Inflammation early in life is a vulnerability factor for emotional behavior at adolescence and for lipopolysaccharide-induced spatial memory and neurogenesis alteration at adulthood.
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Environmental Enteropathy: Elusive but Significant Subclinical Abnormalities in Developing Countries.Microglial Priming and Alzheimer's Disease: A Possible Role for (Early) Immune Challenges and Epigenetics?Stress-Related Immune Markers in Depression: Implications for TreatmentEarly Postnatal Lipopolysaccharide Exposure Leads to Enhanced Neurogenesis and Impaired Communicative Functions in RatsNeuroinflammation in Autism: Plausible Role of Maternal Inflammation, Dietary Omega 3, and MicrobiotaThe interplay of early-life stress, nutrition, and immune activation programs adult hippocampal structure and functionEarly-life enteric infections: relation between chronic systemic inflammation and poor cognition in childrenDeficient adolescent social behavior following early-life inflammation is ameliorated by augmentation of anandamide signaling.Postnatal LPS Challenge Impacts Escape Learning and Expression of Plasticity Factors Mmp9 and Timp1 in Rats: Effects of Repeated Training.Prenatal stress is a vulnerability factor for altered morphology and biological activity of microglia cellsChronic intestinal inflammation alters hippocampal neurogenesis.Systemic inflammation in early neonatal mice induces transient and lasting neurodegenerative effects.Neuroinflammatory Dynamics Underlie Memory Impairments after Repeated Social Defeat.Neurobehavioral comorbidities of epilepsy: Role of inflammation.Neonatal Immune Challenge with Lipopolysaccharide Triggers Long-lasting Sex- and Age-related Behavioral and Immune/Neurotrophic Alterations in Mice: Relevance to Autism Spectrum Disorders.TLR4 response mediates ethanol-induced neurodevelopment alterations in a model of fetal alcohol spectrum disorders.N-acetyl cysteine reverses bio-behavioural changes induced by prenatal inflammation, adolescent methamphetamine exposure and combined challenges.LPS-induced neonatal stress in mice affects the response profile to an inflammatory stimulus in an age and sex-dependent manner.Acute in utero exposure to lipopolysaccharide induces inflammation in the pre- and postnatal brain and alters the glial cytoarchitecture in the developing amygdala.Role of the innate immune system in the neuropathological consequences induced by adolescent binge drinking.Sickness-induced changes in physiology do not affect fecundity or same-sex behavior.Role of maternal health and infant inflammation in nutritional and neurodevelopmental outcomes of two-year-old Bangladeshi children.Revisiting Inbred Mouse Models to Study the Developing Brain: The Potential Role of Intestinal Microbiota
P2860
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P2860
Inflammation early in life is a vulnerability factor for emotional behavior at adolescence and for lipopolysaccharide-induced spatial memory and neurogenesis alteration at adulthood.
description
2014 nî lūn-bûn
@nan
2014 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Inflammation early in life is ...... nesis alteration at adulthood.
@ast
Inflammation early in life is ...... nesis alteration at adulthood.
@en
type
label
Inflammation early in life is ...... nesis alteration at adulthood.
@ast
Inflammation early in life is ...... nesis alteration at adulthood.
@en
prefLabel
Inflammation early in life is ...... nesis alteration at adulthood.
@ast
Inflammation early in life is ...... nesis alteration at adulthood.
@en
P2093
P2860
P50
P921
P1476
Inflammation early in life is ...... enesis alteration at adulthood
@en
P2093
Agnes Aubert
Anne-Laure Dinel
Pascale Le Ruyet
P2860
P2888
P356
10.1186/S12974-014-0155-X
P577
2014-09-17T00:00:00Z
P5875
P6179
1030012004