore.Mouse models of adrenocortical tumors.Regulation of aldosterone biosynthesis by the Kir3.4 (KCNJ5) potassium channel.Potassium channels: structures, diseases, and modulators.Steroidogenesis-adrenal cell signal transduction.Regulation of aldosterone synthesis and secretion.The family of K2P channels: salient structural and functional properties.Of channels and pumps: different ways to boost the aldosterone?An extracellular ion pathway plays a central role in the cooperative gating of a K(2P) K+ channel by extracellular pH.Functional TASK-3-Like Channels in Mitochondria of Aldosterone-Producing Zona Glomerulosa Cells.Mouse Models of Primary Aldosteronism: From Physiology to Pathophysiology.Dkk3 is a component of the genetic circuitry regulating aldosterone biosynthesis in the adrenal cortex.Of Mice and Man and the Regulation of Aldosterone Secretion.

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TASK-3 channel deletion in mice recapitulates low-renin essential hypertension.

http://www.wikidata.org/entity/Q30419556

description

2012 nî lūn-bûn

@nan

2012 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2012 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2012年の論文

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2012年論文

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2012年論文

@zh-hant

2012年論文

@zh-hk

2012年論文

@zh-mo

2012年論文

@zh-tw

2012年论文

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name

TASK-3 channel deletion in mice recapitulates low-renin essential hypertension.

@ast

TASK-3 channel deletion in mice recapitulates low-renin essential hypertension.

@en

type

label

TASK-3 channel deletion in mice recapitulates low-renin essential hypertension.

@ast

TASK-3 channel deletion in mice recapitulates low-renin essential hypertension.

@en

prefLabel

TASK-3 channel deletion in mice recapitulates low-renin essential hypertension.

@ast

TASK-3 channel deletion in mice recapitulates low-renin essential hypertension.

@en

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HYPERTENSIONAHA.111.189662

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TASK-3 channel deletion in mice recapitulates low-renin essential hypertension.

@en

P2093

David A Tyson

http://www.w3.org/2001/XMLSchema#string

Elaine M Schertz

http://www.w3.org/2001/XMLSchema#string

Junlan Yao

http://www.w3.org/2001/XMLSchema#string

Nick A Guagliardo

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Paula Q Barrett

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Robert M Carey

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P2860

Role of angiotensin II in potassium-mediated stimulation of aldosterone secretion in the dog

Studies of the control of plasma aldosterone concentration in normal man. II. Effect of dietary potassium and acute potassium infusion

TASK channels are not required to mount an aldosterone secretory response to metabolic acidosis in mice.

Motoneuronal TASK channels contribute to immobilizing effects of inhalational general anesthetics

TASK channel deletion in mice causes primary hyperaldosteronism.

The neurohormonal natural history of essential hypertension: towards primary or tertiary aldosteronism?

Salt-sensitive blood pressure in mice with increased expression of aldosterone synthase.

Abnormally sustained aldosterone secretion during salt loading in patients with various forms of benign hypertension; relation to plasma renin activity.

K+ channel mutations in adrenal aldosterone-producing adenomas and hereditary hypertension

Low renin hypertensive states: perspectives, unsolved problems, future research.

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999-1005

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scholarly article

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10.1161/HYPERTENSIONAHA.111.189662

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5

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59

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Douglas A Bayliss

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2012-04-09T00:00:00Z

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22493079

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arterial hypertension

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3357084

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