AID activity in B cells strongly correlates with polyclonal antibody affinity maturation in-vivo following pandemic 2009-H1N1 vaccination in humans. - Wikidata - awgldk - TriplyDB

AID activity in B cells strongly correlates with polyclonal antibody affinity maturation in-vivo following pandemic 2009-H1N1 vaccination in humans.

AID activity in B cells strongly correlates with polyclonal antibody affinity maturation in-vivo following pandemic 2009-H1N1 vaccination in humans.

http://www.wikidata.org/entity/Q30421821

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Premature B-cell senescence as a consequence of chronic immune activation.Waning and aging of cellular immunity to Bordetella pertussis The kinase mTOR modulates the antibody response to provide cross-protective immunity to lethal infection with influenza virus.Heterologous prime-boost vaccination with MF59-adjuvanted H5 vaccines promotes antibody affinity maturation towards the hemagglutinin HA1 domain and broad H5N1 cross-clade neutralization Oral priming with replicating adenovirus serotype 4 followed by subunit H5N1 vaccine boost promotes antibody affinity maturation and expands H5N1 cross-clade neutralization.Effects of age on H1N1-specific serum IgG1 and IgG3 levels evaluated during the 2011-2012 influenza vaccine season Age-associated cross-reactive antibody-dependent cellular cytotoxicity toward 2009 pandemic influenza A virus subtype H1N1.Paradoxical aging in HIV: immune senescence of B Cells is most prominent in young age.Understanding immunosenescence to improve responses to vaccines CD25(+) Bcl6(low) T follicular helper cells provide help to maturing B cells in germinal centers of human tonsil.B cell function and influenza vaccine responses in healthy aging and disease.Cytomegalovirus (CMV) seropositivity decreases B cell responses to the influenza vaccine.Profiles of influenza A/H1N1 vaccine response using hemagglutination-inhibition titers MicroRNAs miR-155 and miR-16 Decrease AID and E47 in B Cells from Elderly Individuals High-Affinity H7 Head and Stalk Domain-Specific Antibody Responses to an Inactivated Influenza H7N7 Vaccine After Priming With Live Attenuated Influenza Vaccine Inflammaging decreases adaptive and innate immune responses in mice and humans Young and elderly patients with type 2 diabetes have optimal B cell responses to the seasonal influenza vaccine.Age related differences in dynamics of specific memory B cell populations after clinical pertussis infection.High TNF-α levels in resting B cells negatively correlate with their response.A comparative review of aging and B cell function in mice and humans Age effects on mouse and human B cells.B Cell-Specific Biomarkers for Optimal Antibody Responses to Influenza Vaccination and Molecular Pathways That Reduce B Cell Function with Aging.Aging, cytomegalovirus (CMV) and influenza vaccine responses.Memory B cells form in aged mice despite impaired affinity maturation and germinal center kinetics.Ageing and obesity similarly impair antibody responses.Aging of the immune system: Focus on inflammation and vaccination.Impact of Aging and Cytomegalovirus on Immunological Response to Influenza Vaccination and Infection.The generation of memory B cells is maintained, but the antibody response is not, in the elderly after repeated influenza immunizations.Metformin improves in vivo and in vitro B cell function in individuals with obesity and Type-2 Diabetes.Seeking help: B cells adapting to flu variability.T Follicular Helper Cells and B Cell Dysfunction in Aging and HIV-1 Infection.Dissecting the hemagglutinin head and stalk-specific IgG antibody response in healthcare workers following pandemic H1N1 vaccination.Fighting against a protean enemy: immunosenescence, vaccines, and healthy aging.AS03- and MF59-Adjuvanted Influenza Vaccines in Children.Influenza vaccine response: future perspectives.Adipose Tissue Inflammation Induces B Cell Inflammation and Decreases B Cell Function in Aging.Severe pleuritis and pericarditis associated with very-late-onset systemic lupus erythematosus.Comparative Analysis of B-Cell Receptor Repertoires Induced by Live Yellow Fever Vaccine in Young and Middle-Age Donors Older Human B Cells and Antibodies

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AID activity in B cells strongly correlates with polyclonal antibody affinity maturation in-vivo following pandemic 2009-H1N1 vaccination in humans.

http://www.wikidata.org/entity/Q30421821

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2012 nî lūn-bûn

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2012 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2012 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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AID activity in B cells strong ...... 09-H1N1 vaccination in humans.

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AID activity in B cells strong ...... 09-H1N1 vaccination in humans.

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type

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AID activity in B cells strong ...... 09-H1N1 vaccination in humans.

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AID activity in B cells strong ...... 09-H1N1 vaccination in humans.

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AID activity in B cells strong ...... 09-H1N1 vaccination in humans.

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AID activity in B cells strong ...... 09-H1N1 vaccination in humans.

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AID activity in B cells strong ...... 09-H1N1 vaccination in humans.

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Bonnie Blomberg

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Daniela Frasca

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Hana Golding

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Surender Khurana

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P2860

Hospitalized patients with 2009 H1N1 influenza in the United States, April-June 2009

Class switch recombination and hypermutation require activation-induced cytidine deaminase (AID), a potential RNA editing enzyme

Emergence of a novel swine-origin influenza A (H1N1) virus in humans

Antigenic fingerprinting of H5N1 avian influenza using convalescent sera and monoclonal antibodies reveals potential vaccine and diagnostic targets

Severe respiratory disease concurrent with the circulation of H1N1 influenza.

Critical care services and 2009 H1N1 influenza in Australia and New Zealand.

Vaccines with MF59 adjuvant expand the antibody repertoire to target protective sites of pandemic avian H5N1 influenza virus.

Properly folded bacterially expressed H1N1 hemagglutinin globular head and ectodomain vaccines protect ferrets against H1N1 pandemic influenza virus

Prevalence of seroprotection against the pandemic (H1N1) virus after the 2009 pandemic.

Severe pandemic 2009 H1N1 influenza disease due to pathogenic immune complexes.

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